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1 autoantibodies (rheumatoid factor [RF], anti-cyclic citrullinated peptide 2 [anti-CCP-2], and RF isot
2 llinated alpha-enolase peptide 1 (CEP-1) and cyclic citrullinated peptide 2 were measured by enzyme-l
3 reduced titers of rheumatoid factor and anti-cyclic citrullinated peptide Abs are recorded, the mecha
4 tivity Score 28, rheumatic factor [RF], anti-cyclic citrullinated peptide [aCCP], medications) were e
5 factor STAT3, of antibodies directed against cyclic citrullinated peptides and Bhsp65, and of the act
6 IgA, and IgG rheumatoid factors (RFs), anti-cyclic citrullinated peptide, and antimicrobial antibodi
7 nts were HLA-DRB1 typed, and tested for anti-cyclic citrullinated peptide (anti-CCP) and rheumatoid f
8 two biomarkers related to the disease, anti-cyclic citrullinated peptide (anti-CCP) and rheumatoid f
10 In patients with RA, higher levels of anti-cyclic citrullinated peptide (anti-CCP) antibodies and c
11 In addition to measurement of serum anti-cyclic citrullinated peptide (anti-CCP) antibodies and H
13 and had significantly higher titers of anti-cyclic citrullinated peptide (anti-CCP) antibodies compa
15 actor (IgM-RF), IgG-RF, IgA-RF, and IgG anti-cyclic citrullinated peptide (anti-CCP) antibodies toget
16 ed for rheumatoid factor (RF) isotypes, anti-cyclic citrullinated peptide (anti-CCP) antibodies, 14 c
17 sensitivity C-reactive protein (hsCRP), anti-cyclic citrullinated peptide (anti-CCP) antibodies, inte
18 ent subsets, defined according to their anti-cyclic citrullinated peptide (anti-CCP) antibody status,
19 int examination, rheumatoid factor, and anti-cyclic citrullinated peptide (anti-CCP) antibody testing
20 e impact of disease activity, sex, age, anti-cyclic citrullinated peptide (anti-CCP) antibody titer,
21 or the presence of anti-PAD-4 antibody, anti-cyclic citrullinated peptide (anti-CCP) antibody, and rh
23 e interactions between SE, smoking, and anti-cyclic citrullinated peptide (anti-CCP) status, adjusted
25 heumatoid factors (RF), and of antibodies to cyclic citrullinated peptide (anti-CCP) were assayed.
28 ere tested for the presence of antibodies to cyclic citrullinated peptide (anti-CCP), rheumatoid fact
29 ies that investigated second-generation anti-cyclic citrullinated peptide antibodies (anti-CCP2) in p
30 mmatory arthritis but were positive for anti-cyclic citrullinated peptide antibodies and/or >/=2 rheu
31 m measurements of rheumatoid factor and anti-cyclic citrullinated peptide antibodies, second-generati
32 in antibody (ACPA; by second-generation anti-cyclic citrullinated peptide antibody enzyme-linked immu
33 nalysis Workshop 16 for associated with Anti-cyclic citrullinated peptide antibody further demonstrat
34 variable model, adjusting for age, sex, anti-cyclic citrullinated peptide antibody positivity, and th
35 luding a positive rheumatoid factor, or anti-cyclic citrullinated peptide antibody result, or both.
36 RA in 3 independent seropositive (RF or anti-cyclic citrullinated peptide antibody) RA cohorts but no
38 atoid arthritis (RA), especially more severe cyclic-citrullinated peptide antibody-positive (anti-CCP
39 ients with RA who were positive for the anti-cyclic citrullinated peptide (CCP) antibodies were more
40 atoid factor (RF) and autoantibodies against cyclic citrullinated peptide (CCP) are markers that migh
43 ociated with RA (P<10(-4)): *0301 with anti- cyclic citrullinated peptide-negative RA and *0701 indep
45 No association with rheumatoid factor, anti-cyclic citrullinated peptide, or the Disease Activity Sc
46 odies that can recognize immunoglobulins and cyclic citrullinated peptides, suggesting early defects
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