ライフサイエンスコーパス: cyanotic

1 Twelve patients (75%) displayed cyanotic acral edema of the extremities.

2 Cyanotic acral edema was present in 75% of our patients,

3 proatrial natriuretic peptide (proANP) in 26 cyanotic adults and 28 noncyanotic control subjects.

4 iac surgery and the multisystem disorders of cyanotic adults.

5 surgical repair of TOF from 23 patients: 13 cyanotic and 10 acyanotic.

6 enic factor CCBE1 or VEGFR3 function, appear cyanotic and die shortly after birth due to failure of l

7 Both had erythematous to cyanotic and hyperplastic gingiva.

8 become immobile, breathe irregularly, appear cyanotic, and die within an hour.

9 justed rate ratio, 95% confidence interval): cyanotic CHD (6.44, 3.95-10.50), endocardial cushion def

10 Cyanotic children showed a higher peak troponin-T level

11 is did not influence outcome in infants, but cyanotic children showed worse reperfusion injury and cl

12 Tetralogy of Fallot (TOF) is the most common cyanotic congenital heart defect.

13 Ten patients with cyanotic congenital heart defects and 10 control subject

14 on were upper airway obstruction (13.5%) and cyanotic congenital heart disease (11.5%).

15 Between 1988 and 1995, 162 patients with cyanotic congenital heart disease (mean age 37 years, ra

16 g nonlinear mixed effects model, presence of cyanotic congenital heart disease (odds ratio, 7.35; p <

17 igned infants 92 days of age or younger with cyanotic congenital heart disease and a systemic-to-pulm

18 erebrovascular events in adult patients with cyanotic congenital heart disease and to evaluate any co

19 Adults with cyanotic congenital heart disease are at risk of having

20 Adults with cyanotic congenital heart disease are characterized by i

21 Many adults with cyanotic congenital heart disease are characterized by r

22 he recommendation that routine screening for cyanotic congenital heart disease be added to the panel

23 and BNP are markedly elevated in adults with cyanotic congenital heart disease despite reduced body w

24 ascular events are a serious complication of cyanotic congenital heart disease in infants and childre

25 Tetralogy of Fallot (TOF) is the most common cyanotic congenital heart disease in Taiwan.

26 Pulse oximetry screening for cyanotic congenital heart disease is specific, sensitive

27 Chronic hypoxia (CH) present in infants with cyanotic congenital heart disease may be responsible for

28 Clopidogrel therapy in infants with cyanotic congenital heart disease palliated with a syste

29 Infants with cyanotic congenital heart disease palliated with placeme

30 ble increase in basal coronary blood flow in cyanotic congenital heart disease, flow reserve remains

31 echanism for preservation of flow reserve in cyanotic congenital heart disease.

32 e success story in the surgical treatment of cyanotic congenital heart disease.

33 toward treating microcytosis in adults with cyanotic congenital heart disease.

34 who undergo heart surgery have a congenital cyanotic defect in which the heart is chronically perfus

35 ho undergo cardiac surgery have a congenital cyanotic defect where the heart is chronically perfused

36 Noncyanotic and cyanotic defects were classified as simple and severe, r

37 CHD categories (with septal, obstructive and cyanotic defects).

38 rgery do not take into consideration age and cyanotic differences.

39 Tetralogy of Fallot (TOF) is the commonest cyanotic form of congenital heart disease.

40 In human infants with cyanotic heart defects, PKCepsilon, p38 MAP kinase, and

41 Lack of past cardiac surgery (p = 0.04), cyanotic heart disease (p = 0.03), and early postoperati

42 ffered to patients with a variety of complex cyanotic heart disease at younger ages, and has resulted

43 ry syncytial virus infection, or presence of cyanotic heart disease or residual right-to-left intraca

44 Female sex and complex cyanotic heart disease were significant risk factors for

45 diopathic pulmonary hypertension, congenital cyanotic heart disease, morbid obesity associated with s

46 In patients without cyanotic heart disease, worse daily fluid overload % pre

47 T angiography in the diagnosis of congenital cyanotic heart diseases.

48 decision-making in patients with congenital cyanotic heart diseases.

49 phometric or hemodynamic differences between cyanotic infants with critical PS and asymptomatic infan

50 In such severely ill, cyanotic infants, irregularities, however slight, may ha

51 Women and those with complex cyanotic lesions are at particular risk, as patients may

52 Two children (2/558, 0.4%) had cyanotic lesions.

53 sk is primarily represented by patients with cyanotic or left heart obstructive lesions.

54 Plasma proANP levels were elevated in cyanotic patients compared with control subjects (1828+/

55 here was no difference in oxytocin levels in cyanotic patients compared with control subjects (P=0.49

56 Cyanotic patients had a significantly lower expression o

57 Four cyanotic patients had evidence of iron deficiency.

58 Cyanotic patients had lower mean right atrial pressures

59 Cyanotic patients had reduced body water compared with c

60 tion and hematocrit significantly greater in cyanotic patients than in control subjects (82+/-6 versu

61 old (P=0.02) and 12-fold (P=0.03) greater in cyanotic patients than in control subjects.

62 Cyanotic patients were excluded to avoid confounding fro

63 The cyanotic patients were younger at surgery compared with

64 hest potential for vascular injury: younger, cyanotic patients with longer pump times and longer post

65 2)) slope was not predictive of mortality in cyanotic patients.

66 ein kinases (MAP kinases) in 15 infants with cyanotic (SaO2<85%) or acyanotic (SaO2>95%) heart defect

67 ent animals and found that these mice become cyanotic shortly before death because of lung maturation

68 Caesarean section at embryonic day 18.5 were cyanotic, suffered from respiratory distress, and failed

69 Clinical presentation can range from a cyanotic toe to a diffuse multiorgan systemic disease th