ライフサイエンスコーパス: cylindromatosis

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1 complex (LUBAC) and the deubiquitinase CYLD (cylindromatosis).

2 ination by the deubiquitinases A20 and CYLD (cylindromatosis).

3 und to TRAF6 and the deubiquitinating enzyme cylindromatosis.

4 tumor suppressor that is mutated in familial cylindromatosis.

5 susceptibility, incontinentia pigmenti, and cylindromatosis.

6 25 tumours from 4 individuals with familial cylindromatosis.

7 that should be considered in the context of cylindromatosis.

8 me that is altered in patients with familial cylindromatosis, a condition characterized by numerous b

9 a tumour suppressor gene mutated in familial cylindromatosis, a genetic disorder leading to the devel

10 a tumor suppressor gene mutated in familial cylindromatosis, an autosomal dominant predisposition to

11 tumor suppressor that is mutated in familial cylindromatosis, an autosomal dominant predisposition to

12 recruitment of the deubiquitinating enzyme, cylindromatosis, an inhibitor of NF-kappaB activity.

13 which causes a benign human syndrome called cylindromatosis, as a key negative regulator for NF-kapp

14 Interestingly, four other genes, cylindromatosis, CD9, activating transcription factor 3

15 Recruitment of cylindromatosis coincided with reduced TNFR-associated f

16 und for the first time that tumor suppressor cylindromatosis (CYLD) acts as a negative regulator for

17 tudies that identify deubiquitinating enzyme cylindromatosis (CYLD) as a key regulator for T cell dev

18 Mutations in the cylindromatosis (CYLD) gene cause benign tumors of skin

19 Cylindromatosis (CYLD) is a deubiquitinating enzyme that

20 In addition, the deubiquitination enzyme Cylindromatosis (CYLD) negatively regulates the activity

21 The molecular mechanisms mediating cylindromatosis (CYLD) tumor suppressor function appear

22 in a backcross panel of mice, we identified cylindromatosis (CYLD), a deubiquitinase known to act di

23 Here we show that cylindromatosis (CYLD), a tumor suppressor gene and nega

24 echanism in which the tumor-suppressor gene, cylindromatosis (CYLD), confers protection from hepatoce

25 enes including the tumor suppressor familial cylindromatosis (CYLD), which was found to be a direct t

26 on of TLR2 signaling by the tumor suppressor cylindromatosis (CYLD).

27 nduction, and silencing of miR-181b-1 target cylindromatosis (CYLD).

28 associated with the hypertrophic skin tumor cylindromatosis, disrupt the USP domain, accounting for

29 d CYLD by detecting germline mutations in 21 cylindromatosis families and somatic mutations in 1 spor

30 Brooke-Spiegler syndrome (BSS), familial cylindromatosis (FC), and multiple familial trichoepithe

31 Hereditary cylindromatosis is a rare autosomal dominant disease cha

32 Familial cylindromatosis is an autosomal dominant genetic predisp

33 Brooke-Spiegler syndrome (BSS, familial cylindromatosis or turban tumor syndrome) is an inherite

34 ed to mimic the smallest truncation found in cylindromatosis patients, CYLD interaction with CAP350 i

35 rt here that silencing of the deubiquitinase cylindromatosis protein (CYLD), increases HIV infection

36 ing the Lys63-deubiquitinating enzyme (DUB), cylindromatosis tumor suppressor (CYLD).

37 In contrast to the cylindromatosis tumor suppressor CYLD, which attenuates

38 In contrast, CYLD (cylindromatosis tumor-suppressor protein), a K63-specifi

39 These researchers found that the familial cylindromatosis tumour suppressor gene (CYLD), previousl

40 denced a new locus in the 16q12 region (near cylindromatosis turban tumor syndrome gene [CYLD]) and c

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