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1 tive to killing by t-butyl hydroperoxide and cumene hydroperoxide.
2 ve than the parent strain to both oxygen and cumene hydroperoxide.
3 nts were highly sensitive to paraquat and to cumene hydroperoxide.
4 ot serotype 5 cultures, were able to degrade cumene hydroperoxide.
5 tidylserine externalization upon exposure to cumene hydroperoxide.
6 on upon challenge with hydrogen peroxide and cumene hydroperoxide.
7 d type to growth suppression by paraquat and cumene hydroperoxide.
8 grees C heat shock and following exposure to cumene hydroperoxide.
9 city, with comparable rates for H(2)O(2) and cumene hydroperoxide.
10 xin or ferredoxin system, iodosylbenzene, or cumene hydroperoxide.
11 BaP-7,8-diol at a much higher rate than with cumene hydroperoxide.
14 crynic acid and GSH peroxidase activity with cumene hydroperoxide, 9-hydroperoxy-trans-10, cis-12-oct
15 mal human epidermal keratinocytes exposed to cumene hydroperoxide after metabolic incorporation of th
16 tion correlated with increased resistance to cumene hydroperoxide, an organic hydroperoxide, but not
18 eightened sensitivities to the toxic oxidant cumene hydroperoxide and to the antibiotic rifampin.
19 iCl, and Na(2)SO(4)), oxidants (H(2)O(2) and cumene hydroperoxide), and organic compounds (abscisic a
20 he oxidants tert-butyl hydroperoxide (TBHP), cumene hydroperoxide, and cisplatin resulted in time- an
21 P450 systems utilizing NADPH-P450 reductase, cumene hydroperoxide, and iodosylbenzene use similar but
22 It reduces H2O2, t-butyl hydroperoxide, and cumene hydroperoxide, and is inhibited by sulfhydryl rea
23 nst oxidative damage caused by both H2O2 and cumene hydroperoxide, and that NGO554, a Gc-specific pro
24 etoprolol, and bufuralol between reductase-, cumene hydroperoxide-, and iodosylbenzene-supported syst
26 ld) was also observed in GPX activity toward cumene hydroperoxide, but CAT and SOD activities remaine
27 serotype 1, expression of ohr was induced by cumene hydroperoxide, but not by either hydrogen peroxid
28 egmatis wild-type strain (MSWt) induced with cumene hydroperoxide (CHP) and t-butyl hydroperoxide (t-
29 lted in sensitivity to the organic peroxides cumene hydroperoxide (CHP) and t-butyl hydroperoxide (TB
30 sion, whereas organic hydroperoxides such as cumene hydroperoxide (CHP) deactivate AphB and OhrR.
31 oxidants tert-butyl hydroperoxide (tBHP) and cumene hydroperoxide (CHP) supported the inactivation of
32 ic acid intermediate at Cys(61) generated by cumene hydroperoxide (CHP) treatment was detected in UV-
34 ve to the oxidative damaging agents H2O2 and cumene hydroperoxide compared to the parental strain.
35 t of heterolytic cleavage of the O-O bond of cumene hydroperoxide, consistent with transient formatio
36 reciprocal of P infinity is proportional to cumene hydroperoxide (CuOOH) concentration, but P infini
37 In bovine irides, hydrogen peroxide (H2O2), cumene hydroperoxide (cuOOH), and tert-butyl hydroperoxi
38 e characterization of the phosphorylation of cumene hydroperoxide (CuOOH)-inactivated cytochrome P450
39 xide reductase assays have demonstrated that cumene hydroperoxide, ethyl hydroperoxide, and hydrogen
40 In all extracts tested, the activity against cumene hydroperoxide, even when glutathione S-transferas
41 H-P450 reductase/NADPH and the model oxidant cumene hydroperoxide have been proposed by others to be
43 en, respectively, in the presence of DNA and cumene hydroperoxide, induced two adducts, which mapped
44 from NQO1-mediated reduction of TQ prevented cumene hydroperoxide-induced lipid peroxidation in rat l
46 n addition, reaction of (N4)Pd(II)Me(2) with cumene hydroperoxide involves a heterolytic O-O bond cle
47 re redox-mediated as addition of H(2)O(2) or cumene hydroperoxide leads to the dissociation of OspR f
49 europneumoniae serotype 5 was not induced by cumene hydroperoxide or by other forms of oxidative stre
50 xidative activity of CYP2S1 was supported by cumene hydroperoxide or H(2)O(2), such that CYP2S1 oxidi
51 idizing agents (H2O2, t-butyl hydroperoxide, cumene hydroperoxide, or diamide) or by addition of Zn2+
52 hydrogen peroxide, tert-butyl hydroperoxide, cumene hydroperoxide, or sulfhydryl modification by N-et
55 rogen peroxide, tert-butyl hydroperoxide, or cumene hydroperoxide relative to the parent strain, sugg
57 H mutant was found to be more susceptible to cumene hydroperoxide stress but to be similar in logarit
60 stress-inducing compounds hydrogen peroxide, cumene hydroperoxide, t-butyl hydroperoxide, or diamide.
63 ity, albeit diminished, after treatment with cumene hydroperoxide, the structure of the iron site may
64 xidation using (+)-DET, Ti(O(i)()Pr)(4), and cumene hydroperoxide to give the trans bis-sulfoxides 4a
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