ライフサイエンスコーパス: cytokine secretion

1 igration, proliferation, differentiation and cytokine secretion.

2 igen expression, angiogenic and inflammatory cytokine secretion.

3 ligase gene (waaL) enhanced proinflammatory cytokine secretion.

4 effect is mediated by changes in tumor cell cytokine secretion.

5 e immune signaling, culminating in antiviral cytokine secretion.

6 face responsible for the galectin-3-mediated cytokine secretion.

7 nctions such as proliferation, survival, and cytokine secretion.

8 ry arthritis and colitis, and its effects on cytokine secretion.

9 l activation, metabolism, proliferation, and cytokine secretion.

10 induced caspase-1 activation, signaling, and cytokine secretion.

11 recruitment of Th cells and increased type 1 cytokine secretion.

12 the upregulation of costimulatory ligands or cytokine secretion.

13 plays a role in regulating pro-inflammatory cytokine secretion.

14 iate protective and repair functions through cytokine secretion.

15 resulted in increased Th17 polarization and cytokine secretion.

16 nflammatory cell recruitment, and airway-Th2 cytokine secretion.

17 lA nuclear translocation, NO production, and cytokine secretion.

18 NFkappaB signaling, and, ultimately, overall cytokine secretion.

19 ired cell autonomously for optimal iNKT cell cytokine secretion.

20 vascular cell proliferation, migration, and cytokine secretion.

21 tivity was measured through proliferation or cytokine secretion.

22 ells, suppressing Th1 cell proliferation and cytokine secretion.

23 ulator of interferon genes (STING)-dependent cytokine secretion.

24 nces, as reported by T-cell proliferation or cytokine secretion.

25 oliferation and expression of Bcl-xL but not cytokine secretion.

26 y with monocytes and induced proinflammatory cytokine secretion.

27 n, surfactant clearance, and proinflammatory cytokine secretion.

28 ng oligomerization domain 2 (NOD2)-initiated cytokine secretion.

29 nel in the regulation of alveolar epithelial cytokine secretion.

30 PS28, as negative regulators of NOD2-induced cytokine secretion.

31 ent of T cells did not directly alter T cell cytokine secretion.

32 gnaling cascades and induced proinflammatory cytokine secretion.

33 explained by the lower NFATc1 expression and cytokine secretion.

34 ype and proliferation and immunoglobulin and cytokine secretion.

35 sregulated intestinal immune homeostasis and cytokine secretion.

36 expression of TH17 and Treg cell markers and cytokine secretion.

37 (ICOS) expression and interleukin-21 (IL-21) cytokine secretion.

38 onal T-cell receptor repertoire and enhanced cytokine secretion.

39 ne secretion and an increase in regenerative cytokine secretion.

40 for mediating Fbxl2 disposal and stimulating cytokine secretion.

41 d HIF-1alpha stabilization, and inflammatory cytokine secretion.

42 rus-sensing pathway that drives inflammatory cytokine secretion.

43 We analyzed caspase 1 activity and cytokine secretion.

44 rough the control of bile hydrophobicity and cytokine secretion.

45 tight junctions but not for the induction of cytokine secretion.

46 d no effect on both T cell proliferation and cytokine secretion.

47 ted in a 3-fold decrease in pressure-induced cytokine secretion.

48 ates transformation but not pro-inflammatory cytokine secretion.

49 sponsive to LPS ex vivo, exhibiting elevated cytokine secretion.

50 dent, it was not accompanied by inflammatory cytokine secretion.

51 a, IL-6, and IL-17 despite normal splenocyte cytokine secretion.

52 lity, growth, gene expression signature, and cytokine secretion.

53 ability to induce T-cell proliferation, and cytokine secretion.

54 ly kinases (SFKs) and SFK inhibition blocked cytokine secretion.

55 signaling events that drive ISG15-dependent cytokine secretion.

56 ation also inhibits innate gammadelta17 cell cytokine secretion.

57 ctivation, secondary granule exocytosis, and cytokine secretion.

58 ells to degranulate and induce chemokine and cytokine secretion.

59 potent than regulatory T cells at inhibiting cytokine secretion.

60 n factor NF-kappaB, leading to chemokine and cytokine secretion.

61 rway hyperreactivity (AHR) and driven by Th2 cytokine secretion.

62 ith CHS had normal cytokine compartments and cytokine secretion.

63 cytes through the induction of suppressor of cytokine secretion 1 and suppressor of cytokine secretio

64 or of cytokine secretion 1 and suppressor of cytokine secretion 3.

65 cal TCR-induced signaling events that led to cytokine secretion, a high multiplicity of TCR-CD3 ITAMs

66 IL-5 augmented cytokine secretion, activation, phagocytosis, and surviv

67 because their removal led to an increase in cytokine secretion after activation.

68 downregulating signaling and proinflammatory cytokine secretion after chronic NOD2 and TLR4 stimulati

69 RAKM and secreted mediators, to downregulate cytokine secretion after chronic NOD2 stimulation.

70 or (TLR) expression by quantitative PCR, and cytokine secretion after stimulation with mitogenic, TLR

71 d IL-10 levels and decreased proinflammatory cytokine secretion, ameliorated bacterial translocation

72 iated with increased GM-CSF, IL-4, and IL-13 cytokine secretion among Ag-stimulated low-affinity iNKT

73 six statins tested, suppressing IgE-induced cytokine secretion among mouse MCs and basophils.

74 ted induction of endothelial proinflammatory cytokine secretion and adhesion molecule expression.

75 h resulted in a reduction in proinflammatory cytokine secretion and an increase in regenerative cytok

76 gainst TLR2 and TLR4 significantly inhibited cytokine secretion and attenuated Lsa21 induced phosphor

77 ate decreased PRR-induced mtROS, signalling, cytokine secretion and bacterial clearance.

78 lling, mitochondrial ROS (mtROS) production, cytokine secretion and bacterial clearance.

79 (ESLD) suppressed macrophage proinflammatory cytokine secretion and bacterial killing in vitro in a P

80 possess a range of suppressive function and cytokine secretion and can exert a genomic footprint on

81 tion of primed CD8(+) T cells as revealed by cytokine secretion and CD107a degranulation was not infl

82 ases caspase-8 and caspase-1 in coordinating cytokine secretion and cell death in response to immunos

83 pathogens by activating caspase-1-dependent cytokine secretion and cell death.

84 d T-cell cytokine interleukin-2, we show how cytokine secretion and competitive uptake determine this

85 f an miR-155 mimic increased proinflammatory cytokine secretion and costimulatory surface marker expr

86 expressed CD45RO and demonstrated decreased cytokine secretion and cytolytic potential after specifi

87 gulation of activation markers, induction of cytokine secretion and cytotoxicity.

88 by myeloid cells and promote proinflammatory cytokine secretion and endothelial cell adhesion, sugges

89 d-type counterparts, but they exhibit normal cytokine secretion and expression of cytotoxic mediators

90 nization of the secretory pathway to control cytokine secretion and facilitate formation of the VAC f

91 ion of the B-cell compartment and influences cytokine secretion and immunoglobulin production.

92 with sham-operated mice, including elevated cytokine secretion and impaired bacterial clearance.

93 ose from WT mice had reduced proinflammatory cytokine secretion and impaired macrophage activation.

94 aB expression in KCs in obese mice decreased cytokine secretion and improved insulin sensitivity and

95 is of bacteria while decreasing inflammatory cytokine secretion and increased intracellular ATP level

96 dotoxin sensitivity, as measured by enhanced cytokine secretion and lethality following LPS challenge

97 in vitro priming of CD8(+) T cells improved cytokine secretion and lytic capacity of high-avidity T

98 C growth advantage through NF-kappaB-related cytokine secretion and metastatic TNBC cells exhibit gai

99 ergistically with the TLR pathway to promote cytokine secretion and neutrophil migration, whereas the

100 e-1 or caspase-11 activation, culminating in cytokine secretion and obliteration of the replicative n

101 hages, we investigated the role of Syt XI in cytokine secretion and phagocytosis.

102 Thus, the TCR-driven pathways that initiate cytokine secretion and proliferation are separable and a

103 cell migration to inflamed skin, but not for cytokine secretion and proliferation in regional lymph n

104 The link between cytokine secretion and proliferation rate holds both in

105 disease progression exhibited decreased Th1 cytokine secretion and proliferative capacity, and reduc

106 ivating both caspase-1-mediated inflammatory cytokine secretion and pyroptosis.

107 Inhibiting TIFA perturbed leukemic cytokine secretion and reduced the IC50 of chemotherapeu

108 The helminth-induced anti-inflammatory cytokine secretion and regulatory T cell suppressor acti

109 phages that coincides with the modulation of cytokine secretion and specific cellular processes.

110 of Pol III activity in macrophages restrains cytokine secretion and suppresses phagocytosis, two key

111 Early postvaccination cytokine secretion and T lymphocyte and plasmablast acti

112 se data demonstrate that sortilin influences cytokine secretion and that targeting sortilin in immune

113 ed in the inhibition of both proinflammatory cytokine secretion and the upregulation of costimulatory

114 nction, likely further disrupting regulatory cytokine secretion and ultimately exacerbating inflammat

115 c infections induce T cells showing impaired cytokine secretion and up-regulated expression of inhibi

116 PD1/28 chimeric molecule, exhibited enhanced cytokine secretion and upregulation of activation marker

117 Therefore, we studied B cell cytokine secretion and/or Ab production across obesity m

118 could include roles in lymphoid development, cytokine secretion, and Ag presentation; however, these

119 T-cell proliferation, cytokine secretion, and antibody titers were measured by

120 f DC, through cyclin-dependent pathways, Th1 cytokine secretion, and by adding a nonviral Ag highly o

121 f T cell function, augmenting proliferation, cytokine secretion, and cell survival.

122 3(bright) surface phenotype, proinflammatory cytokine secretion, and cytotoxicity.

123 roduction, collagen deposition, dysregulated cytokine secretion, and elevated inflammation.

124 in upregulation of antiapoptotic molecules, cytokine secretion, and enhanced effector function.

125 t v 1, epitope specificity, allergen-induced cytokine secretion, and expression of integrins alpha4be

126 mediators, rapidly communicating danger via cytokine secretion, and functioning as guardians of tiss

127 ll myeloperoxidase activity, proinflammatory cytokine secretion, and inflammation, were significantly

128 d for optimal MAPK and NF-kappaB activation, cytokine secretion, and intracellular bacterial clearanc

129 conjugate and analyzed for cell activation, cytokine secretion, and metabolic state.

130 urface strongly inhibited B cell activation, cytokine secretion, and proliferation.

131 ages were used to evaluate barrier function, cytokine secretion, and protein expression under basal c

132 ted cytokine secretion, elevated TH1-related cytokine secretion, and reduced human monocyte recruitme

133 s of immunity including antibody production, cytokine secretion, and T-cell activation; moreover, B c

134 The maturation, cytokine secretion, and T-cell stimulatory capacity of r

135 e characterized in vitro on their phenotype, cytokine secretion, and Th2 polarizing ability.

136 overexpressing PC cells, inducing apoptosis, cytokine secretion, and the recruitment of human periphe

137 raneoplastic syndrome mechanisms via ectopic cytokine secretion, and/or a non-HHV-8 virus.

138 pacity, cytokine production, and kinetics of cytokine secretion are associated with HIV-1 control.

139 n predominantly on dying cells and stimulate cytokine secretion as well as leukocyte recruitment in v

140 The newly developed cytokine secretion assay consists of anti-IL-22 and anti

141 et, and the combination of several different cytokine secretion assays can be used to purify and char

142 ls was performed with the combination of two cytokine secretion assays detecting IL-17A- and IL-22-pr

143 aded with tumor peptide and antigen-specific cytokine secretion assays, we determined that TNF-alpha

144 activation by myeloperoxidase activity, and cytokine secretion at specific time points.

145 de data to clinicians and doctors concerning cytokines secretion at minute concentrations and the pre

146 s recent advances in deciphering pathways of cytokine secretion, both from intact eosinophils and fro

147 STK4 dampened TLR4/9-induced proinflammatory cytokine secretion but enhanced TLR3/4-triggered IFN-bet

148 is implicated in controlling proinflammatory cytokine secretion, but the intracellular signaling path

149 of HIV have largely relied on measurement of cytokine secretion by effector T cells.

150 iacetate succinimidyl ester dye dilution and cytokine secretion by ELISpot.

151 nts demonstrate that nGO-PEG indeed provokes cytokine secretion by enhancing integrin beta8-related s

152 I interferon and stimulated proinflammatory cytokine secretion by human peripheral blood cells.

153 as IL-10 and TGF-beta1 significantly reduce cytokine secretion by ILC2s.

154 In this study, we assessed rhythms of cytokine secretion by immune cells and tested their resp

155 yanin resulted in enhanced proliferation and cytokine secretion by keyhole limpet hemocyanin-experien

156 to A1 and A3 adenosine receptors, increases cytokine secretion by LPS activated monocytes.

157 extracellular DNA to trigger proinflammatory cytokine secretion by monocytes, in a STING- and inflamm

158 t TH17 subsets on modulating proinflammatory cytokine secretion by monocytes.

159 integrity, and induction of proinflammatory cytokine secretion by S. Paratyphi A but not by S. Typhi

160 Cytokine secretion by T lymphocytes plays a central role

161 ), histamine and mast cell protease release, cytokine secretion, calcium flux, and changes in cell nu

162 gnition receptor (PRR)-induced signaling and cytokine secretion can lead to inflammatory bowel diseas

163 Virus replication, cytokine secretion, cell viability, and type I interfero

164 peptides from the initial culture to analyze cytokine secretion (Cytokine-driven assay).

165 vating receptor for NK cell cytotoxicity and cytokine secretion, did not inhibit NK cell immigration,

166 ding a given antigen for 48 hours to measure cytokine secretion (Direct assay).

167 ent the importance of PKR, TRIF, and TBK1 in cytokine secretion during L. pneumophila infection of ma

168 r expression and TH1/TH2 and proinflammatory cytokine secretion early in life.

169 In contrast, a lack of Kif5b did not affect cytokine secretion, early FcepsilonRI-initiated signalin

170 LO/LTB4 inhibition downregulated TH2-related cytokine secretion, elevated TH1-related cytokine secret

171 can modulate the immune response by inducing cytokine secretion, especially IL-10 and MIP-1beta.

172 es increased cytolytic effector capacity and cytokine secretion, even in posttransplant samples in wh

173 locks both TCR-dependent and TCR-independent cytokine secretion from a Sezary syndrome-derived cell l

174 (rapa-ECs) stimulated less proliferation and cytokine secretion from allogeneic CD4+ memory cells, an

175 gh fibrillar amyloid beta (Abeta)-stimulated cytokine secretion from both wild-type and APP knock-out

176 for functional immunophenotyping to examine cytokine secretion from human immune cells stimulated ex

177 ligase component, Fbxo3, potently stimulates cytokine secretion from human inflammatory cells by dest

178 component, termed Fbxo3, potently stimulates cytokine secretion from human inflammatory cells by medi

179 OVA-specific IgE, IgG1, and IgG2a and cytokine secretion from mesenteric lymph node (MLN) cell

180 AMA0825 did not affect pro-inflammatory cytokine secretion from other ROCK-positive cell types,

181 However, the mechanisms underlying cytokine secretion from platelets are undefined.

182 trated by potent and sustained inhibition of cytokine secretion from T cells, a therapeutic target fo

183 Here we show that TRAIL-triggered cytokine secretion from TRAIL-resistant cancer cells is

184 Tregs have more potent suppressive effect on cytokines secretion from CD4(+) CD25(-) responder T cell

185 1BB CAR T cells retained their cytotoxic and cytokine secretion functions longer than CD28 CAR T cell

186 Cytokine secretion, gene expression, and transcription f

187 of IL-33 on goblet cell differentiation and cytokine secretion has not been described.

188 In contrast to cytokine secretion, HMGB1 redistribution required the p5

189 ffects of lung endothelium and epithelium on cytokine secretion, identify new biomarkers of disease e

190 a higher threshold to induce TRIF-dependent cytokine secretion (IL-1beta, IL-6, IL-10, and TNF-alpha

191 ificantly inhibited CRE-induced inflammatory cytokine secretion (IL-4, IL-13, IL-17, and IFN-gamma) b

192 assified into distinct groups based on their cytokine secretion: ILC1 produce IFN-gamma, ILC2 secrete

193 ride-induced inflammatory gene expression or cytokine secretion in any cell type examined, including

194 injury, bacterial clearance from the lungs, cytokine secretion in bronchoalveolar lavage, lung antim

195 e the link between cell stress and increased cytokine secretion in CAPS.

196 Thrombin enhanced cytokine secretion in CD8(+) T cells from healthy contro

197 However, EVT did not enhance cytokine secretion in dNK, whereas stimulation of dNK wi

198 Interestingly, MOMP proteosomes induce cytokine secretion in endocervical epithelial cells (End

199 protein 3 (MTMR3) in amplifying PRR-induced cytokine secretion in human macrophages and defined MTMR

200 arasite replication in mouse infections, and cytokine secretion in macrophage infections.

201 urium-induced pyroptosis and proinflammatory cytokine secretion in macrophages.

202 -initiated MAPK/NF-kappaB/PI3K signaling and cytokine secretion in macrophages.

203 IL-1 signaling, thereby leading to decreased cytokine secretion in MDMs upon stimulation of a broad r

204 18RAP was critical in amplifying PRR-induced cytokine secretion in MDMs.

205 cantly reduced crypt damage and inflammatory cytokine secretion in NOD2(-/-) mice 3 d after anti-CD3

206 pecific CLA(+) and CCR4(+) proliferation and cytokine secretion in patients with and without APT reac

207 nt levels to induce NF-kappaB activation and cytokine secretion in primary DCs and peripheral blood m

208 The nitric oxide production and cytokine secretion in rat peritoneal cells indicate immu

209 8.0 trauma, p < 0.05) and reduced leukocyte cytokine secretion in response to lipopolysaccharide sti

210 ired for B cell survival, proliferation, and cytokine secretion in response to signaling through seve

211 GF, bFGF, EGF, IL-6, and other proangiogenic cytokine secretion in stromal and tumor cells.

212 induction of pyroptosis and proinflammatory cytokine secretion in the control of growth and eliminat

213 roliferation and to reduced pro-inflammatory cytokine secretion in the lymph nodes of ISLAD-treated E

214 associated with increased proliferation and cytokine secretion in the spleen, intraepithelial lympho

215 imulation and is required for attenuation of cytokine secretion in vitro in human macrophages and in

216 ished their proliferation, cytotoxicity, and cytokine secretion in vitro in response to CD19 recognit

217 MDA-induced cytokine secretion in vitro was dependent on the presenc

218 ge associated with decreased proinflammatory cytokine secretion in vivo.

219 ndritic cell maturation and T helper 1 (Th1)-cytokine secretion, in turn driving stronger effector CD

220 erized by enhanced glycolysis and an altered cytokine secretion (interleukin-6 P<0.0001, interleukin-

221 (mtDNA) integrity, morphology, phenotype and cytokine secretion into storage buffer.

222 Regulation of microbially induced cytokine secretion is critical in intestinal immune home

223 Paracrine signalling mediated by cytokine secretion is essential for liver regeneration a

224 ronic NOD2 stimulation in human macrophages, cytokine secretion is significantly attenuated, similar

225 e underlying mechanisms are unknown, massive cytokine secretion is thought to be involved.

226 li modulating amino acid catabolism, as were cytokine secretion levels and regulatory T cell numbers.

227 udy, we show that M. tuberculosis impairs DC cytokine secretion, maturation, and Ag presentation thro

228 Despite the absence of inflammatory cytokine secretion, mDCs incubated with P. falciparum-in

229 uppressed MDDC chemokine and proinflammatory cytokine secretion, nuclear factor kappaB and activator

230 Furthermore, reduced cytokine secretion observed at high Ag density for both

231 The H4R modulated the cytokine secretion of CD4+ T cells and splenocytes and a

232 effect of synthesized iminosugars 1-3 on the cytokine secretion of IL-4, IL-6, and IFN-gamma was eval

233 the proliferation (72%) and proinflammatory cytokine secretion of pathogenic MOG(35-55)-specific T-c

234 Cs on T cell effector function as assayed by cytokine secretion of T cells.

235 ory responses, and increased proinflammatory cytokine secretions of TNF-alpha, IFN-gamma, and IL-13 c

236 o T cells but had no defects in endocytosis, cytokine secretion, or expression of costimulatory molec

237 al a novel role for Syt XI as a regulator of cytokine secretion, particle uptake, and macrophage micr

238 l as the induction of cytotoxicity-promoting cytokine secretion, particularly interleukin-12, both of

239 activation test (MAT), MPLA induced the same cytokine secretion pattern as LPS (ESM: IL-6, IL-12, TNF

240 ), such as Sezary syndrome, display aberrant cytokine secretion patterns that contribute to pathology

241 T2, and NKT17 subsets, defined through their cytokine-secretion patterns and the expression of key tr

242 ntly infected cells to limit proinflammatory cytokine secretion, perhaps as an immune evasion strateg

243 maturation and their acquisition of an NKT17 cytokine secretion phenotype in the thymus.

244 D4(+) T-lymphocyte memory subpopulations and cytokine secretion phenotypes, although cellular immune

245 e detected in most subjects with CSU, with a cytokine secretion profile more typical of a TH1-cell re

246 We observed that the magnitude, breadth, and cytokine secretion profile of HIV-1-specific CD8 T cell

247 HP-NAP-activated DCs had a Th1 cytokine secretion profile, with high IL-12 and relative

248 patients with ABPA is skewed to a T helper 2 cytokine secretion profile.

249 istinct helper subsets on the basis of their cytokine-secretion profile.

250 atural killer targets confirmed the distinct cytokine secretion profiles of dNK and peripheral blood

251 n primary T cells: they can drive a la carte cytokine secretion profiles, biased T cell differentiati

252 h together with specific gene expression and cytokine secretion profiles, FL-TFH constitute a heterog

253 activity, expression of surface markers, and cytokine secretion profiles.

254 AMPK and reductions in NK cell cytotoxicity, cytokine secretion, proliferation, and telomerase expres

255 c immunophenotyping and functional assays of cytokine secretion/proliferation to TH cells from 18 CHL

256 d not markedly increase IL-10/IL-2 (Th2/Th1) cytokine secretion ratios, suggesting that menthone, far

257 ized by impairment of cytolytic activity and cytokine secretion, reduced expression of IL-2Ralpha (CD

258 trophoblast cells in vitro are by modulating cytokine secretion, reducing apoptosis to levels seen in

259 Macrophage cytokine secretion, relevant to infarct healing and repa

260 rol their homeostasis and downregulate their cytokine secretion remain poorly understood.

261 The altered balance of cytokine secretion results in preferential recruitment o

262 rsed the proinflammatory gene expression and cytokine secretion seen in BMDM from high fat-fed mice.

263 decrease of plasma levels of proinflammatory cytokine secretion such as tumor necrosis factor-alpha,

264 at could be targeted to inhibit the aberrant cytokine secretion that drives the immunopathogenesis of

265 a combination of cell-to-cell signalling and cytokine secretion that elicit unique biological effects

266 ed with this, and based on their patterns of cytokine secretion, there was a difference in their capa

267 Notably, TNFSF15 treatment also induced cytokine secretion through a caspase-8-dependent pathway

268 osomal proteins and inducing proinflammatory cytokine secretion through protease-activated receptor 2

269 ts secreted oxidized HMGB1, which stimulated cytokine secretion through TLR-4 signaling.

270 dose-dependently promoted DC maturation and cytokine secretion through TLR4 signaling pathway.

271 drive the Th1 immune response by suppressing cytokine secretion (TNF-alpha, IL-6, IFN-gamma, and IL-1

272 alters how DCs metabolize glutamate, skewing cytokine secretion to bias T cell function.

273 sess the effects of the miRNAs identified on cytokine secretion (tumor necrosis factor alpha [TNF-alp

274 overall magnitude of TNF-alpha and IFN-gamma cytokine secretion upon Ab-dependent and -independent ac

275 (CTLR) triggering cellular cytotoxicity and cytokine secretion upon high-affinity interaction with t

276 c cells, were used to compare differences in cytokine secretions upon stimulation.

277 fects and through reduction of proangiogenic cytokine secretion via the microenvironment.

278 Type 1 cytokine secretion was also enhanced by DC and was most

279 Cytokine secretion was assayed by enzyme-linked immunoso

280 ortantly, this costimulation of TLR2-induced cytokine secretion was dependent on regiospecific methyl

281 otype was investigated by flow cytometry and cytokine secretion was determined by ELISA.

282 ng TLRs were exposed to cSSSI pathogens, and cytokine secretion was determined by enzyme-linked immun

283 The hCG effect on Treg frequency and cytokine secretion was determined in Foxp3(gfp) females.

284 t pattern-recognition receptor (PRR)-induced cytokine secretion was diminished in human monocyte-deri

285 Cytokine secretion was measured 24 hours postinfection b

286 Cytokine secretion was measured using multiplex and ELIS

287 that were sufficient in LAIR-1, CD3-induced cytokine secretion was significantly suppressed in the p

288 ng night shifts, with the exception of IL-2, cytokine secretion was still rhythmic but with peak leve

289 ted IL-10 secretion, but not proinflammatory cytokine secretion, was inhibited by rapamycin in mDCs.

290 ell tropism, effect on tissue integrity, and cytokine secretion were compared.

291 infection, studies of inflammasome-dependent cytokine secretion were conducted with the human THP-1 m

292 Significantly high levels of cytokine secretion were induced by either viable or heat

293 Drug-specific proliferative responses and cytokine secretion were measured.

294 udies, including migration, Ca(2+) flux, and cytokine secretion, were conducted with primary human mo

295 from PC1/3 KO mice also demonstrate elevated cytokine secretion when treated with LPS.

296 of NETs alone did not induce proinflammatory cytokine secretion, whereas LPS-induced production of IL

297 Pam-(Lys-betaNSpe)6-NH2, blocks LPS-induced cytokine secretion with a potency comparable to that of

298 to 37 degrees C does not normalize monocyte cytokine secretion within 36 hours, whereas warming to 3

299 diated indirect alloantigen presentation and cytokine secretion within the GI tract.

300 ntigen-mediated adhesion, degranulation, and cytokine secretion without changes to extracellular calc