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1 This tendency is substantially suppressed by cytochrome oxidase.
2 per metallation of the Cu(A) site in Cox2 of cytochrome oxidase.
3 electrostatically near CuA in subunit II of cytochrome oxidase.
4 ts I and II in the assembly of mitochondrial cytochrome oxidase.
5 c content, and the rate of oxidation through cytochrome oxidase.
6 t and a diminished rate of oxidation through cytochrome oxidase.
7 competing with O(2) for its binding site on cytochrome oxidase.
8 effect of mutations in the catalytic core of cytochrome oxidase.
9 inhibit the essential mitochondrial enzyme, cytochrome oxidase.
10 ear CuA binding region of N(2)O reductase or cytochrome oxidase.
11 talysts are comparable to those reported for cytochrome oxidase.
12 ding pattern in brain sections processed for cytochrome oxidase.
13 er membrane implicated in copper transfer to cytochrome oxidase.
14 myeloarchitecture, acetylcholinesterase, and cytochrome oxidase.
15 H dehydrogenase, alternative complex III and cytochrome oxidase.
16 mation of the Cu(A) center on the aa(3)-type cytochrome oxidase.
17 of the high-resolution crystal structures of cytochrome oxidases.
18 group of all eukaryotic and some prokaryotic cytochrome oxidases.
19 ic properties of the ferryl intermediates in cytochrome oxidases.
20 gulated the cba genes, encoding one of three cytochrome oxidases.
22 dehydrogenase 1 (ND1) by 11-fold, P < 0.005; cytochrome oxidase 1 (COX1) by 4-fold, P < 0.001; and AT
23 ignificantly decreased immunoreactivities of cytochrome oxidase 1 and cytochrome b were found in HD p
24 o of the candidate glycoproteins identified (cytochrome oxidase 2 (COX2) and NADH:ubiquinone oxidored
25 ), photoreceptors (L-M opsin, rhodopsin, and cytochrome oxidase 2), and the outer limiting membrane (
27 been reported to modulate the expression of cytochrome oxidase, a marker for metabolic activity, in
28 on by covalent modification of mitochondrial cytochrome oxidase, a phenomenon of pathological relevan
30 mal adult animals by staining flatmounts for cytochrome oxidase activity after enucleation of one eye
31 Abeta levels, hydrogen peroxide production, cytochrome oxidase activity and carbonyl proteins in Tg2
32 al bioenergetics, characterized by decreased cytochrome oxidase activity and defective calcium handli
33 of reduced membrane potential due to loss of cytochrome oxidase activity because Cox2p C-tail export
36 e on forced swim behavior and regional brain cytochrome oxidase activity in congenitally helpless rat
40 -dose methylene blue increases mitochondrial cytochrome oxidase activity in the brain and improves me
41 ess of separation group, females had greater cytochrome oxidase activity in the habenula and ventral
42 tegmental area, and subiculum, but increased cytochrome oxidase activity in the lateral frontal corte
43 is revealed that maternal separation reduced cytochrome oxidase activity in the medial prefrontal cor
46 rease in hydrogen peroxide and a decrease in cytochrome oxidase activity were found in young Tg2576 m
48 e sex-dependent effects of early handling on cytochrome oxidase activity were limited to the medial p
51 athway is not always directly linked to Cbb3 cytochrome oxidase activity, at least with respect to cb
52 s of the flattened cortex were processed for cytochrome oxidase activity, Nissl substance, or myelin.
59 ssembled 104 complete body maps, revealed by cytochrome-oxidase activity in layer 4 of 8 rodent and 1
61 as significant decrease in oxyhemoglobin and cytochrome oxidase and an increase in deoxyhemoglobin (P
62 nuclei and reduced mitochondrial density and cytochrome oxidase and citrate synthase activities relat
63 Photosynthetic organisms need copper for cytochrome oxidase and for plastocyanin in the fundament
64 he complexity surrounding copper delivery to cytochrome oxidase and highlight additional roles for so
68 seal and sea lion pups for Nissl substance, cytochrome oxidase, and vesicular glutamate transporters
69 that intermediates of the catalytic cycle of cytochrome oxidase are dynamically modulated with metabo
70 rogenase, ubiquinone-cytochrome c reductase, cytochrome oxidase, as well as ATP synthase and UCP3.
77 on of dioxygen to water by the fully reduced cytochrome oxidase at pH 6.2, 7.5, and 8.5 in the visibl
78 s also prevented the emergence of the normal cytochrome oxidase barrel pattern in forepaw and hindpaw
79 PCu(A)C and YcnJ, where they play a role in cytochrome oxidase biogenesis and copper transport, resp
80 ells are located in color-activated regions (cytochrome oxidase blobs and bridges) of primary visual
81 Color domains were tightly colocalized with cytochrome oxidase blobs in V1 and with thin stripes in
83 eurons are located preferentially underneath cytochrome oxidase blobs, indicating that MT-projecting
84 ing in the gene encoding the Cox1 subunit of cytochrome oxidase but contain wild-type levels of the b
86 the properties of one of the cell's terminal cytochrome oxidases, causing an increase in superoxide p
87 y intermediate, which weakly associates with cytochrome oxidase (CcO) in a respiratory supercomplex.
88 utations in genes encoding a subunit of Cbb3 cytochrome oxidase, ccoP, and a global response regulato
90 zing behavioral experience effects on neural cytochrome oxidase (CO) activity is well recognized, the
91 ransneuronal transporters and examination of cytochrome oxidase (CO) activity patterns after monocula
92 input nucleus HVc showed sharp increases in cytochrome oxidase (CO) activity relative to surrounding
94 osest evolutionary relative of humans, using cytochrome oxidase (CO) and acetylcholinesterase (AChE)
97 s of layer IV, and K cells send axons to the cytochrome oxidase (CO) blobs of layer III and to layer
99 elationships between orientation domains and cytochrome oxidase (CO) blobs or interblobs, CO blobs te
101 in the primary visual cortex was labeled by cytochrome oxidase (CO) histochemistry analysis or [(3)H
102 PO (TPOr, TPOi, and TPOc) were examined with cytochrome oxidase (CO) histochemistry and neurofilament
105 l serotonin transporter, in conjunction with cytochrome oxidase (CO) histochemistry, to investigate t
110 mary (striate) visual cortex, the pattern of cytochrome oxidase (CO) staining was examined in four ma
113 d 4B send segregated projections to distinct cytochrome oxidase (CO) stripes in area V2: neurons in C
117 ion, Pc, stains moderately dark for AChE and cytochrome oxidase (CO), and very light for Cat-301.
118 y cortex in the rat and by layers IIIa/b and Cytochrome Oxidase (CO)-blobs boundaries in the human pr
125 distributed indiscriminately with respect to cytochrome oxidase compartment in layer 4B, revealing a
126 import of a nuclear-encoded component of the cytochrome oxidase complex, cytochrome oxidase subunit V
127 sed a transient and potent surge in isolated cytochrome oxidase (complex IV) activity, with rapid rec
128 The yeast bc1 complex (complex III) and cytochrome oxidase (complex IV) are mosaics of core subu
129 he mitochondrial gene COX1, for subunit 1 of cytochrome oxidase, contains multiple exons and introns.
130 on fragment length polymorphisms (RFLPs) and cytochrome oxidase (cox 1/2) sequence typing was perform
133 e used the nuclear-encoded components of the cytochrome oxidase (COX) complex of the trypanosome resp
134 e involvement of Oxa1 in the assembly of the cytochrome oxidase (COX) complex, where it facilitates t
136 rial protein required for full expression of cytochrome oxidase (COX) in Saccharomyces cerevisiae.
137 aled that SphK2 is complexed with NCDase and cytochrome oxidase (COX) subunit 1 in mitochondria and t
140 x1p, one of the three core subunits of yeast cytochrome oxidase (COX), was previously shown to associ
141 mitochondrial genes, cytochrome b (cob) and cytochrome oxidase (cox1), for multiple populations of s
142 rther impaired tissue oxygenation (decreased cytochrome oxidase CuA redox state and increased deoxyhe
144 hemoglobin (HbO2), deoxyhemoglobin (Hb), and cytochrome oxidase (Cyt Ox), which reflect ATP productio
145 induction of steady-state protein levels of cytochrome oxidase, cytochrome c, and adenine nucleotide
146 formation processing and its relation to the cytochrome oxidase (CytOx) modules in visual area V2, we
147 ant has high expression of aox2, whereas the cytochrome oxidase-defective NCS6 mutant predominantly e
148 pling mechanism in humans might underlie the cytochrome oxidase deficiency that causes a form of Leig
150 here was also a close correspondence between cytochrome oxidase density and VGluT2-ir puncta distribu
151 ovudine-induced mitochondrial DNA depletion, cytochrome oxidase depletion, and mitochondrial prolifer
152 t a temperature-sensitive mutation affecting cytochrome oxidase, driving decreases in the abundance o
153 PV and S2 are less myelinated and have less cytochrome oxidase enzyme activity than area 3b; (3) The
154 al findings from the present study examining cytochrome oxidase expression replicated previous findin
156 O increases the apparent K(m) of endothelial cytochrome oxidase for O(2), allowing the endothelium to
157 ons based on these patterns include separate cytochrome oxidases for aerobic growth and oxygen scaven
160 Low Ka/Ks ratios of apocytochrome b and cytochrome oxidase genes support their utility as marker
162 gional metabolic activity using quantitative cytochrome oxidase histochemistry as in our previous stu
163 and non-helpless strains were compared using cytochrome oxidase histochemistry, an endogenous marker
164 ptide abundance, succinate dehydrogenase and cytochrome oxidase histochemistry, and electron microsco
165 rains of preweanling Holtzman rat pups using cytochrome oxidase histochemistry, which reflects long-t
169 ity trade data and mitochondrial DNA (mtDNA) cytochrome oxidase I (COI) and cytochrome b (Cyt b) gene
170 To date, two mitochondrial gene markers, Cytochrome Oxidase I (COI) and Cytochrome b oxidase (COB
172 genetic analyses of a 920 bp fragment of the cytochrome oxidase I (COI) gene revealed a well-supporte
173 obe which amplifies a 276 bp fragment of the cytochrome oxidase I (COI) mitochondrial DNA region.
178 exhibited 98-100% partial mitochondrial DNA Cytochrome Oxidase I (mtCOI) gene identity with the B. t
180 cid changes found in ATP6, cytochrome b, and cytochrome oxidase I appeared to be functionally signifi
181 diversity, and large-scale mitochondrial DNA cytochrome oxidase I barcoding has exposed many potentia
182 pacer, mitochondrial large-subunit rDNA, and cytochrome oxidase I DNA) from all 11 recognized taxa.
183 etic markers: a portion of the mitochondrial cytochrome oxidase I gene, nine polymorphic nuclear micr
184 on a single locus, the barcode region in the cytochrome oxidase I mitochondrial gene, and analyzed re
185 icularly variable in the temperate zone, and cytochrome oxidase I was notably more variable in the tr
186 ture of P. coloradensis using mitochondrial (cytochrome oxidase I) and nuclear (elongation factor 1 a
187 bomoylphosphate synthase) and mitochondrial (cytochrome oxidase I) genes representing 216 individuals
188 morphological identifications, DNA barcodes (cytochrome oxidase I) revealed significant cryptic speci
189 fic primers were designed from cytochrome b, cytochrome oxidase I, and 16S rRNA genes to generate PCR
191 has an RNase III motif, specifically cleave cytochrome oxidase II (COII) pre-mRNA insertion editing
194 e identify COII(G177S), a mtDNA hypomorph of cytochrome oxidase II, which specifically impairs male f
195 with this mitochondrial regulation, NOS and cytochrome oxidase immunoreactivity demonstrated mitocho
196 hat the membrane biogenesis of subunit II of cytochrome oxidase in bacteria and mitochondria have con
199 acrophages inhibits heme-containing terminal cytochrome oxidases, inactivates iron/sulfur proteins, a
200 tation with exogenous cytochrome c overcomes cytochrome oxidase inhibition and improves cardiac funct
202 rvational study aimed to overcome myocardial cytochrome oxidase inhibition with excess cytochrome c a
204 tions processed for Nissl substance, myelin, cytochrome oxidase, ionic zinc, neurofilaments, and vesi
209 oint for protons going into the K-channel of cytochrome oxidase is the surface-exposed glutamic acid
211 -encoded subunit of the mitochondrial enzyme cytochrome oxidase, is up-regulated in the striatum of m
212 by mitochondria was increased by inhibiting cytochrome oxidase, it induced nucleotide-sensitive unco
213 nic mice resulted in enhanced Pgc-1alpha and cytochrome oxidase IV protein expression in fast-twitch
214 creased threonine phosphorylation of COX IV (cytochrome oxidase IV), increased mitochondrial membrane
216 lutamate transporter-2, which suggested that cytochrome-oxidase maps closely mirror thalamic innervat
217 which is synthesized in mss51 mutants and in cytochrome oxidase mutants in which Cox1p translation is
218 ture; and 3) the overexpression of Cox15p in cytochrome oxidase mutants that accumulate heme O leads
219 by binding the metal centres of enzymes and cytochrome oxidase, necessitating a release mechanism fo
220 s, and three galagos that were processed for cytochrome oxidase, Nissl bodies, or the vesicular gluta
221 rkably changed in histological appearance in cytochrome oxidase, Nissl, and Wisteria floribunda agglu
222 the gracile nuclei in sections processed for cytochrome oxidase or stained for cell bodies (Nissl sta
224 ) xenobiotic metabolism, bioenergetics (e.g. cytochrome oxidase), osmotic balance (e.g. Na(+)/K(+) AT
225 during the reduction of dioxygen to water by cytochrome oxidase (P(R)) is a pH-dependent mixture of c
226 are just two main streams, originating from cytochrome oxidase patches and interpatches, that projec
227 One hypothesis is that the projection from cytochrome oxidase patches to thin stripes is responsibl
228 ns from both the inner and outer surfaces of cytochrome oxidase, perhaps accounting for the long-obse
230 siological maps of body representations with cytochrome oxidase-reacted cortical sections we were abl
231 cally, we examined the myeloarchitecture and cytochrome oxidase reactivity for several well-identifie
232 rate constant of 20,000 s-1 with the bovine cytochrome oxidase, regardless of whether the enzyme had
233 opper donors to the Cu(B) and Cu(A) sites of cytochrome oxidase, respectively, whereas Cox17 is belie
234 mitochondrial respiratory chain, Complex IV (cytochrome oxidase) retains all partially reduced interm
236 nactivation of ccoN, part of the cbb(3)-type cytochrome oxidase shown to regulate the kinase activity
238 y processing flattened cortex for myelin and cytochrome oxidase so that borders of V1 and V2 could be
239 biotically essential, Cu(A)-free cbb(3)-type cytochrome oxidase specifically in endosymbiotic bactero
240 s traversed through a complete complement of cytochrome-oxidase stained clusters (called barrelettes)
242 induces a learning-specific expansion of the cytochrome oxidase staining expression for conditioned b
243 inals, TUNEL-labeled photoreceptors, loss of cytochrome oxidase staining in photoreceptors, neurite o
251 led the existence of a group I intron in the cytochrome oxidase subunit 1 (cox1) gene in 13 of 41 gen
252 nnoprotein [MP1], and a gene fragment of the cytochrome oxidase subunit 1 gene [COX1] of the P. marne
253 r two mitochondrial DNA-encoded genes, cox1 (cytochrome oxidase subunit 1) and cob (apocytochrome b).
254 sion of the mitochondrially encoded proteins Cytochrome oxidase subunit 1, Apocytochrome b, and ATP s
255 sted that the NCS6 mitochondrial mutation, a cytochrome oxidase subunit 2 (cox2) deletion, is associa
256 transcription factor A of the mitochondria, cytochrome oxidase subunit 4 isoform 1, cAMP-response el
257 o the cytochrome c promoter and NRF-2 to the cytochrome oxidase subunit 4 promoter increased in respo
261 barcoding region of the mitochondrial gene, cytochrome oxidase subunit I (COI), is highly variable a
262 nd the use of CCG as an initiation codon for cytochrome oxidase subunit I (COI); these represent the
263 the studied species using the mitochondrial cytochrome oxidase subunit I gene (COXI) as molecular ma
265 De novo synthesis of the apocytochrome b and cytochrome oxidase subunit I proteins was no longer dete
266 , lower VDAC and the mitochondrially encoded cytochrome oxidase subunit I relative to actin; in corte
269 logenies of SFV polymerase and mitochondrial cytochrome oxidase subunit II from African and Asian mon
271 ocyclic form of T. brucei and functions as a cytochrome oxidase subunit II-specific k-RNA-editing acc
272 uding those encoding the bovine orthologs of cytochrome oxidase subunit III, IL-1 receptor type I, an
273 etion of cardiolipin synthase, the levels of cytochrome oxidase subunit IV and cytochrome c1, reflect
274 . brucei impairs the mitochondrial import of cytochrome oxidase subunit IV, an N-terminal signal-cont
275 he anti-TbTim17 antibody inhibited import of cytochrome oxidase subunit IV, indicating a direct invol
279 s were defective in C-to-U editing events in cytochrome oxidase subunit2 and NADH dehydrogenase subun
281 a and heme c levels and reduced activity of cytochrome oxidase, suggesting a defect between protopor
282 dues each induced stabilization of the bc(1):cytochrome oxidase supercomplex in a Bcs1-dependent mann
283 enotype, including defects in the aa(3)-type cytochrome oxidase, symbiotic nitrogen fixation, and ano
284 ntibodies to rod opsin, S and M cone opsins, cytochrome oxidase, synaptophysin, glial fibrillary acid
285 in the electron transport chain proximal to cytochrome oxidase that contribute to the ischemic damag
286 is a clear involvement with the assembly of cytochrome oxidases that contain the Cu(A) center in sub
287 chnique was used to form the pulsed state of cytochrome oxidase (the "OH" state) from several sources
288 abnormalities in the function of complex IV (cytochrome oxidase), the final electron acceptor in this
289 The first step in the catalytic cycle of cytochrome oxidase, the one-electron reduction of the fu
290 The final step in the catalytic cycle of cytochrome oxidase, the reduction of oxyferryl heme a(3)
292 ectron transfer by targeting haem-containing cytochrome oxidases under microaerobic conditions to mai
294 n of cellular respiration by knockout of the cytochrome oxidases was sufficient to attenuate bacteric
296 myelin, acetylcholinesterase, calbindin, or cytochrome oxidase, we identified three PI chemoarchitec
297 lattened sections of neocortex processed for cytochrome oxidase were used to determine the topography
298 I contained genes, such as that for the aa3 cytochrome oxidase, whose expression levels increased af
299 tion of redox in air, achieved by inhibiting cytochrome oxidase with cyanide, induces HIF-1 mediated
300 y the Cu(A) and intramembrane Cu(B) sites of cytochrome oxidase, within the trans-Golgi network to su
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