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1 pitulated by respiratory chain inhibition in cultured myotubes.
2 prevents lipid-induced insulin resistance in cultured myotubes.
3 ation and the dispersion of AChR clusters in cultured myotubes.
4 can (rhBGN) increases utrophin expression in cultured myotubes.
5 on of the illuminated AChRs from clusters on cultured myotubes.
6 e and HDAC4 knockdown enhances glycolysis in cultured myotubes.
7 e inhibition of UPR causes severe atrophy in cultured myotubes.
8 ay arise from the NMJ basement membrane with cultured myotubes.
9 and cardiac muscles, murine limb muscle, and cultured myotubes.
10 ression in mouse limb muscles in vivo and in cultured myotubes.
11 -4) inhibit agrin-induced AChR clustering on cultured myotubes.
12 gulating mitochondrial oxidative function in cultured myotubes.
13  calcium in agrin-induced AChR clustering on cultured myotubes.
14 on associated with AChR cluster dispersal in cultured myotubes.
15 s and triggers formation of AChR clusters on cultured myotubes.
16 vated AMPK and stimulated lipid oxidation in cultured myotubes.
17 e receptor (AChR) clusters on the surface of cultured myotubes.
18                                           In cultured myotubes, Abl kinase activity was required for
19                                           In cultured myotubes, agrin stimulates the rapid phosphoryl
20  agrin and neuregulin-1 (Nrg-1) signaling in cultured myotubes and developing muscle fibers in vivo.
21 nduced reactive oxygen species production in cultured myotubes and improved insulin-stimulated glucos
22 mor-induced muscle catabolism and wasting in cultured myotubes and in mice.
23  endogenous AChR beta-subunit transcripts in cultured myotubes and in vivo, and this binding is incre
24  number of lipid-challenged models including cultured myotubes and isolated muscles strips incubated
25 ored whether MTX promotes AMPK activation in cultured myotubes and isolated skeletal muscle.
26                                              Cultured myotubes and primary brown adipocytes treated w
27 tes was comparable between muscle tissue and cultured myotubes, and temporal lipid profiles correlate
28                                           In cultured myotubes, APP synergistically increases agrin-i
29 s increased in skeletal muscle tissue and in cultured myotubes basally and in response to insulin in
30 e readily extracted from the cell surface of cultured myotubes by non-ionic detergent.
31                Addition of agonist scFv to a cultured myotube cell line induced AChR clustering and t
32                        Silencing of Perm1 in cultured myotubes compromises respiratory capacity and d
33                                           In cultured myotubes, CTRP9 specifically activates AMPK, Ak
34                                              Cultured myotubes derived from two patients with GFPT1 m
35 maging of acetylcholine receptors (AChRs) in cultured myotubes differentiated ex vivo from immortaliz
36  exposure on glucose and lipid metabolism in cultured myotubes established from people with normal gl
37                                              Cultured myotubes exposed to high CO2 had reduced fiber
38 as supported by elevated oxidative stress in cultured myotubes exposed to palmitate in the presence o
39 mparisons with changes in gene expression in cultured myotubes following treatment with a non-damagin
40            Studies were conducted in primary cultured myotubes from beta1 knockout (KO), ryanodine re
41  the levels of GLUT4 ( approximately 50%) in cultured myotubes from C57BL6 mice.
42 ell as IL-6 secretion, was unaltered between cultured myotubes from normal glucose tolerant or type 2
43 f transcription 3 (STAT3) phosphorylation in cultured myotubes from normal glucose tolerant subjects.
44 athies, the relatively relaxed zebrafish and cultured myotubes from patients with RYR1-related myopat
45         Both in transfected COS cells and in cultured myotubes from rapsyn-negative and rapsyn-positi
46 tractility and Ca(2+) release from the SR of cultured myotubes from Stac3 mutant mice could be restor
47 hen TrkB-mediated signaling was disrupted in cultured myotubes in the absence of motor nerve terminal
48 rs (AChRs) at laminin-associated clusters on cultured myotubes in the absence or presence of the nerv
49               Inhibition of ERK signaling in cultured myotubes increased slow-twitch fiber-specific p
50 y, forced expression of Gadd45a in muscle or cultured myotubes induces atrophy in the absence of upst
51                                           In cultured myotubes, inhibition of ERK, but not Jun NH2-te
52 e acetylcholine receptor (AChR) synthesis in cultured myotubes, is a member of the neuregulin family
53         Thus, in muscles in vivo, but not in cultured myotubes, neural agrin promotes the recycling o
54 Recombinant human IL-6 (rhIL-6) treatment of cultured myotubes only minimally increased SOCS3, howeve
55  was used to monitor Ca2+ signals in primary-cultured myotubes, prepared from forelimbs of wild-type
56             Agrin-induced AChR clustering in cultured myotubes proceeds via the initial formation of
57         Addition of soluble TWEAK protein to cultured myotubes reduced the mean myotube diameter and
58        Gain- and loss-of-function studies in cultured myotubes show that regulation of MuSK by PDZRN3
59                      Immunohistochemistry of cultured myotubes shows that not only is the beta 1 subu
60                                     Like the cultured myotubes, these tissues accumulated ceramides b
61                                           In cultured myotubes, Tid1 knockdown inhibited AChR cluster
62                            Exposure of C2C12 cultured myotubes to either high glucose concentration,
63                        Here, we show that in cultured myotubes undergoing atrophy, the activity of th
64 hR density at agrin-induced AChR clusters in cultured myotubes via PI3 kinase acting through GSK3beta
65 ed aggregation of acetylcholine receptors on cultured myotubes was completely blocked by antibodies t
66                                           In cultured myotubes, we determined that alphaLNNd expressi
67  was used but not when nuclear extracts from cultured myotubes were used.
68 so coordinately regulated on the surfaces of cultured myotubes where MuSK and AChRs colocalize both i
69                                     Treating cultured myotubes with LDL containing ceramide promoted

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