ライフサイエンスコーパス: cytokine profile

1 f the TH2 response with a shift toward a TH1 cytokine profile.

2 sociated with HSV-2 infection and a distinct cytokine profile.

3 -cell function and secrete a proinflammatory cytokine profile.

4 their immune response is biased toward a TH2 cytokine profile.

5 sts potentially controls the decidual T cell cytokine profile.

6 ractions with immune cells and the resultant cytokine profile.

7 sinophilia, local IgE formation, and a T(H)2 cytokine profile.

8 entifies Th17 and Tc17 cells with a peculiar cytokine profile.

9 infection was not associated with a specific cytokine profile.

10 ivariate ANOVA were used to analyze the T(H) cytokine profile.

11 e-derived dendritic cells with a tolerogenic cytokine profile.

12 on was determined from surface phenotype and cytokine profile.

13 TLR4 allele-specific DNA sequences with the cytokine profile.

14 ithout affecting responding T cell number or cytokine profile.

15 inees were CD45RA(+) and exhibited a Th0/Th1 cytokine profile.

16 ligands revealed dependence on the elicited cytokine profile.

17 Th-2; ie IL-4, IL-13) to Th-1 (ie IFN-gamma) cytokine profile.

18 skewing of the immune response toward a Th2 cytokine profile.

19 osite to the elevated donor pro-inflammatory cytokine profile.

20 l activation threshold and broadened the Th1 cytokine profile.

21 d T lymphocytes, contributed to these innate cytokine profiles.

22 according to their transcription factor and cytokine profiles.

23 culture in IL-1beta and IL-2 modified NK-22 cytokine profiles.

24 nse to CpG-A IC and CpG-B exhibited distinct cytokine profiles.

25 ullas in some individuals may shape opposite cytokine profiles.

26 the myotube surface area as well as gene and cytokine profiles.

27 articular, their ability to produce selected cytokine profiles.

28 re generally rare and heterogeneous in their cytokine profiles.

29 d as instructors of subsequent CD4(+) T cell cytokine profiles.

30 tent viral blips, were associated with these cytokine profiles.

31 l surface markers, transcription factors and cytokine profiles.

32 rtially activated populations with different cytokine profiles.

33 e response as judged by antibody isotype and cytokine profiles.

34 was analyzed histopathologically and through cytokine profiling.

35 signature is supported by flow cytometry and cytokine profiling.

36 Cytokine profiles 72 hours after injury are consistent w

37 hildren with ASD and a more activated T cell cytokine profile after phytohemagglutinin stimulation we

38 lammatory response with an anti-inflammatory cytokine profile, an extracellular matrix rich in type I

39 In contrast, phenotyping and cytokine profile analyses of PA-reactive CD4(+) T cells

40 Cytokine profile analysis during EAU revealed MC5r and A

41 y weight assessment, histologic scoring, and cytokine profile analysis.

42 ection was assessed by histopathological and cytokine profile analysis.

43 Young patients with CHB have a Th1-cell cytokine profile and a partial profile of T-cell exhaust

44 that have a broad specificity and favorable cytokine profile and are suitable for adoptive T-cell th

45 (LOX) in the aortic wall, improved systemic cytokine profile and attenuated adipose inflammation.

46 exhibit either a Th1-, a Th2-, or a Th0-type cytokine profile and dot not produce IL-10 or TGF-beta.

47 , in poor healing associated with an altered cytokine profile and fewer alternatively activated macro

48 e effects of corticosterone were assessed on cytokine profile and glucocorticoid receptor activation

49 phenotype associated with a proinflammatory cytokine profile and impaired antifungal activity of pol

50 ccompanied by an exaggerated proinflammatory cytokine profile and increased STAT3 signaling.

51 h HDM and cockroach induced a type 2/type 17 cytokine profile and mixed granulocytic inflammation in

52 Cytokine profile and proliferation of the CD4 versus CD8

53 ficient mice had an exacerbated inflammatory cytokine profile and showed enhanced CCR2-mediated macro

54 es, have a predominant T helper type-2 (Th2) cytokine profile and significantly elevated plasma level

55 ed neurological disease by alteration of the cytokine profile and virus-specific immune responses.

56 2A, T(reg) cells altered their metabolic and cytokine profile and were unable to suppress effector im

57 rrent literature regarding advances in serum cytokine profiles and associated challenges preventing t

58 clinical data and disease severity indices, cytokine profiles and C-reactive protein (CRP) concentra

59 hin the immune system are known to influence cytokine profiles and disease susceptibility.

60 We compared bacteria- and fungi-induced cytokine profiles and found that most cytokine responses

61 xpression of PLZF, T-bet, and RORgammat, and cytokine profiles and found that, although monoclonal iN

62 uman Treg cells changed their phenotypes and cytokine profiles and had potent suppressive function.

63 sferred T lymphocytes exhibited Th1 and Th17 cytokine profiles and minimal Th2.

64 have evaluated the BAL cellular composition, cytokine profiles and protein constituents in lung trans

65 ces could be mediated by the modification of cytokine profiles and regulatory T cell numbers.

66 Cytokine profiles and staining with flow cytometry were

67 The cytokine profiles and STAT signaling of these tetramer-p

68 The comparison of the inflammatory response, cytokine profiles and Th-1, Th-2 and Th-17 cells numbers

69 Human cytokine profiling and murine models of muscle inflammat

70 ession, BAL and tissue inflammatory cell and cytokine profile, and apoptosis were assessed.

71 nts with Chagas disease, determine the serum cytokine profile, and evaluate the potential association

72 ide GC Tfh cells by gene expression profile, cytokine profile, and functional properties.

73 analyzed their association with viral load, cytokine profile, and severity.

74 gen species, activation status, inflammatory cytokine profile, and type I IFN expression and signatur

75 hallenged wild-type mice maintain a distinct cytokine profile, and, unlike eosinophils isolated from

76 Work in humans suggests different cytokine profiles, and different treatment strategies fo

77 estradiol-associated side effects, deviated cytokine profiles, and enhanced suppressive activity of

78 e pattern of cell surface marker expression, cytokine profiles, and gene expression, suggesting that

79 r bacterial counts, histological evaluation, cytokine profiles, antibody level and binding activity d

80 ), bronchoalveolar fluid (BALF) cellular and cytokine profile, antigen-specific IgE and IgG1, and lun

81 Distinct airway cytokine profiles are present in current smokers and nev

82 Cytokine profiling arrays demonstrated that tumor necros

83 h-RC viruses exhibit a distinct inflammatory cytokine profile as well as significantly elevated T-cel

84 differences exist in associated magnitude or cytokine profiles as a function of disease severity.

85 In contrast, the same 10-cytokine profile assessed in serum was far less effectiv

86 xhibit an IL-4(hi)IFN-gamma(lo)TNF-alpha(hi) cytokine profile associated with a high capacity to sust

87 eutrophil phenotype and an anti-inflammatory cytokine profile associated with heightened LPS levels.

88 ematurity, reduced fetal weight, altered the cytokine profile at the maternal-fetal interface, and in

89 stric H. pylori colonization, pathology, and cytokine profiles at 6 and 19 months post inoculation (p

90 We demonstrate that peripheral cytokine profiles at birth are associated with ASD later

91 These preliminary cytokine profiling-based observations provide a possible

92 Human Th17 cells have a unique cytokine profile because the majority coexpress TNF-alph

93 ared acute HCV-specific T-cell responses and cytokine profiles between 20 HIV/HCV-coinfected and 20 H

94 we did not detect significant differences in cytokine profiles between PARP-1(-/-) and WT spinal cord

95 However, colonization altered LPMC cytokine profiles, blocking gamma interferon (IFN-gamma)

96 d that CD1d(hi)CD5(+) B cells altered T cell cytokine profile but did not directly inhibit T cell pro

97 havioral effects and tissue-specific altered cytokine profile, but absence of glial activation in spi

98 rmal in terms of both phenotype and effector cytokine profile, but they exhibited a significantly red

99 eages with distinct transcription factor and cytokine profiles, but the mechanisms underlying such fa

100 metry using HLA class II tetramers and their cytokine profile by intracellular staining.

101 metry using HLA class II tetramers and their cytokine profile by intracellular staining.

102 to harvest jejunal tissue for histology and cytokine profiles by ELISA.

103 Sputum cytokine profiling can determine distinct and overlappin

104 ial, delays loss of CD28 expression, retards cytokine profile changes, and enhances telomerase activi

105 To study the systemic cytokine profile characteristic of Strongyloides infecti

106 e p35(-/-) mice also demonstrated a distinct cytokine profile characterized by a shift from a T-helpe

107 iving BCG and Mtb72F/AS02A had a distinctive cytokine profile characterized by an increased ratio bet

108 gh-affinity CD4(+) T cells showed an altered cytokine profile characterized by the production of prot

109 ophoblast cultures induced a proinflammatory cytokine profile, characterized by elevated levels of se

110 ls from obese mice produce a proinflammatory cytokine profile compared with B cells from lean mice.

111 Th17 cells induced by CT have a unique cytokine profile compared with those induced by IL-6 and

112 demonstrated a high IL-17 and a low IFNgamma cytokine profile compared with WT B6 mice.

113 stinct T-cell receptor usage and genomic and cytokine profiles compared with the classical type I NKT

114 , but displayed a cell-surface phenotype and cytokine profile consistent with memory precursors, rais

115 A 10-cytokine profile, consisting of IL-12, CCL4, TNF-alpha,

116 -Rapa cell recipients had a balanced Th2/Th1 cytokine profile, conversion of mixed chimerism toward f

117 Follicular helper T cells with different cytokine profiles could be isolated as conjugates with B

118 onstrate this concept by determining whether cytokine profiles could discriminate RA patients accordi

119 ith chronic liver disease, but comprehensive cytokine profiling data across different clinical charac

120 ycobacterium antigen display an altered Th17 cytokine profile, decreased accumulation of granuloma-as

121 Comprehensive serum multiplex cytokine profiling demonstrated a heightened Th1-Th17 pr

122 functions that included antigenic response, cytokine profile, diabetogenicity, and suppressive funct

123 nemia, cytopenias, hypofibrinogenemia, and a cytokine profile diagnostic for HLH.

124 Cytokine profiles did not distinguish cirrhotic HBV pati

125 ity, including response rate, magnitude, and cytokine profile, did not differ between vaccinated male

126 vident across all tissues examined, the iNKT cytokine profile differed more by tissue of origin than

127 Among HCC and non-HCC patients, cytokine profiles differed between HBV and HCV patients

128 ion in cytokine responses, yet the resulting cytokine profile discriminated only between survivors an

129 an altered surface phenotype and acquired a cytokine profile distinct from that of equivalent cells

130 n accrue when vessel wall inflammation has a cytokine profile distinct from the typical proinflammato

131 eriodontitis (CP) patients to assess whether cytokine profiles distinguish patients with RA and patie

132 The serum cytokine profile during acute infection indicated a sign

133 We measured viral loads and cytokine profile during patients' acute infections.

134 An 11-cytokine profile effectively differentiated patients wit

135 hat the suppression of arthritis and the Th2 cytokine profile elicited by A9 is dependent on the pres

136 Intriguingely, as predicted from their cytokine profile, endogenous biTregs displayed additiona

137 The low IL-12p40 and high IL-6 cytokine profile expressed by BRD509(pSBRT7)-stimulated

138 se results suggest that C1q may modulate the cytokine profile expressed in response to inflammatory s

139 oll-like receptor-8 (TLR8) evokes a distinct cytokine profile favoring the generation of Type 1 helpe

140 s of the protein and a more balanced Th1/Th2 cytokine profile from antigen-specific T cells.

141 ements involved the serological response and cytokine profile from reactivated splenocytes, plethysmo

142 e the effect of EMD and its fractions on the cytokine profiles from human gingival fibroblasts in vit

143 was to examine the molecular mechanisms and cytokine profiles generated following beta-glucan stimul

144 We investigated their phenotype, cytokine profile, generation, and pathological relevance

145 tory syndromes result in the same pathogenic cytokine profile, implying that a personalized approach

146 zing the T cell immune response toward a Th2 cytokine profile in a process that is mediated by dendri

147 eron-gamma by re-expressing CD8 and a type 1 cytokine profile in association with partial Cd8a demeth

148 We assessed the Th2 cytokine profile in bile samples and brush cytology samp

149 We initially examined the cytokine profile in cultured human AML compared with AML

150 is associated with survival and a serum Th1 cytokine profile in HIV-infected individuals.

151 oorly controlled T2D may differ from the GCF cytokine profile in medically healthy individuals with p

152 otch and TLR stimulation results in a unique cytokine profile in mouse bone-marrow derived DCs charac

153 ort the hypothesis of an endogenously poised cytokine profile in neonates and suggest a link between

154 The GCF cytokine profile in patients with and without T2D seems

155 zed that the gingival crevicular fluid (GCF) cytokine profile in patients with periodontitis with poo

156 ontrast, the gut mucosa presented an anergic cytokine profile in relation to ANXA1 expression.

157 NSPT) on the gingival crevicular fluid (GCF) cytokine profile in sites with standardized periodontal

158 AT treatment of the donor induced a Th-2 cytokine profile in the adoptively transferred T cells a

159 The cytokine profile in the gut-associated lymphoid tissue (

160 Th1/Th2 immune responses, as measured by the cytokine profile in the lungs and antibody isotype of th

161 ) T cell pool may be controlled by the local cytokine profile in the microenvironment.

162 h conditions are associated with an abnormal cytokine profile in the wound bed.

163 The cytokine profile in unstimulated whole saliva (UWS) of p

164 id (CSF) cultures, inflammatory markers, and cytokine profiles in ART-naive patients with AIDS who di

165 Cytokine profiles in bronchoalveolar lavage fluids showe

166 RESEARCH DESIGN AND We determined cytokine profiles in cultured macrophages and identified

167 s trial compares the clinical parameters and cytokine profiles in gingival crevicular fluid of patien

168 by house dust mites, pulmonary function and cytokine profiles in Htr4-null mice differed little from

169 ormal semen quality and changes in chemokine-cytokine profiles in infertile men.

170 ombocytopenia and vascular leak with altered cytokine profiles in plasma are features of severe dengu

171 ent after UUO, or macrophage proinflammatory cytokine profiles in response to interferon-gamma/lipopo

172 in MR1(+/+) Valpha19i-Tg mice had disparate cytokine profiles in response to RL Ag.

173 portion of Atp6v0a2) and secreted chemokine-cytokine profiles in seminal fluid were measured.

174 IL-4), and Th2/regulatory T ([T(reg)] IL-10) cytokine profiles in serum and intestinal contents (IC)

175 The aim was to review the cytokine profiles in the GCF of patients with periodonti

176 nally, we observed pronounced differences in cytokine profiles in the livers of mutant-infected mice,

177 iated with modifications in the cellular and cytokine profiles in the lung.

178 We sought to investigate SC IL-1 cytokine profiles in the uninvolved skin of controls and

179 Cytokine profiling in locally inflamed DRG showed increa

180 metabolism and reversed the proinflammatory cytokine profile, in part due to the restoration of Lin2

181 ders had markedly different changes in serum cytokine profiles induced by duvelisib.

182 mune polarization and whether post-treatment cytokine profiles influence reinfection status.

183 We suggest that the combination of FOXP3 and cytokine profile is useful for further functionally dist

184 On cytokine profiling, LDA increased placental growth facto

185 lial cell proliferation and differentiation, cytokine profiles, lung inflammation, and fibrogenesis a

186 ifying the risk of an asthma exacerbation by cytokine profile may aid the targeting of personalized t

187 es and resolution were characterized through cytokine profiling, microscopy, and quantitation of epit

188 a Th1-skewed immune response: a Th1-shifted cytokine profile, modest lung pathology, and no signific

189 ombination of proinflammatory and regulatory cytokine profiles, natural killer cells showed a predomi

190 Inflammatory cell and cytokine profile (nuclear factor-kappaB, IL-6, tumor nec

191 The cytokine profile observed suggests a skew towards inappr

192 The inflammatory CSF cytokine profiles observed at time of IRIS can distingui

193 a source of C3a, and its uptake altered the cytokine profile of activated CD4+ T cells.

194 ite antigens and pollen allergens and/or the cytokine profile of allergic individuals.

195 lls, which in turn are able to influence the cytokine profile of antigen-specific effector T cells.

196 The cytokine profile of Barrett's metaplasia is predominantl

197 g that expression of TL by IEC modulates the cytokine profile of CD4(+) T cells favoring IL-17 produc

198 tion and T-cell priming, it did not skew the cytokine profile of DCs and pathogenic Th cells, as loca

199 t neonatal TBI study demonstrated a reversed cytokine profile of downregulation.

200 The cytokine profile of gammadelta T cells is hard-wired alr

201 However, differences persist in the cytokine profile of genital tract vs peripheral Ag-speci

202 ation of autoreactive T cells and alters the cytokine profile of helper T cells.

203 n this article, we sought to investigate the cytokine profile of IFN-gamma-activated keratinocytes, a

204 The phenotype and cytokine profile of IL-17(+)Foxp3(+)CD4(+) T cells was o

205 Further examination of the cytokine profile of iNKT-keratinocyte cocultures showed

206 -infected Kb-/-/Db-/- mice confirmed the Tc1 cytokine profile of P1- and P4-specific CD8+ T cells and

207 We studied the effect of HLA-G5 on the cytokine profile of purified human macrophages and Ag-sp

208 The development and cytokine profile of Th17 T cells differs in mice and hum

209 ve response was evaluated by determining the cytokine profile of the draining lymph node (LN) cells o

210 re, we examined the functional phenotype and cytokine profile of the in vitro induced FOXP3(+) T cell

211 e IL-6R activity might serve to maintain the cytokine profile of the Th cell infiltrate.

212 subsets which mirror the transcriptional and cytokine profile of their T cell subset counterparts.

213 nantly by the boosting immunogen whereas the cytokine profile of these cells is shaped by both the pr

214 ppressor cells, and changes in the chemokine/cytokine profile of tumors.

215 We examined serum cytokine profiles of 411 patients with HCC (n = 102: 32%

216 Cytokine profiles of DCs isolated from ethanol-fed mice

217 We generated proteomic cytokine profiles of FL (N = 50) and follicular hyperpla

218 We examined the cytokine profiles of human PBMCs and found that subsets

219 We aimed to examine the cytokine profiles of individuals sensitized to eight com

220 T cells derived from leprosy patients, while cytokine profiles of LILRA2-activated monocytes demonstr

221 We assessed cytokine profiles of liver tissues using a multiplexed a

222 V populations, potential differences in milk cytokine profiles of natural and nonnatural SIV hosts we

223 aturation influences cell fate decisions and cytokine profiles of stimulated CD8 T cells, with reperc

224 rikingly, there were also differences in the cytokine profiles of the chronically infected patients r

225 Moreover, cytokine profiles of the influenza-specific CD8+ T cells

226 Sickness behavior, temperature, and cytokine profiles of the pregnant monkeys confirmed a st

227 Sputum cellular and cytokine profiles of the validation subgroups were simil

228 Extensive cytokine profiling of sputum revealed a TH2-dominated mi

229 Cytokine profiling of supernatants from noncontact cocul

230 city associated with a raised serum Th1 type cytokine profile on transfer into preconditioned mice.

231 , plasma aspartate aminotransferase, hepatic cytokine profile, oxidative stress, and terminal deoxynu

232 analysis of cytokine responses and compared cytokine profiles, pathologies, and macrophage (Mac) pol

233 A T-helper (Th) 2 cell cytokine profile predominates in liver tissues from thes

234 a Th2-skewed immune response: a Th2-shifted cytokine profile, pulmonary eosinophilia, severe lung pa

235 anti-Ly6C mAb modulated the Ly6Chi monocyte cytokine profile, reduced monocyte recruitment to the sp

236 Histological examination and cytokine profiling reveal that intramuscular (i.m.) admi

237 Factor and cluster analyses of the sputum cytokine profiles revealed 3 biological clusters: cluste

238 Multiplex cytokine profiling revealed production of an array of pr

239 Cytokine profiling revealed similar increases in both IF

240 Cytokine profiling revealed that ODNs promote polarizati

241 Further cytokine profiling revealed that this IL-10 induction is

242 T(3) reversed many of the lung chemokine and cytokine profiles seen in response to VILI, demonstratin

243 these cells are absent, an anti-inflammatory cytokine profile shifts the alloimmune response toward a

244 d upon activation produce a pro-inflammatory cytokine profile similar to blood monocyte-derived macro

245 as associated with higher IL-17A and IL-6, a cytokine profile similar to other autoimmune diseases.

246 with TNF-alpha after toxin altered the renal cytokine profile so that the expression of proinflammato

247 This occurred with a modification of the cytokine profile, such as an increase in IFN-gamma and a

248 th or without HCC, have distinctly different cytokine profiles, suggesting potential differences in d

249 y analyzing the cell morphology, LPS-induced cytokine profile, surface marker expression, and phagocy

250 lper T cell 1 (Th1) to helper T cell 2 (Th2) cytokine profile switch.

251 fficacious and, based on its proinflammatory cytokine profile, targeted treatment option in PRP.

252 CACs expressed a broader cytokine profile than CSCs, with 3 cytokines in common.

253 re found to produce a more immunosuppressive cytokine profile than that observed by Cnt CD11b(+)Gr-1(

254 /-) mice was associated with an enhanced Th2 cytokine profile that contributed to an inappropriate im

255 and cultured T cell clones did not express a cytokine profile that indicated immune-dampening propert

256 sing hESC-DCs also induced a proinflammatory cytokine profile that may favor the T cell priming.

257 n-presenting cells (APCs) produce an altered cytokine profile that results in decreased Tregs and inc

258 isplay a dominant TH1 signature and atypical cytokine profiles that link to allergic status.

259 However, regardless of their cytokine profiles, the Foxp3-transduced CD4 T cells and

260 In patients with PMF who were studied by cytokine profiling, the prognostic value of an increased

261 ough T cell subsets are routinely defined by cytokine profiles, there may be important subdivisions b

262 n DCs accelerated maturation and shifted the cytokine profile, thereby favoring the differentiation o

263 Based on their unique surface markers and cytokine profiles, these cells were confirmed as group 3

264 FLG) is associated with an increased SC IL-1 cytokine profile; this profile is also seen in a murine

265 latent herpesviruses can directly alter host cytokine profiles through viral expression of cytokine-l

266 ction, murine mphis elicited an inflammatory cytokine profile (TNF-alpha >> TGF-beta + IL-10) and low

267 and therapy was shown to alter diabetogenic cytokine profile, to diminish T-cell effector frequency

268 patients with SR asthma by directing the SR cytokine profile toward a more SS immune phenotype, sugg

269 Based on their cytokine profile upon exposure to pathogenic stimuli, hu

270 , CTLA-4, and ICOS and had a Th1/Th17 skewed cytokine profile upon polyclonal stimulation.

271 We monitored the cytokine profile (using enzyme-linked immunosorbent assa

272 ociated with ASD later in childhood and that cytokine profiles vary depending on ASD severity.

273 The anti-C1q-induced inflammatory cytokine profile was accompanied by a downregulation of

274 An inflammatory mucosal and systemic cytokine profile was characterized by expression of IL-1

275 activated CD4(+)-T cells with a Th1 and Th17 cytokine profile was increased in cMy-mOVA-OT-II mice af

276 The same cytokine profile was observed in ex vivo culture of cerv

277 altered T cell functional potential; T cell cytokine profile was preserved.

278 sal TH1 to TH2 or T-cytotoxic 1 (TC1) to TC2 cytokine profiles was not evident.

279 ctivation, animal survival, lung injury, and cytokine profile were assessed.

280 ntitumor efficacy, whereas T cell number and cytokine profile were not.

281 TRIF-signaling pathways and their associated cytokine profiles were altered in the absence of CD14 in

282 ed using computer morphometry and intragraft cytokine profiles were analyzed with Western Blotting.

283 Lamina propria dendritic cell phenotype and cytokine profiles were assessed by flow cytometric analy

284 iferation of the transferred cells and their cytokine profiles were assessed by fluorescence tagging

285 intestinal histology and systemic and local cytokine profiles were compared between the treated and

286 Cytokine profiles were evaluated by ELISA.

287 Cell supernatant cytokine profiles were evaluated by multiplex assays.

288 rkers than those without ACLF; (2) different cytokine profiles were identified according to the type

289 ent to lesions, degree of tissue damage, and cytokine profiles were measured.

290 miRNA-mediated changes in cytokine profiles were observed at transcriptional and/o

291 Cytokine profiles were obtained 20 min after the first s

292 Plasma pro- and antiinflammatory cytokine profiles were performed by enzyme-linked immuno

293 nohistochemical testing, flow cytometry, and cytokine profiling were performed on samples from the pa

294 oduction by skewing toward a proinflammatory cytokine profile, whereas when these cells are absent, a

295 CD91 interaction on APCs stimulates a unique cytokine profile, which dictates priming of specific Th

296 ular mechanisms were associated with a mixed cytokine profile with a tendency toward increased levels

297 cells, yet they possessed a proinflammatory cytokine profile with high tumor necrosis factor-alpha a

298 Alterations in immune cytokine profiles with aging show significant gender spe

299 on resulted in a significant reversal of the cytokine profile, with increased levels of proinflammato

300 Melanoma patients may exhibit a T(H)2-skewed cytokine profile within blood and tumor-infiltrating lym