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1 normal ovarian surface epithelium and benign cystadenoma.
2 as, endolymphatic sac tumors, and epididymal cystadenomas.
3 ignant potential and carcinomas but not with cystadenomas.
4 ents underwent surgery for 14 benign (serous cystadenomas = 10, Hamoudi = 2, lymphoepithelial = 1, si
5 denoma 9, borderline 9, cancer 4), 23 serous cystadenomas, 13 other pancreatic cysts, 27 pancreatic e
7 oup were IPMN without invasive cancer (30%), cystadenoma (17%), and pancreatic ductal adenocarcinoma
8 hronic pancreatitis (24%), benign pancreatic cystadenoma (22%), pancreatic adenocarcinoma (18%), neur
9 lasms of the pancreas, 54 were classified as cystadenomas, 23 as noninvasive proliferative cystic muc
10 y 100% incidence of multiple bilateral renal cystadenomas, 50% incidence of liver hemangiomas, and 32
14 of BRAF and KRAS occur in the epithelium of cystadenomas adjacent to SBTs and strongly suggest that
16 ovarian cancers compared with benign ovarian cystadenomas and that gankyrin regulates FSH upregulatio
17 seven were confirmed benign at surgery (four cystadenomas and three inflammatory), one had a benign p
18 Nineteen patients with intrahepatic biliary cystadenomas and two patients with biliary cystadenocarc
20 d in both the cell lines and Tsc mouse renal cystadenomas, and ectopic expression of PDGFRbeta in Tsc
23 h many cysts, such as pseudocysts and serous cystadenomas, are benign and can be monitored clinically
24 the first documentation of a DALM (mucinous cystadenoma) arising in the appendix in the setting of U
26 re we show that a subset of cultured ovarian cystadenoma cells expressing SV40 large T-antigen, which
28 alized ovarian surface epithelia and ovarian cystadenoma cells showed much higher similarity to prima
33 detected in SBTs were also identified in the cystadenoma epithelium adjacent to the SBTs in six (86%)
35 es of histopathologically proved paraovarian cystadenomas from January 1993 through December 1996 in
36 y genetic background, with fewer large renal cystadenomas in the outbred Black Swiss background and m
37 atients had pathologically confirmed biliary cystadenomas, including one with a biliary cystadenocarc
39 lecular genetic studies suggest that ovarian cystadenomas, low malignant potential tumors, and carcin
44 papillary mucinous neoplasms (n=15), serous cystadenomas (n=12), or pseudocysts (n=9), with confirma
46 total of 106 patients presenting with serous cystadenoma of the pancreas from 1976-2004 were identifi
47 ine whether surgical enucleation of mucinous cystadenoma of the pancreas is a safe and adequate opera
51 rience suggests that enucleation of mucinous cystadenomas of the pancreas can be performed safely and
52 ween January 1990 and June 1997, 36 mucinous cystadenomas of the pancreas were resected at The Johns
54 reviewed to confirm the diagnosis of biliary cystadenoma or biliary cystadenocarcinoma by 2 GI pathol
56 findings of surgically excised lesions were cystadenoma or cystadenofibroma (n = 14; 11 serous, thre
58 eoplasms) from benign cystic lesions (serous cystadenomas+pseudocysts) with a 78% sensitivity at 80%
59 ors, we recommend resection for large serous cystadenomas regardless of the presence or absence of sy
61 eveloped a novel pancreatic neoplasm, serous cystadenoma (SCA), presenting as large epithelial tumors
62 h of the major neoplastic cyst types: serous cystadenomas (SCAs), intraductal papillary mucinous neop
65 neoplasms, mucinous cystic neoplasms, serous cystadenomas, solid pseudopapillary neoplasms, cystic va
68 ation about the imaging findings for biliary cystadenoma to reduce the demonstrated delay in appropri
71 are potentially malignant and that mucinous cystadenomas, when completely removed, are biologically
72 DLX4 was not detected in normal ovary and cystadenomas, whereas its expression in ovarian carcinom
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