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1 of the effects of intracellular dialysis of cyclic ADP ribose.
2 rolene, but not by ruthenium red, heparin or cyclic ADP-Ribose.
3 from that of inositol-1,4,5-trisphosphate or cyclic ADP-ribose.
4 s dependent on Ca++ mobilization mediated by cyclic ADP-ribose.
5 to that of a global messenger, like IP(3) or cyclic ADP-ribose.
6 of the cyclic ADP ribose antagonists 8-amino-cyclic ADP ribose (10-100 microM) and 8-bromo-cyclic ADP
7 yclic ADP ribose (10-100 microM) and 8-bromo-cyclic ADP ribose (100-1000 microM) reduced this inhibit
11 at catalyzes the synthesis and hydrolysis of cyclic ADP-ribose, a recently identified Ca2+ mobilizing
13 11) but still did in the presence of 8-bromo cyclic ADP-ribose, an antagonist of cyclic ADP-ribose (c
14 ximal inhibition concentrations for 8-amino- cyclic ADP ribose and 8-bromo-cyclic ADP ribose were aro
15 , by inositol-1,4,5-trisphosphate (InsP(3)), cyclic ADP ribose and nicotinic acid adenine dinucleotid
16 joined by two other Ca-releasing messengers, cyclic ADP ribose and nicotinic acid adenine dinucleotid
18 mbinant CD38 catalyzes the formation of both cyclic ADP-ribose and ADP-ribose products from NAD+ and
19 a(2+) messengers by cyclizing NAD to produce cyclic ADP-ribose and exchanging nicotinic acid with the
20 or be metabolized by ecto-enzymes to produce cyclic ADP-ribose and nicotinic acid adenine dinucleotid
22 n CD38 catalyzes the conversion of NAD(+) to cyclic ADP-ribose and to ADP-ribose via a common covalen
23 s the interaction of inositol trisphosphate, cyclic ADP ribose, and nicotinic acid adenine dinucleoti
24 l chemoattractants through its production of cyclic ADP-ribose, and acts as a critical regulator of i
25 ble of producing the potent second messenger cyclic ADP-ribose, and SLO and SPN act synergistically t
27 Application, via the patch pipette, of the cyclic ADP ribose antagonists 8-amino-cyclic ADP ribose
32 A induces production of the second-messenger cyclic ADP ribose (cADPR), which controls release of int
34 volved in gene silencing regulation, and for cyclic ADP ribose (cADPR)-dependent Ca(2+) signaling.
35 cts pulmonary arteries in part by increasing cyclic ADP-ribose (cADPR) accumulation in the smooth mus
37 nodine receptors (RyR), the second messenger cyclic ADP-ribose (cADPR) also accelerates the activity
38 ectoenzyme CD38 catalyzes the production of cyclic ADP-ribose (cADPR) and ADP-ribose (ADPR) from its
39 m nicotinamide adenine dinucleotide (NAD) to cyclic ADP-ribose (cADPR) and from cADPR to ADP-ribose (
41 at the two Ca2+-mobilizing second messengers cyclic ADP-ribose (cADPR) and nicotinic acid adenine din
42 olved in metabolizing two Ca(2+) messengers, cyclic ADP-ribose (cADPR) and nicotinic acid adenine din
43 sis and metabolism of two Ca(2+) messengers, cyclic ADP-ribose (cADPR) and nicotinic acid adenine din
46 d adenine dinucleotide phosphate (NAADP) and cyclic ADP-ribose (cADPR) are Ca(2+)-mobilizing messenge
48 onal ectoenzyme, catalyzing the synthesis of cyclic ADP-ribose (cADPR) from NAD+ and the hydrolysis o
52 s potent as inositol trisphosphate (IP3) and cyclic ADP-ribose (cADPR) in mobilizing intracellular Ca
61 8-bromo cyclic ADP-ribose, an antagonist of cyclic ADP-ribose (cADPR) signaling at RyR (S2/S1=0.48+/
62 bose (8Br-cADPr), a competitive inhibitor of cyclic ADP-ribose (cADPr) signaling that partially relie
65 hat catalyze the synthesis and hydrolysis of cyclic ADP-ribose (cADPR), a Ca(2+) messenger molecule r
70 CD38, an ectoenzyme that converts NAD(+) to cyclic ADP-ribose (cADPr), may play a role in cytokine-i
71 ysiological processes that produce peroxide, cyclic ADP-ribose (cADPR), nicotinamide adenine dinucleo
72 ecently discovered Ca2+-releasing messenger, cyclic ADP-ribose (cADPR), which activates ryanodine rec
73 receptor is the nucleotide second messenger cyclic ADP-ribose (cADPR), which is generated by an ecto
84 gnaling mechanisms in each cell type, namely cyclic ADP-ribose-dependent Ca2+ mobilization from the s
86 aging showed that following the Ca2+ influx, cyclic ADP-ribose enhanced the spatial spread of the Ca2
88 d hepatocytes showed that both ryanodine and cyclic ADP-ribose evoked a slow Ca2+ leak from intracell
91 These powerful actions suggest a role for cyclic ADP-ribose in the functional coupling of neuronal
95 es at the single channel level indicate that cyclic ADP ribose may not act directly on the M-channels
96 ctoenzyme that produces the paracrine factor cyclic ADP ribose-mediates the effects of calorie restri
97 that inositol 1,4,5-trisphosphate (IP3) and cyclic ADP-ribose mobilize Ca2+ from the sarcoplasmic re
99 sphosphate, intracellular messengers include cyclic ADP ribose, nicotinic acid adenine dinucleotide p
101 are different from those activated by either cyclic ADP-ribose or inositol 1,4,5-trisphosphate (IP3).
108 croM ryanodine, which suggests that the CD38/cyclic ADP-ribose/RyR pathway is not a primary mechanism
111 er, unlike previously established effects of cyclic ADP ribose, the ryanodine receptor is not require
115 s were also induced by cyclic GMP (cGMP) and cyclic ADP-ribose, two molecules that can serve as secon
117 s for 8-amino- cyclic ADP ribose and 8-bromo-cyclic ADP ribose were around 40 microM and 1 mM, respec
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