ライフサイエンスコーパス: cytogenetic analysis

1 trisomy 21 Down's syndrome (as confirmed by cytogenetic analysis).

2 l imbalances not detectable with traditional cytogenetic analysis.

3 ultured separately and subjected to standard cytogenetic analysis.

4 ll somatic deletions not evident by standard cytogenetic analysis.

5 omplexity than anticipated from conventional cytogenetic analysis.

6 adequate for histopathologic examination and cytogenetic analysis.

7 n was suspected on the basis of conventional cytogenetic analysis.

8 thout relying on the success of conventional cytogenetic analysis.

9 tions often are not detected by conventional cytogenetic analysis.

10 eved a complete response by Southern blot or cytogenetic analysis.

11 ients with and without t(15; 17) on standard cytogenetic analysis.

12 y karyotyping and then by more sophisticated cytogenetic analysis.

13 abnormalities of chromosome 6 determined by cytogenetic analysis.

14 Cytogenetic analysis and array comparative genomic hybri

15 We studied by cytogenetic analysis and DNA polymorphisms the nature of

16 sed on patient-specific parameters including cytogenetic analysis and gene expression profiling.

17 mice with mutations in ATM and/or p53 using cytogenetic analysis and mRNA transcriptional profiling.

18 immunohistochemistry combined with classical cytogenetic analysis and spectral karyotyping to investi

19 , possibly due to the limits of conventional cytogenetic analysis and the presence of different break

20 By applying comparative cytogenetic analysis and whole-chloroplast phylogenetics

21 well-known aberrations detectable mostly by cytogenetic analysis, and these cases are classified as

22 Cytogenetic analysis, array comparative genomic hybridiz

23 pies of this repeat unit are identifiable by cytogenetic analysis as a previously described 8p23.1 eu

24 We conclude that cytogenetic analysis at diagnosis should be used to guid

25 gene rearrangements was compared with marrow cytogenetic analysis before the initiation of treatment

26 These results support the use of interphase cytogenetic analysis, but not IgVH, CD38 expression, or

27 Cytogenetic analysis by G-banding did not detect aneuplo

28 High-resolution molecular cytogenetic analysis can now detect deletions or duplica

29 On cell-cycle profiling and cytogenetic analysis, cells derived from Rassf1a(-/-) p5

30 Standard G-band cytogenetic analysis (CG) provides information on approx

31 Cytogenetic analysis demonstrated the presence of numero

32 ed non-Hodgkin lymphomas, rendering complete cytogenetic analysis difficult.

33 However, cytogenetic analysis does not always find the translocat

34 Over the years, methods of cytogenetic analysis evolved and became part of routine

35 Standard cytogenetic analysis had been performed pretransplant in

36 In each case, cytogenetic analysis had either failed or had shown no a

37 Previous cytogenetic analysis has revealed frequent losses of chr

38 Classical cytogenetic analysis has shown a unique translocation in

39 Our cytogenetic analysis identified 16 polymorphic inversion

40 In the present study, cytogenetic analysis identified different structural rea

41 90%), N-myc gene expression in 14 (70%), and cytogenetic analysis in 10 (50%).

42 By integrating mutational and cytogenetic analysis in 1274 CLL samples and using both

43 Cytogenetic analysis indicated that, although gains of c

44 One of the known limitations of cytogenetic analysis is the inability to detect genomic

45 d diagnosis was confirmed either by means of cytogenetic analysis, molecular testing, or both.

46 the techniques, we use a recently published cytogenetic analysis of 206 melanoma cases to derive gra

47 Cytogenetic analysis of a subset of HOXB6-induced AMLs r

48 Cytogenetic analysis of acute lymphoblastic leukemia (AL

49 Cytogenetic analysis of acute myeloid leukemia (AML) cel

50 iagnosis of chromosomal abnormality requires cytogenetic analysis of amniotic fetal cells.

51 CML should be monitored routinely by serial cytogenetic analysis of bone marrow until Ph negativity

52 Cytogenetic analysis of cells from 7 sex-mismatched lung

53 Cytogenetic analysis of childhood acute lymphoblastic le

54 Cytogenetic analysis of childhood acute lymphoblastic le

55 Integration of molecular, phylogenetic, and cytogenetic analysis of dispersed repetitive DNA may she

56 We conclude that cytogenetic analysis of ependymomas may help to classify

57 FISH-based methods have greatly enhanced the cytogenetic analysis of hematopoietic and solid tumors a

58 Furthermore, cytogenetic analysis of hepatocytes transplanted from fe

59 Molecular cytogenetic analysis of leukaemic cells by banded karyot

60 who achieved a complete remission after DLI, cytogenetic analysis of marrow cells also demonstrated c

61 escence in situ hybridization studies in the cytogenetic analysis of MM.

62 Cytogenetic analysis of mouse embryonic fibroblasts and

63 Furthermore, cytogenetic analysis of mouse embryonic fibroblasts reve

64 Cytogenetic analysis of MPB collected from AP/BC patient

65 During routine cytogenetic analysis of patients with different manifest

66 overcomes many of the limitations of routine cytogenetic analysis of POC samples while enhancing the

67 lasia, or fibrous dysplasia were examined by cytogenetic analysis of short-term cultures and bi-color

68 Our expanded cytogenetic analysis of these families further identifie

69 Importantly, cytogenetic analysis of these tumors indicated that mult

70 Cytogenetic analysis of this group showed a variety of d

71 Here we present molecular and cytogenetic analysis of two cell lines (A and B) derived

72 Cytogenetic analysis of unfractionated marrow and periph

73 Cytogenetic analysis of Wilms tumours (WT) have shown th

74 oietic stem-cell engraftment was verified by cytogenetic analysis or restriction-fragment--length pol

75 Combined with cytogenetic analysis, our data demonstrate that the regr

76 Cytogenetic analysis plays a direct, potentially decisiv

77 y-seven patients with aniridia, referred for cytogenetic analysis predominantly to assess Wilms tumor

78 Genetic and cytogenetic analysis produced eight well-resolved linkag

79 Cytogenetic analysis provides critical information of di

80 graft studies combined with histological and cytogenetic analysis reveal that Separase-induced tumors

81 Cytogenetic analysis revealed a recurrent clonal inversi

82 Cytogenetic analysis revealed clonal chromosome aberrati

83 Cytogenetic analysis revealed clonal karyotypic abnormal

84 Lineage and cytogenetic analysis revealed that bone marrow progenito

85 Cytogenetic analysis revealed that SKEM cells have a hom

86 Cytogenetic analysis revealed the presence of the Philad

87 Traditional cytogenetic analysis revealed trisomy 8 in two cases of

88 Cytogenetic analysis reveals AgHex-1.1 to be encoded by

89 Cytogenetic analysis reveals the identical t(6;11)(p21.1

90 verestimating the degree of response, marrow cytogenetic analysis should be performed when patients a

91 a second unrelated family with signs of PMD, cytogenetic analysis showed a pericentric inversion of t

92 Pollen typing, segregation and cytogenetic analysis showed decreased numbers of crossov

93 f CIN precedes tumor formation, quantitative cytogenetic analysis, SKY analysis, and quantitative cen

94 f the long arm of chromosome 4 detectable by cytogenetic analysis (standard karyotyping), fluorescent

95 Findings from molecular cytogenetic analysis suggest that other genes or genetic

96 By cytogenetic analysis, the t(11;14)(q13;q32) translocatio

97 Cytogenetic analysis using comparative genomic hybridiza

98 d molecular markers on chromosome 1L and (2) cytogenetic analysis using fluorescence in situ hybridiz

99 Cytogenetic analysis was an additional useful test in de

100 genetic studies, a result that suggests that cytogenetic analysis was not merely selecting for a mino

101 Cytogenetic analysis was performed on peripheral blood l

102 Cytogenetic analysis was unsuccessful at diagnosis, but

103 Among 3,578 patients with ALL and successful cytogenetic analysis, we identified 35 patients with the

104 ee of allelic loss underestimated by routine cytogenetic analysis, which failed to detect 9 of these

105 PCR-based markers and cytogenetic analysis with genomic in situ hybridisation