ライフサイエンスコーパス: cyclin-dependent kinase inhibitor p16

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1 had increased levels of APA-1 as well as the cyclin-dependent kinase inhibitor p16.

2 s the "guardian of the genome," p53, and the cyclin-dependent kinase inhibitor p16.

3 d NF-kappaB activation (CARD10, CARD14), and cyclin-dependent kinase inhibitors (p16 and p18).

4 Recently, the genes for the cyclin-dependent kinase inhibitors p16 and p15 have been

5 ermore, the growth-suppressive activities of cyclin-dependent kinase inhibitors p16 and p21 were effi

6 (stasis) associated with elevated levels of cyclin-dependent kinase inhibitors p16 and/or p21.

7 r Cdk4 levels, and reduced expression of the cyclin-dependent kinase inhibitor p16/CDKN2.

8 argeting the CDKN2A locus, which encodes the cyclin-dependent kinase inhibitor p16, decreased CDKN2A

9 The tumor suppressor gene encoding the cyclin-dependent kinase inhibitor p16 has, remarkably, b

10 The cyclin-dependent kinase inhibitor p16(INK4a) (hereafter

11 -->S cell cycle progression by targeting the cyclin-dependent kinase inhibitor p16(INK4a) (p16).

12 on of the Ink4a/Arf locus, which encodes the cyclin-dependent kinase inhibitor p16(INK4a) and tumor s

13 Mutations or deletions in the cyclin-dependent kinase inhibitor p16(INK4A) are associa

14 Cyclin-dependent kinase inhibitor p16(INK4a) is the foun

15 inal truncation mutant or with the unrelated cyclin-dependent kinase inhibitor p16(INK4a), although a

16 e CDKN2A (p16/MTS1/INK4A), which encodes the cyclin-dependent kinase inhibitor p16(INK4a), is a targe

17 -independent senescent state mediated by the cyclin-dependent kinase inhibitor p16(INK4a).

18 ctivity is inhibited when it is bound to the cyclin-dependent kinase inhibitor, p16(INK4A).

19 Cyclin-dependent kinase inhibitors p16(INK4a) and p15(IN

20 lls isolated from INK4a-/- mice, lacking the cyclin-dependent kinase inhibitors p16(INK4a) and p19(AR

21 that G(1)/S arrest by overexpression of the cyclin-dependent kinase inhibitors p16(INK4a), p21(Cip1)

22 hese results suggest that high levels of the cyclin-dependent kinase inhibitor p16 mediate senescence

23 r, androgen down-regulated expression of the cyclin-dependent kinase inhibitor p16 (MTS1, CDKN2) gene

24 lving CDKN2A (the genetic locus encoding the cyclin-dependent kinase inhibitor p16(NK4a)) is a mechan

25 arrest did not depend on the function of the cyclin-dependent kinase inhibitors p16 or p21 because ME

26 OGF treatment increased cyclin-dependent kinase inhibitor p16 protein expression

27 MIS up-regulated the cyclin-dependent kinase inhibitor p16 through an MIS typ

28 genic transformation of cells can induce the cyclin-dependent kinase inhibitor, p16, which leads to h

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