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1 ss of UVR-induced inflammation as a model of cutaneous hypersensitivity.
2 anical deep muscle hypersensitivity, but not cutaneous hypersensitivity.
3 elivered into a milieu of changes induced by cutaneous hypersensitivity.
5 t affecting levels of allergen-specific IgE, cutaneous hypersensitivity and allergen-specific T cell
6 sitization on IgE production, immediate type cutaneous hypersensitivity and development of altered ai
7 ection of anti-OVA IgE resulted in immediate cutaneous hypersensitivity and, after airway challenge w
8 cluding spontaneous unevoked pain and evoked cutaneous hypersensitivity, appear following spinal cord
9 afety and tolerability with the exception of cutaneous hypersensitivity at the 10 and 30 mg/kg doses.
10 eceived the intended treatment had a delayed cutaneous hypersensitivity (DCH) response to the third v
11 T-cell responses, IgE production, immediate cutaneous hypersensitivity (ICH), and increased airway r
12 ferents reduced both ongoing pain and evoked cutaneous hypersensitivity in the context of cystitis, b
15 sting with antigens that elicit delayed-type cutaneous hypersensitivity reactions is commonly used to
16 sporozoites because many individuals develop cutaneous, hypersensitivity reactions to mosquito saliva
17 impaired antibody formation; loss of delayed cutaneous hypersensitivity; reduced immunoglobulin conce
18 To address this question, we studied the cutaneous hypersensitivity response of lymphocyte-defici
21 Nevertheless, both mutants mounted partial cutaneous hypersensitivity responses and normal T cell d
22 investigated the relation between immediate cutaneous hypersensitivity to common aeroallergens and t
24 elivered both to individuals who may exhibit cutaneous hypersensitivity to mosquito bite and to other
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