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1 ptor and ERBB2 expression, and expression of cytokeratin 5.
2 s, although none expressed the basal marker, cytokeratin 5.
3 e putative epithelial progenitor cell marker cytokeratin-5.
4 immunohistochemistry for basal cells using a cytokeratin 5/14 antibody; and immunohistochemistry for
5 ology and costain with the basal cell marker Cytokeratin 5/14, whereas the CD44(-) cancer cells resem
6 ell population also expressed the basal cell cytokeratins 5/14 and epidermal growth factor receptor.
7 pancytokeratin MNF116 (which reacts with the cytokeratins 5, 6, 8, 17, and probably 19), to PCK-26 (w
8 uclear hormone receptor and HER2 negativity, cytokeratin 5/6 and vimentin expression, and stem cell e
9 und that expression of cytokeratin 17 and/or cytokeratin 5/6 in tumor cells was associated with a poo
10 or PR+, HER2+), basal-like (ER-, PR-, HER2-, cytokeratin 5/6 positive, and/or HER1+), HER2+/ER- (ER-,
12 c breast cancers expressing the basal marker cytokeratin 5/6, and age- and grade-matched controls, fo
13 n epidermal growth factor receptor 2 (HER2), cytokeratin 5/6, epidermal growth factor receptor, and K
14 , and "proliferation factor" (cytokeratin 5, cytokeratin 5/6, epidermal growth factor receptor, P53).
17 nitiating marker CD44, the progenitor marker cytokeratin 5 (CK5) and are more resistant to standard e
18 is positive for the early epithelial marker cytokeratin 5 (CK5) and the chemokine receptor CXCR4.
19 n of de-differentiated cell markers CD44 and cytokeratin 5 (CK5), lose luminal markers ER and PR, and
21 ve breast cancers contain a subpopulation of cytokeratin 5 (CK5)-expressing cells that are therapy re
22 uminal breast cancer, progesterone induces a cytokeratin-5 (CK5)-positive basal cell-like population.
24 roid sulfatase), and "proliferation factor" (cytokeratin 5, cytokeratin 5/6, epidermal growth factor
27 have discovered that a previously described cytokeratin 5 (K5)-Cre gene construct is expressed in ea
28 odel in which a ras oncogene is expressed in cytokeratin 5 (K5)-expressing cells on doxycycline admin
31 ontain increased numbers of p63/AR-positive, cytokeratin 5-negative basal cells compared with WT or A
34 lial cells fail to progress from an immature cytokeratin 5-positive to a cytokeratin 5-negative pheno
35 tch signalling to activate the DeltaNp63 and cytokeratin 5 program, and subsequent Notch blockade pro
36 ivate a DeltaNp63 (a p63 splice variant) and cytokeratin 5 remodelling program after influenza or ble
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