ライフサイエンスコーパス: cupric

1 emonstrated that this peak is insensitive to cupric A(x) and A(y) hyperfine interaction.

2 in the presence of either palladium acetate/cupric acetate catalytic system under oxygen atmosphere

3 se experiment, levels of (64)Cu from [(64)Cu]cupric acetate decreased from 4 to 24 h postadministrati

4 to 4 and then manganic triacetate dihydrate/cupric acetate induced radical cyclization, gave 1-subst

5 Cupric acetate is the copper source, and triethylamine b

6 different from that for (64)Cu from [(64)Cu]cupric acetate.

7 ance (ENDOR) of protons at Type 2 and Type 1 cupric active sites correlates with the enzymatic pH dep

8 se (PHM) with two coppers that cycle through cupric and cuprous oxidation states.

9 were diazotized with tert-butyl nitrite and cupric chloride to furnish the isomerically pure 5-chlor

10 ble form of copper such as cupric sulfate or cupric chloride.

11 The metal-catalyzed oxidation (ascorbate/cupric chloride/oxygen) of recombinant human relaxin (rh

12 arged, coordinatively unsaturated trinuclear cupric cluster.

13 be controlled in vivo to avoid formation of cupric CzrA.

14 binuclear center in the "as-isolated" ferric/cupric enzyme is sluggish and without linkage to proton

15 y oxidants leading to the elimination of the cupric EPR signal consistent with formation of an antife

16 zotization in the presence of an appropriate cupric halide.

17 reminiscent of plastocyanin, but the Type 1 cupric HOMO ground-state electronic g value and copper h

18 t charge at the adjacent metal site from +1 (cupric hydroxide) in wild-type enzyme to +2 (cobaltous H

19 or contains a benzenesulfonamide prong and a cupric iminodiacetate (IDA-Cu(2+)) prong separated by li

20 ed by oxidation at Calpha by the neighboring cupric ion and cleavage of the Calpha-C(O) bond to give

21 y the presence of chelators, suggesting that cupric ion had previously bound to these LPSs.

22 The participation of cupric ion in the oxidation process was demonstrated in

23 gnals are attributed to type 2 Cu2+ in which cupric ion is bound to four (less likely three) nitrogen

24 Cupric ion may also be taken up, but those processes are

25 cavenging, lipid peroxidation inhibition and cupric ion reducing activities of different fractions we

26 ntent (TPC), ascorbic acid (AA) content, and cupric ion reducing antioxidant capacity (CUPRAC) were d

27 ng from differences in quenching rate by the cupric ion.

28 ciated with the axial position of the Type-1 cupric ion.

29 Dosed cupric ions acted as a disinfectant in stratified stagna

30 It was shown that ferric and cupric ions are effective oxidants of TEMPONE-H.

31 s and their respective primary antibodies by cupric ions at high pH.

32 However, cupric ions did not impair CzrA-DNA complex formation bu

33 ed to a linker sequence at the N-terminus to cupric ions embedded in a polyethylene-glycol-coated gla

34 imately proportional to the concentration of cupric ions in the medium, but increased more rapidly in

35 trophotometric method, based on reduction of cupric ions in the presence of cuproine complex, with a

36 ve inhibition of the proton transfer step by cupric ions in wild-type CA XII.

37 During distillation, cupric ions may catalyse the conversion of cyanide to EC

38 When the pattern of cupric ions on a complex matches the surface pattern of

39 Ferrous, ferric, and cupric ions were also effective catalysts, indicating th

40 han does presence of copper dosed as soluble cupric ions.

41 d to influence the redox potential of Type-1 cupric ions.

42 The activity was stimulated by ferric and cupric metal ions in addition to the cytochrome b-specif

43 disulfide bond formation in the presence of cupric orthophenanthroline.

44 Cupric oxide (CuO) and nickel oxide (NiO) showed signifi

45 attice exchange in the improper multiferroic cupric oxide (CuO) creates electromagnons at substantial

46 bromide-containing waters in the presence of cupric oxide (CuO).

47 (SPH) relationships are established for nano-cupric oxide (n-CuO) as a function of shape, including n

48 We used cupric oxide ligninolysis coupled with gas chromatograph

49 tudy demonstrates the intrinsic abilities of cupric oxide nanoparticles (CuO-NP) towards arsenic adso

50 Dual-functional cupric oxide nanorods (CuONRs) as peroxidase mimics are

51 mg beta-carotene, 80 mg zinc oxide, and 2 mg cupric oxide per day was instituted on study day 2.

52 orms silicon carbide compounds in the heated cupric oxide reactor, rather than forming silicon dioxid

53 C, vitamin E, beta-carotene, and zinc (with cupric oxide) is recommended for AMD but not cataract.

54 no copper, 40% contained the poorly absorbed cupric oxide, and < 30% contained a highly bioavailable

55 Metallic copper (Cu), cupric oxide/hydroxide (Cuox), and copper sulfide (CuxS)

56 nce and for cross-linking in the presence of cupric-phenanthroline by SDS-PAGE and Western blot analy

57 in either mercury-polyvinyl alcohol (PVA) or cupric PVA.

58 berries of Rubus and Ribes genera, had high cupric reducing antioxidant capacity, comparable with th

59 say, beta-carotene/linoleic acid, ferric and cupric reducing power.

60 A cupric reductase activity is also increased in copper-de

61 al characteristics of the regulation of this cupric reductase are compatible with its involvement in

62 e was regulated by copper and could act as a cupric reductase.

63 ns suggest they are physiologically relevant cupric reductases and ferrireductases in vivo.

64 teap4, are not only ferrireductases but also cupric reductases that stimulate cellular uptake of both

65 days from weaning were evaluated with amino cupric silver neurodegeneration stain.

66 Using the amino-cupric silver stain on brain sections from canine narcol

67 generating neurons was found using de Olmos' cupric silver stain.

68 limited seizure activity (stage 1); however, cupric-silver and Fluoro-Jade B stains revealed signific

69 ed for histological evaluation utilizing the cupric-silver neurodegeneration stain, immunohistochemis

70 Amino-cupric-silver staining demonstrated degenerative changes

71 taneous mutant model of human NP-C, by amino-cupric-silver staining, showed that the terminal fields

72 milar, indicating that the structures of the cupric sites, and the spin density distributions onto th

73 n was also observed when cuprous SOD but not cupric SOD was added to a H(2)O(2) solution.

74 consistently undersaturated with respect to cupric solid phases.

75 n reduction of an initially added air-stable cupric species (Cu(II)/Ligand).

76 uttles via a metastable but activated ferric/cupric state (O(H)), which may decay into a more stable

77 ents have shown that reduction of the ferric/cupric state of the enzyme's binuclear heme a3/CuB cente

78 a fibers formed by hydrodynamic injection of cupric sulfate into silicate solution.

79 a highly bioavailable form of copper such as cupric sulfate or cupric chloride.

80 Previous efforts to synthesize a cupric superoxide complex possessing a thioether donor h

81 f a dioxygen adduct with [LCu(I)][B(C6F5)4], cupric superoxo complex [LCu(II)(O2(*-))]+ (1) (L = TMG3

82 ity patterns for copper(I)-O2 adducts, a new cupric-superoxo complex [(DMM-tmpa)Cu(II)(O2(*-))](+) (2