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1  duct (CHD) and in 0.8% of subjects into the cystic duct.
2 intestine, pancreas, liver, gallbladder, and cystic duct.
3  min was insufficient to identify all patent cystic ducts.
4 the secondary transitions is associated with cystic ducts.
5 opically placed clips were used for both the cystic duct and artery, thus, obviating the need for any
6 xic bile acids into the duodenum through the cystic duct and common bile duct system.
7 ept that motor disorders of the gallbladder, cystic duct and sphincter of Oddi can cause painful synd
8 gh numbers in hepato-pancreatic common duct, cystic duct, and hilum.
9           Network formation begins where the cystic duct combines with hepatic ducts to form the comm
10 ponse in which the sphincter of Oddi and the cystic duct contract (and impede bile flow) instead of u
11 covered metallic stent would not overlap the cystic duct, cSEMS were not inferior to multiple plastic
12 e, 14.6; 95% confidence interval, 7.8-21.3); cystic duct fistula, 1.7% and 0%; cholangitis, .9% and .
13              Stents placed through the donor cystic duct have been used to attempt to reduce tube-rel
14 yos, together with the appearance of ectopic cystic duct-like epithelia in their gallbladders.
15 phine augmentation improved the detection of cystic duct patency compared with CCK pretreatment only.
16  on whether they occurred above or below the cystic duct, respectively.
17                                  CCK-induced cystic duct spasm is the cause of low GBEF in CAC and CC
18                                  CCK-induced cystic duct spasm is the etiology for low EF in both CAC
19 m, at the distal end of the common duct, and cystic duct syndrome, in the gallbladder.
20 incter of Oddi spasm and cholecystectomy for cystic duct syndrome.
21  common duct, erroneously believed to be the cystic duct, was deliberately cut.
22 those with an intact gallbladder in whom the cystic duct would be overlapped by a cSEMS were excluded

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