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1 e beta-sheets, representing an evolutionary "cul-de-sac".
2  mutations and thus leads to an evolutionary cul-de-sac.
3  terminal gas-exchange structures, which are cul-de-sacs.
4 oncentrated in the posteriormost tips of the cul-de-sacs and recesses at the reflection of the OE at
5  the electrode device placed in the superior cul-de-sac away from the limbus and on the cornea adjace
6                   Patients with fluid in the cul-de-sac had similar injury and surgical injury rates
7 n lead to charge trapping at bottlenecks and cul-de-sacs in the conducting pathways to the electrodes
8                           Ascites beyond the cul-de-sac, irrespective of attenuation, is most predict
9  age with trauma, free fluid isolated to the cul-de-sac is likely physiologic; clinical follow-up sho
10 hese results, but rather than being climatic cul-de-sacs, many mountain streams appear poised to be r
11 illing 50 mul of sterile saline to the lower cul-de-sac of each eye and using capillary action microc
12  or 0.1% or 1.0% dexamethasone, in the lower cul-de-sac of each eye.
13 oadhesive microdiscs was administered in the cul-de-sac of the rabbit eye in vivo, these microdiscs e
14 unopositive with 2C6 are concentrated in the cul-de-sacs of ectoturbinates 1 and 2 and of endoturbina
15 scans were positive if fluid was outside the cul-de-sac or if suspicious parenchymal abnormalities we
16 psular vs extracapsular), ascites beyond the cul-de-sac, peritoneal implants, ipsilateral pleural eff
17    In 56, anechoic fluid was isolated to the cul-de-sac: Two had injuries; one was surgical.
18                           Ascites beyond the cul-de-sac was the single best indicator of rupture for
19 or flow, rebounding off the leaflet into the cul-de-sac, was noted in 82% of the obstructed HCM, 9% o

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