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1 ransformations, from normal to semicoiled to curly 1 and then, when the motor again spun CCW, back to
2 to occur in the sequence normal, semicoiled, curly 1, with changes in the direction of movement of th
3                                         The "curly arrow" of Robinson and Ingold is the primary tool
4                     Our results clarify that curly arrows are rooted in physical reality-a notion whi
5 rt that the bond rearrangements expressed by curly arrows can be directly observed in ab initio compu
6                            The new mutation, curly bare (cub), was mapped to distal Chr 11 and the mo
7 es of morphology: monopolar, simple bipolar, curly bipolar and radial shaped; they were localized in
8                                          The curly bipolar cells had a higher membrane conductance, h
9 urons (monopolar, radial, simple bipolar and curly bipolar) were identified.
10 oiety containing a {Se(V)-Se(V)} bond (where curly brackets indicate a moiety, not a molecule) and re
11 lis and results in the formation of aberrant curly cells.
12  of the dominant Mcub allele confers a full, curly coat in cub/cub mice.
13  association studies for milk production and curly coat, using imputed sequence variants, and identif
14 lotype spanning ~600 Kb was found in all the curly coated cats.
15 trol association study was conducted using 9 curly coated Selkirk Rex and 29 controls, including stra
16 well as a coding SNP that is associated with curly-coated dogs.
17 could form open-coiled, straight, normal, or curly conformations.
18                        In the present study, curly endive (Cichorium endivia L. var. crispum) and esc
19 were significantly affected by variety, with curly endive showing on average higher scores in compari
20 ominated by a straight body segment but with curly entangled top, we have created gecko-foot-mimetic
21 d at lower concentrations, whereas short and curly fibrils containing a mixture of alpha-helical and
22                   The formation of short and curly fibrils is preceded by the formation of a small nu
23 conditions, the oligomers convert into thin, curly fibrils that interact with thioflavin-T, suggestin
24                                The zebrafish curly fry (cfy) mutation leads to embryonic lethality an
25 ment, and differentiation, between Caucasian curly hair and naturally straight hair was performed.
26 lude taurodontism, enamel hypoplasia, kinky, curly hair at birth and increased thickness and density
27 he principal clinical features include kinky/curly hair in infancy, enamel hypoplasia, taurodontism,
28  features with Lrrc8a(-/-) mice that include curly hair, infertility, reduced longevity, and kidney a
29  hair xenografts obtained from straight- and curly-haired individuals was found to result in the decr
30 nrichment with frozen (13%) and freeze-dried curly kale (3%), the naturally cloudy apple juice was ch
31                                              Curly kale is a robust, cold tolerant plant with a high
32 ice with addition of frozen and freeze-dried curly kale leaves.
33 t activity of beverages with the addition of curly kale ranged from 6.6 to 9.4mumol Trolox/mL.
34               A combination of eol1 with the curly leaf (clf) allele, carrying a mutation in the cata
35 , the repressive histone methyl transferases CURLY LEAF (CLF) and SWINGER (SWN), and the gene silenci
36      The plant Polycomb-group (Pc-G) protein CURLY LEAF (CLF) is required to repress targets such as
37 come by partial loss of polycomb activity in curly leaf (clf) mutants, implicating the SWI2/SNF2 chro
38                                              CURLY LEAF (CLF), an Arabidopsis homolog of enhancer of
39 llele of the Enhancer of Zeste E(z) ortholog CURLY LEAF (CLF), clf-59.
40 at of severe mutations in another Pc-G gene, CURLY LEAF (CLF), suggesting that the two genes may act
41 gated the molecular mechanism underlying the curly leaf and early flowering phenotypes caused by redu
42                    It is shown here that the CURLY LEAF gene of Arabidopsis is necessary for stable r
43 d by the suppression of petal defects of the curly leaf mutant by the pie1 mutation.
44 but defines a locus distinct from phyB, clf (curly leaf), and elf3 (early-flowering 3).
45 redundantly by the E(z) homologs SWINGER and CURLY LEAF.
46 t counteracts the PcG-repressive activity of CURLY LEAF.
47 oss in MIPS1 resulted in smaller plants with curly leaves and spontaneous production of lesions.
48 der plants of the over-expressing lines have curly leaves and were generally smaller in stature.
49 notypes including long and bending petioles, curly leaves, accelerated senescence, and constitutive e
50 s due to the shear-induced alignments of the curly segments of the nanotubes.
51             The two mutant strains examined, curly tail (ct) and splotch-delayed (Pax3Sp-d), develop
52 scued morphological abnormalities, including curly tail and cyst formation in the pronephric kidney,
53  recently identified as a candidate gene for curly tail based on chromosomal location and the occurre
54 PKC expression and applied PKC inhibitors to curly tail embryos developing in culture.
55 ed in the spinal region of neurulation-stage curly tail embryos.
56 revention, we examined cell proliferation in curly tail embryos.
57 is known to lead directly to spina bifida in curly tail embryos.
58 the known cellular defect leading to NTDs in curly tail is unknown.
59 sion of Grhl3, we generated Grhl3-expressing curly tail mice by bacterial artificial chromosome-media
60                                              Curly tail mice do not carry a coding region mutation in
61  to test the hypothesis that spina bifida in curly tail mice results from insufficient expression of
62 tly reducing the incidence of spinal NTDs in curly tail mice, a genetic model of folate-resistant NTD
63 ogenesis and etiology of spina bifida in the curly tail mouse closely resemble defects in humans, pro
64 hl3 expression in causing spinal NTDs in the curly tail mouse model.
65                                           In curly tail mutant mice, there can be a failure of the ne
66                                          The curly tail mutant mouse provides a model of folate-resis
67                       However, the causative curly tail mutation has not been established, while the
68                        Mutant mice display a curly tail phenotype consistent with Zic2 loss-of-functi
69                              Spina bifida in curly tail results from a cell type-specific proliferati
70 s characteristic of defective cilia, such as curly tail up, edema, and abnormal pronephric kidney dev
71 nd is also characterized by transient edema, curly tail, generalized organ hypoplasia, including the
72 ding neural tube defects, a shortened and/or curly tail, no genital tubercle, blind-ended colons, hyd
73 ramatically reduced body length and a short, curly tail.
74 f the pathogenic sequence leading to NTDs in curly tail.
75  klc2 in Danio rerio revealed mild to severe curly-tail phenotype, which is suggestive of a neuromusc
76 s sagrei, and one of its main predators, the curly-tailed lizard Leiocephalus carinatus.
77 tious clones corresponding to CaLCuV or Beet curly top virus (BCTV), which are classified in differen
78  determine if the related L2 protein of Beet curly top virus (BCTV, genus Curtovirus) also has suppre
79 ng the replication origin of the Beet severe curly top virus (BSCTV), a model DNA virus, was created.
80               The related L2 protein of Beet curly top virus (genus Curtovirus) lacks transcription a
81               The related L2 protein of Beet curly top virus (genus Curtovirus) shares the pathogenic
82            Finally, we demonstrate that Beet curly top virus L2- mutant DNA present in tissues that h
83 syringae pv tomato DC3000 bacterium and Beet curly top virus.
84                       The zebrafish mutation curly up (cup) affects the zebrafish ortholog of polycys
85 ture formation in polycystin 2 morphants and curly up polycystin 2 mutants.
86 nt monomers in properdin oligomers conform a curly vertex that holds together the AP convertase, inte
87 ltaCR1, display a phenotype characterized by curly vibrissae and wavy hair.
88 eyelids open and demonstrate a wavy coat and curly vibrissae.
89 ld be easily identified by the phenotypes of curly whiskers and wavy, sheen pelage hair.
90 e are viable and fertile but have wavy hair, curly whiskers, and abnormal hair follicle shape and arr
91 These mice have a mild phenotype (wavy coat, curly whiskers, and runted stature) and normally develop
92 s, such as delayed growth, a waved coat, and curly whiskers, correlated with decreased transforming g
93 e Trpv3 gene also exhibit wavy hair coat and curly whiskers.
94 o a phenotype characterized by wavy hair and curly whiskers.

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