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1 optosis and suppressing the proliferation of cultured tumor cells.
2 d displayed on oil drops induce apoptosis of cultured tumor cells.
3  with respect to EC20 was then determined in cultured tumor cells.
4 duced under hypoxic conditions in tumors and cultured tumor cells.
5  only approximately 2% of expressed genes in cultured tumor cells.
6 ncharacterized mutations present only in the cultured tumor cells, a subset of which has been reporte
7 s resulted in enhanced antitumor activity in cultured tumor cells and animal models.
8 duces proteasome inhibition and apoptosis in cultured tumor cells and cancer cells from acute myeloid
9 factor-1 expression and promoter activity in cultured tumor cells and HaCaT keratinocytes, as well as
10 s demonstrated an allelic loss of an NOL7 in cultured tumor cells and human tumor samples.
11 NA methyltransferase 1-STAT3 interactions in cultured tumor cells and in tumors.
12 d in altered phospholipid metabolism both in cultured tumor cells and in vivo.
13 eported that overexpression of the enzyme in cultured tumor cells and mice activates metabolic flux t
14 hamide potentiated oncolytic effects against cultured tumor cells and subcutaneous tumor xenografts e
15 w that p300 is susceptible to acetylation in cultured tumor cells and that its acetylation status is
16 l spread at later stages, as demonstrated in cultured tumor cells, and in A549 and PC-3 solid tumor x
17 hat testisin was localized on the surface of cultured tumor cells as a glycosyl-phosphatidylinositol-
18  that RAGE is present in the mitochondria of cultured tumor cells as well as primary tumors.
19 proliferation, viability and invasiveness of cultured tumor cells, as well as the growth rate and met
20 ted anticancer prodrug whose toxicity toward cultured tumor cells can be potentiated up to 100-fold b
21                  Down-regulation of LRRK2 in cultured tumor cells compromises MET activation and sele
22  expression between the solid tumors and the cultured tumor cells correlate with the expression of ga
23  not expressed or is abnormally expressed in cultured tumor cells derived from HCC.
24                                        As in cultured tumor cells, Enzastaurin treatment suppresses t
25  results from the prior Hip1 mutant mice, we cultured tumor cells from homozygous Delta 3-5 allele-be
26 1 or PV mainly in the nuclear compartment of cultured tumor cells from TRbeta(PV/PV) mice, but cytopl
27 by quantitative reverse transcriptase PCR in cultured tumor cells in vitro and in a mouse xenograft m
28                      In contrast to in vitro cultured tumor cells, in vivo growing tumor cells are mo
29 6His protein did not affect the viability of cultured tumor cells, indicating that the antitumor effe
30                             Liver tumors and cultured tumor cell lines from dHGF transgenics showed h
31 od lymphocytes and to a far lesser extent in cultured tumor cell lines.
32 erties of 99mTc-HYNIC-folate were studied in cultured tumor cells that overexpress the folate recepto
33                   iRGD-CDD internalized into cultured tumor cells through a neuropilin-1-activated pa
34 vo, and they raise concerns about the use of cultured tumor cells to test the efficacy of Shh pathway
35                  LNAs can be introduced into cultured tumor cells using cationic lipid, with diffuse
36 tor-mediated uptake of 99mTc-HYNIC-folate by cultured tumor cells was approximately 300 times higher
37                DBCCR1-mediated cell death in cultured tumor cells was independent of caspase-3 activa
38                                              Cultured tumor cells were available to assess in vitro c
39       Despite their distinct morphology, all cultured tumor cells were Tamoxifen resistant but highly
40                                 Treatment of cultured tumor cells with PES promotes cell death that i

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