ライフサイエンスコーパス: cyclin-dependent kinase 9

1 putative GRIP1 kinases, casein kinase 2 and cyclin-dependent kinase 9.

2 anscription factor b, P-TEFb, is composed of cyclin-dependent kinase 9 and a regulatory cyclin (T1/T2

3 on elongation factor b (P-TEFb), composed of cyclin-dependent kinase 9 and cyclin T1 or T2, is requir

4 on elongation factor b (P-TEFb), composed of cyclin-dependent kinase 9 and cyclin T1, stimulates the

5 general transcription factor IIB (TFIIB) and cyclin-dependent kinase 9 are upregulated during hypertr

6 factor b (P-TEFb), a complex containing the cyclin-dependent kinase 9 (CDK-9) and cyclin T1 subunits

7 Consisting of cyclin dependent kinase 9 (Cdk9) and cyclin T1, P-TEFb p

8 ranscriptional elongation complex containing cyclin dependent kinase-9 (CDK9; a kinase necessary for

9 Cyclin-dependent kinase 9 (CDK9) and CDK12 have each bee

10 on elongation factor b (P-TEFb), composed of cyclin-dependent kinase 9 (CDK9) and cyclin T, is a glob

11 ion factor b (P-TEFb) complexes, composed of cyclin-dependent kinase 9 (CDK9) and cyclin T1 or T2, ar

12 n elongation factor b (P-TEFb), comprised of cyclin-dependent kinase 9 (CDK9) and cyclins T1 (CycT1)

13 This important factor is a heterodimer of cyclin-dependent kinase 9 (Cdk9) and one of four cyclin

14 Cyclin-dependent kinase 9 (CDK9) and serine-2-phosphoryl

15 defined Myc-driven HCC model, we identified cyclin-dependent kinase 9 (Cdk9) as required for disease

16 ICP22 binds the cyclin-dependent kinase 9 (cdk9) but not cdk7, and this

17 infection enhances the activated fraction of cyclin-dependent kinase 9 (CDK9) by promoting its associ

18 is thought to require the kinase activity of cyclin-dependent kinase 9 (CDK9) for the phosphorylation

19 lated cyclins T1, T2a, and T2b interact with cyclin-dependent kinase 9 (CDK9) forming multiple nuclea

20 The cyclin-dependent kinase 9 (Cdk9) in P-TEFb then phospory

21 In particular, we focused on the cyclin-dependent kinase 9 (CDK9) inhibitor, FIT-039, whi

22 Cyclin-dependent kinase 9 (cdk9) is a multifunctional ki

23 we examine the role of the Tat/TAR-specified cyclin-dependent kinase 9 (CDK9) kinase activity in regu

24 f cyclin T1 and concomitant stabilization of cyclin-dependent kinase 9 (CDK9) may facilitate producti

25 t of an IL-6-inducible complex of STAT3 with cyclin-dependent kinase 9 (CDK9) on gamma-FBG expression

26 Mechanistically, BRD4 is required for cyclin-dependent kinase 9 (CDK9) recruitment and phospho

27 HMBA triggered cyclin-dependent kinase 9 (CDK9) recruitment to the LTR,

28 ress responses by regulating the activity of cyclin-dependent kinase 9 (CDK9), a protein required for

29 a heterodimer of a cyclin-dependent kinase, cyclin-dependent kinase 9 (Cdk9), and one of four cyclin

30 ofactors of HIV-1 Tat, cyclin T1 (CycT1) and cyclin-dependent kinase 9 (CDK9), are required for LTR-d

31 ding Ras GTPase-activating protein (RasGAP), cyclin-dependent kinase 9 (Cdk9), fibronectin, and Ras h

32 factor b (P-TEFb), containing cyclin T1 and cyclin-dependent kinase 9 (CDK9), interacts with the hum

33 several components of transcription such as cyclin-dependent kinase 9 (cdk9), localize at these site

34 and the enhancer regions, and inhibition of cyclin-dependent kinase 9 (CDK9), that regulates these e

35 Cyclin-dependent kinase 9 (CDK9), the catalytic componen

36 We found that cyclin-dependent kinase 9 (Cdk9), the catalytic subunit

37 on and led to higher enzymatic activities of cyclin-dependent kinase 9 (CDK9), which serves as a tran

38 omain containing 4 (BRD4) with NF-kappaB and cyclin-dependent kinase 9 (CDK9).

39 ive to flavopiridol, a specific inhibitor of cyclin-dependent kinase 9 (Cdk9).

40 complex proteins, bromodomain 4 (Brd4), and cyclin-dependent kinase 9 (CDK9).

41 TEFb, a complex containing cyclin T1 and the cyclin-dependent kinase 9 (Cdk9).

42 or b (P-TEFb) contains cyclin T1 (CycT1) and cyclin-dependent kinase 9 (Cdk9).

43 in T and the previously cloned small subunit cyclin-dependent kinase 9 (CDK9).

44 tivated by HIV-1 Tat protein, which recruits cyclin-dependent kinase 9 (CDK9)/cyclin T1 and other hos

45 ster orthologs of the vertebrate PITSLRE and cyclin-dependent kinase-9 (CDK9) kinases, as Hh regulato

46 horylated at an N-terminal serine cluster by cyclin-dependent kinase-9 (CDK9), which is recruited int

47 longation factor b (P-TEFb), consisting of a cyclin-dependent kinase 9-cyclin T heterodimer, stimulat

48 II transcriptional elongation factor P-TEFb (cyclin-dependent kinase 9/cyclin T) is a cellular protei

49 Cyclin-dependent kinase 9/cyclin T, the protein kinase h

50 y, we found that Thr 4 was phosphorylated by cyclin-dependent kinase 9 in cells and dephosphorylated

51 pression of HIV-1 transcription by selective cyclin-dependent kinase-9 inhibitors may be a useful the

52 roscovitine, newly identified inhibitors of cyclin-dependent kinase-9, markedly decrease HIV-1 promo

53 e inhibitors of the transcription regulating cyclin-dependent kinase 9 on the development and progres

54 either by expression of a dominant-negative cyclin-dependent kinase 9 transgene or through the use o