ライフサイエンスコーパス: cumene

1 mol(-1) (cyclohexane) to 84.5 kcal mol(-1) (cumene).

2 Vitamin E in a polar medium comprised of 80% cumene and 20% methanol.

3 In the inhibited autoxidation of cumene and styrene at 303 K, magnolol trapped four perox

4 Oxidation of the prochiral substrate cumene by CYP4B1, but not CYP2B1 or CYP102, resulted in

5 ene, together with the purification of (iii) cumene from its major impurities (benzene, n-propylbenze

6 titutively resistant to 100 mM H2O2 and 5 mM cumene hydroperoxide (15-min exposure).

7 hydroperoxide (11.1 +/- 0.3 M(-2) s(-1)) or cumene hydroperoxide (25 +/- 2 M(-2) s(-1)).

8 egmatis wild-type strain (MSWt) induced with cumene hydroperoxide (CHP) and t-butyl hydroperoxide (t-

9 lted in sensitivity to the organic peroxides cumene hydroperoxide (CHP) and t-butyl hydroperoxide (TB

10 sion, whereas organic hydroperoxides such as cumene hydroperoxide (CHP) deactivate AphB and OhrR.

11 oxidants tert-butyl hydroperoxide (tBHP) and cumene hydroperoxide (CHP) supported the inactivation of

12 ic acid intermediate at Cys(61) generated by cumene hydroperoxide (CHP) treatment was detected in UV-

13 In the presence of cumene hydroperoxide (CHP), oxidation of OhrR leads to a

14 reciprocal of P infinity is proportional to cumene hydroperoxide (CuOOH) concentration, but P infini

15 In bovine irides, hydrogen peroxide (H2O2), cumene hydroperoxide (cuOOH), and tert-butyl hydroperoxi

16 e characterization of the phosphorylation of cumene hydroperoxide (CuOOH)-inactivated cytochrome P450

17 mal human epidermal keratinocytes exposed to cumene hydroperoxide after metabolic incorporation of th

18 was determined during oxidant challenge from cumene hydroperoxide and H(2)O(2).

19 eightened sensitivities to the toxic oxidant cumene hydroperoxide and to the antibiotic rifampin.

20 vity for the epoxidation of cyclohexene with cumene hydroperoxide as oxidant.

21 ve to the oxidative damaging agents H2O2 and cumene hydroperoxide compared to the parental strain.

22 H-P450 reductase/NADPH and the model oxidant cumene hydroperoxide have been proposed by others to be

23 l-t-9,10-epoxide (BaP-diol-t-epoxide), using cumene hydroperoxide in lieu of NADPH/O(2).

24 n addition, reaction of (N4)Pd(II)Me(2) with cumene hydroperoxide involves a heterolytic O-O bond cle

25 re redox-mediated as addition of H(2)O(2) or cumene hydroperoxide leads to the dissociation of OspR f

26 europneumoniae serotype 5 was not induced by cumene hydroperoxide or by other forms of oxidative stre

27 xidative activity of CYP2S1 was supported by cumene hydroperoxide or H(2)O(2), such that CYP2S1 oxidi

28 Although cumene hydroperoxide produced significant oxidation of c

29 rogen peroxide, tert-butyl hydroperoxide, or cumene hydroperoxide relative to the parent strain, sugg

30 Cumene hydroperoxide stimulated radical-mediated lipid p

31 H mutant was found to be more susceptible to cumene hydroperoxide stress but to be similar in logarit

32 also more sensitive to hydrogen peroxide and cumene hydroperoxide than the parental strain.

33 xidation using (+)-DET, Ti(O(i)()Pr)(4), and cumene hydroperoxide to give the trans bis-sulfoxides 4a

34 high degree of protection against killing by cumene hydroperoxide to the host E. coli cells.

35 Cumene hydroperoxide was a reversible inhibitor of the r

36 eraction between lipoxygenase-1 (and -3) and cumene hydroperoxide was investigated.

37 acing NADPH and O2 with the oxygen surrogate cumene hydroperoxide yielded similar results.

38 iCl, and Na(2)SO(4)), oxidants (H(2)O(2) and cumene hydroperoxide), and organic compounds (abscisic a

39 crynic acid and GSH peroxidase activity with cumene hydroperoxide, 9-hydroperoxy-trans-10, cis-12-oct

40 tion correlated with increased resistance to cumene hydroperoxide, an organic hydroperoxide, but not

41 he oxidants tert-butyl hydroperoxide (TBHP), cumene hydroperoxide, and cisplatin resulted in time- an

42 P450 systems utilizing NADPH-P450 reductase, cumene hydroperoxide, and iodosylbenzene use similar but

43 It reduces H2O2, t-butyl hydroperoxide, and cumene hydroperoxide, and is inhibited by sulfhydryl rea

44 nst oxidative damage caused by both H2O2 and cumene hydroperoxide, and that NGO554, a Gc-specific pro

45 ld) was also observed in GPX activity toward cumene hydroperoxide, but CAT and SOD activities remaine

46 serotype 1, expression of ohr was induced by cumene hydroperoxide, but not by either hydrogen peroxid

47 t of heterolytic cleavage of the O-O bond of cumene hydroperoxide, consistent with transient formatio

48 xide reductase assays have demonstrated that cumene hydroperoxide, ethyl hydroperoxide, and hydrogen

49 In all extracts tested, the activity against cumene hydroperoxide, even when glutathione S-transferas

50 en, respectively, in the presence of DNA and cumene hydroperoxide, induced two adducts, which mapped

51 idizing agents (H2O2, t-butyl hydroperoxide, cumene hydroperoxide, or diamide) or by addition of Zn2+

52 hydrogen peroxide, tert-butyl hydroperoxide, cumene hydroperoxide, or sulfhydryl modification by N-et

53 Unlike protection against cumene hydroperoxide, protection afforded by ahpC agains

54 stress-inducing compounds hydrogen peroxide, cumene hydroperoxide, t-butyl hydroperoxide, or diamide.

55 Exposing PDGF to cumene hydroperoxide, t-butyl hydroperoxide, or hydrogen

56 ity, albeit diminished, after treatment with cumene hydroperoxide, the structure of the iron site may

57 etoprolol, and bufuralol between reductase-, cumene hydroperoxide-, and iodosylbenzene-supported syst

58 from NQO1-mediated reduction of TQ prevented cumene hydroperoxide-induced lipid peroxidation in rat l

59 In addition, cumene hydroperoxide-induced lipid peroxidation was inhi

60 Cumene hydroperoxide-mediated (CuOOH-mediated) inactivat

61 Cumene hydroperoxide-supported metabolism was measured t

62 iodosylbenzene- but not in the reductase- or cumene hydroperoxide-supported reactions.

63 city, with comparable rates for H(2)O(2) and cumene hydroperoxide.

64 BaP-7,8-diol at a much higher rate than with cumene hydroperoxide.

65 tive to killing by t-butyl hydroperoxide and cumene hydroperoxide.

66 ve than the parent strain to both oxygen and cumene hydroperoxide.

67 nts were highly sensitive to paraquat and to cumene hydroperoxide.

68 ot serotype 5 cultures, were able to degrade cumene hydroperoxide.

69 tidylserine externalization upon exposure to cumene hydroperoxide.

70 on upon challenge with hydrogen peroxide and cumene hydroperoxide.

71 d type to growth suppression by paraquat and cumene hydroperoxide.

72 grees C heat shock and following exposure to cumene hydroperoxide.

73 xin or ferredoxin system, iodosylbenzene, or cumene hydroperoxide.

74 to the oxidations of toluene, ethylbenzene, cumene, indene, and cyclohexene.

75 ree radical-mediated aerobic peroxidation of cumene than is PBN and the experimental stroke drug NXY-

76 an bind guests such as 2-methyl-2-butene and cumene to form stable solid host-guest complexes.

77 ctivity as well as the selective reaction of cumene to the tertiary alcohol, alpha,alpha'-dimethyl be

78 ying mixtures of toluene, ethyl benzene, and cumene were analyzed by purge-and-trap/direct sampling m