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1 d by electron redistribution between CuA and cytochrome a.
3 od for determining the cytochrome c oxidase, cytochrome a, a3, content in tissue and mitochondria sam
6 port resulted in depletion of electrons from cytochrome a,a3 during ischemia and early reperfusion, w
8 entricular developed pressure (LVDP) and the cytochrome a,a3 redox state (near infrared spectroscopy)
9 istration of succinate infusion restores the cytochrome a,a3 redox state balance and myocardial funct
10 es in tissue oxygen stores and mitochondrial cytochrome a,a3 redox state in rhythmically contracting
11 gnificant correlations between mitochondrial cytochrome a,a3 redox state, cardiac output, and oxygen
13 yoglobin oxygen saturation, cytochrome c and cytochrome a/a3 redox state, and ventricular pressure we
14 rapidly with cytochrome c oxidase to reduce cytochrome a and CuA and to form a charge-transfer compl
16 cytochrome c oxidase activity and assembled cytochromes a and a3, but the other respiratory chain ca
22 lowed by rapid electron transfer from CuA to cytochrome a to produce F(II) [a3(4+)=O CuB2+-OH- a2+ Cu
23 rusticyanin and cytochrome c, respectively; cytochrome a was present as one molecule for every 91 nm
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