ライフサイエンスコーパス: cyclic adenosine 3',5'-monophosphate

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1 , enabling a comparison of the mechanisms of cyclic adenosine 3',5'-monophosphate biosynthesis.

2 Regulation of intracellular cyclic adenosine 3 ',5 '-monophosphate (cAMP) is integra

3 Cyclic adenosine 3', 5'-monophosphate (cAMP) is a ubiqui

4 An elevation of intracellular cyclic adenosine 3', 5'-monophosphate (cAMP) is known to

5 I-LTD requires cyclic adenosine 3', 5'-monophosphate (cAMP)-dependent p

6 4B (PDE4B) inactivates the second messenger cyclic adenosine 3',5' monophosphate (cAMP) and abrogate

7 of all SCF deletion constructs treated with cyclic adenosine 3',5'-monophosphate (cAMP) and forskoli

8 the hypothesis that increased intracellular cyclic adenosine 3',5'-monophosphate (cAMP) could inhibi

9 Increased cyclic adenosine 3',5'-monophosphate (cAMP) has been pre

10 Experiments were performed with cyclic adenosine 3',5'-monophosphate (cAMP) in the pipet

11 Cyclic adenosine 3',5'-monophosphate (cAMP) is a recogni

12 iocyte proliferation and secretin-stimulated cyclic adenosine 3',5'-monophosphate (cAMP) levels.

13 secretin receptor (SR) gene expression, and cyclic adenosine 3',5'-monophosphate (cAMP) levels.

14 or (SR) gene expression and secretin-induced cyclic adenosine 3',5'-monophosphate (cAMP) synthesis an

15 ellular development is organized by periodic cyclic adenosine 3',5'-monophosphate (cAMP) waves, which

16 PDE3A inhibition generates compartmentalized cyclic adenosine 3',5'-monophosphate (cAMP), which furth

17 ical level, CC2D1A transduces signals to the cyclic adenosine 3',5'-monophosphate (cAMP)-protein kina

18 ocess induced by bicarbonate and mediated by cyclic adenosine 3',5'-monophosphate (cAMP).

19 ctivation of parallel fibres (PFs) induces a cyclic adenosine 3'5'-monophosphate (cAMP) and protein k

20 tion in alpha1A-transfected cells, inhibited cyclic adenosine 3'5'-monophosphate (cAMP) formation in

21 PKC agonists induced mkp1 mRNA levels, only cyclic adenosine 3':5'-monophosphate (cAMP)-PKA inhibiti

22 Cyclic adenosine 3, 5-monophosphate (cAMP) levels were m

23 We have examined the cytotoxic effects of cyclic adenosine-3', 5'-monophosphate (cAMP) derivatives

24 ace of intact cells revealed the presence of cyclic adenosine 3':5'-monophosphate-(cAMP) dependent pr

25 cting with secretin receptor (SR) activating cyclic adenosine 3',5'-monophosphate/cystic fibrosis tra

26 Similarly, in mammalian cells the cyclic adenosine 3',5'-monophosphate-dependent protein k

27 Agonists that stimulate cyclic adenosine 3',5'-monophosphate production led to a

28 receptor activation to adenylyl cyclase and cyclic adenosine 3'-5' monophosphate production and the

29 The cyclic adenosine 3',5'-monophosphate receptor protein (C

30 e of protein kinase A (PKA) and dopamine and cyclic adenosine 3',5' monophosphate-regulated phosphopr

31 ian transcription factors-nuclear receptors, cyclic adenosine 3',5'-monophosphate-regulated enhancer

32 in kinase, mitogen-activated protein kinase, cyclic adenosine 3',5'-monophosphate response element bi

33 modulin-dependent protein kinase (CaMK), and cyclic adenosine 3',5'-monophosphate-responsive element-

34 or CaMK kinase in mediating mGluR5-dependent cyclic adenosine 3',5'-monophosphate-responsive element-

35 2+)/calmodulin-dependent protein kinase, and cyclic adenosine 3',5'-monophosphate-responsive element-

36 te after bile duct ligation by activation of cyclic adenosine 3', 5'-monophosphate signaling.

37 The cyclic adenosine 3',5'-monophosphate signalling pathway

38 ts result in the sustained activation of the cyclic adenosine 3',5'-monophosphate system in specific

39 The activated cyclic adenosine 3',5'-monophosphate system leads to the

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