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1 s and IOL power calculations attributable to cycloplegia.
2 ne-induced accommodation or atropine-induced cycloplegia.
3 to a more myopic state and with atropine by cycloplegia.
4 e same autorefractor with the subjects under cycloplegia.
5 ith the same autorefractor in subjects under cycloplegia.
6 , LT, and axial length both before and after cycloplegia.
7 d vitreous chamber depth were measured after cycloplegia.
8 orthoptist using the PR2000 without inducing cycloplegia.
9 metropic refractive errors when used without cycloplegia.
12 on has been shown to cause mydriasis without cycloplegia and to increase the rate of aqueous humor fl
13 Vision, open-field autorefraction (without cycloplegia), and ocular biometry were measured in each
14 s and Measures: Visual acuity, refraction in cycloplegia, and manifest strabismus were evaluated and
15 can affect how ocular parameters respond to cycloplegia, and therefore intraocular lens (IOL) power
16 All children then underwent retinoscopy with cycloplegia by an examiner who was unaware of the result
17 tive errors by using videorefraction without cycloplegia could effectively serve as a first stage of
28 easurements of retinoscopic refraction under cycloplegia were taken at 4- to 6-month intervals up to
29 yopia, as determined by autorefraction after cycloplegia with 2 drops of 1% tropicamide at each annua
33 was determined by subjective methods before cycloplegia, with noncycloplegic autorefraction values a
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