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1 n of alcohols were studied on the example of cyclohexanol.
2 he enantiopure (1S,2S)-2-(1-H-imidazol-1-yl)-cyclohexanol.
3 olvent consisting of 70% 1-dodecanol and 30% cyclohexanol.
4 ot by the addition of an equimolar amount of cyclohexanol.
5 hat is observed at optimal concentrations of cyclohexanol.
6 thylheptyl)phenyl]-trans-4-(3-hydroxyprop yl)cyclohexanol].
7 he oxidation of cyclohexane to cyclohexanone/cyclohexanol (100 degrees C, conversion: 17.7%) is super
8 thylheptyl)phenyl]-trans-4-(3-hydroxyp ropyl)cyclohexanol ([3H]CP55,940) in a concentration-dependent
9 ol (AH5183; (-)-trans-2-[4-phenylpiperidino] cyclohexanol), a drug which blocks the refilling of syna
10 ,1-dimethylheptyl)phenyl]-4-(3-hydroxypropyl)cyclohexanol], a potent cannabinoid that binds with simi
11 functionalized stereoselectively to provide cyclohexanols after oxidation of the carbon-silicon bond
12 l methacrylate in the presence of mixture of cyclohexanol and 1-dodecanol as a porogenic solvent.
13 ith formaldehyde or methane, and on ethanol, cyclohexanol and 1-hydroxymethyl-tetrahydropyran are pre
14 ties >95% are obtained with trans-2-phenyl-1-cyclohexanol and 2,2-diphenylcyclopentanol vinyl ethers.
16 solid-acid-catalyzed phenol alkylation with cyclohexanol and cyclohexene in the apolar solvent decal
17 ignificant migration of the hydroxy group in cyclohexanol and the double bond in cyclohexene with res
18 aturation kinetics for oxidation of ethanol, cyclohexanol, and 1-butanol are quantitatively explained
20 e HBEA catalyse aqueous phase dehydration of cyclohexanol at a rate significantly higher than hydroni
21 ot caused by the limited access of phenol to cyclohexanol, but is due to the absence of a reactive el
22 iation of NADH from the abortive enzyme-NADH-cyclohexanol complex than from the enzyme-NADH complex.
24 yoxy-methyl-1-cyclo-hexanone to give a vinyl cyclohexanol derivative and (2) a highly stereoselective
25 Thus, it is demonstrated that protonated cyclohexanol dimers dehydrate without the formation of a
26 y accounts for the initial rates of 1-(13) C-cyclohexanol disappearance and the appearance of the dif
29 acetophenone from 1-phenylethanol oxidation, cyclohexanol from cyclohexane hydroxylation, and cyclohe
30 ethyl-heptyl)-2,6-dimethoxy-phenyl]-3-methyl-cyclohexanol), greatly attenuated leukocyte adhesion in
31 Transient kinetics experiments show that cyclohexanol inhibition is due to a slower rate of disso
32 r condensation, one chiral separation of the cyclohexanol intermediate, an ether formation using a tr
35 exemplified by intramolecular dehydration of cyclohexanol, is markedly influenced by steric constrain
37 occurs with myo-inositol-d-galactopyranose, cyclohexanol, mannitol, or glycerol as acyl acceptor.
38 echanism; for both cases, the dehydration of cyclohexanol occurs via an E1 mechanism with the cleavag
39 C spectra show that dehydration of 1-(13) C-cyclohexanol occurs with significant migration of the hy
40 ic and mechanistic study of the reactions of cyclohexanol on zeolite HBEA in 130 degrees C water.
46 sandwiched between the two methylenes in the cyclohexanol ring and the hydroxyl group of ethanol hydr
50 netobacter sp. strain SE19, and oxidation of cyclohexanol to adipic acid was demonstrated in recombin
53 trace [(3)H]-(-)-trans-2-(4-phenylpiperidino)cyclohexanol (vesamicol) with ACh, and Michaelis-Menten
54 potent anticholinergic 2-(4-phenylpiperidino)cyclohexanol (vesamicol, 1) in which the cyclohexyl frag
55 ine transporter ligand 2-(4-phenylpiperidino)cyclohexanol (vesamicol, 1), 22 N-hydroxy(phenyl)alkyl d
56 value up to 98%) synthesis of trisubstituted cyclohexanols was achieved by using a tandem Henry--Mich
59 ectly produces trans-2-(dimethylphenylsilyl)-cyclohexanol, whereas the less favored boat-like transit
60 )-vesamicol [(-)-trans-2-(4-phenylpiperidino)cyclohexanol], which binds tightly to an allosteric site
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