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1 first step to determine the function of the cytoplasmic filaments.
2 nd electron microscopy confirmed the lack of cytoplasmic filaments.
3 RE3] cells, nor do the control cells exhibit cytoplasmic filaments.
4 g compaction of the NPC and extension of the cytoplasmic filaments.
5 requires Dbp5 interaction with Nup159 in NPC cytoplasmic filaments and activation of Dbp5's ATPase ac
6 n-specific nucleoporin that localizes to the cytoplasmic filaments and provides several binding sites
7 alysis suggests that it is unlikely that the cytoplasmic filaments and the flagellar apparatus are ph
12 e unique structures, the gene coding for the cytoplasmic filaments, cfpA, was identified in various t
13 induction also occurs in response to keratin cytoplasmic filament collapse, absence, or ablation of K
17 Moreover, chains of cells are formed by the cytoplasmic filament-deficient mutant, and those cells s
19 domain and the NLS proved necessary for both cytoplasmic filament formation and nuclear targeting.
21 nderstanding of the involvement of bacterial cytoplasmic filaments in cell division requires the eluc
22 y condensed chromosomal DNA suggest that the cytoplasmic filaments may be involved in chromosome stru
25 Nup) and 4000-fold higher than to Nup42p (a cytoplasmic filament Nup), revealing a steep gradient of
27 f the NPC and is implicated in anchoring the cytoplasmic filaments of the NPC and recruiting the RNA
29 itment of the DEAD-box helicase Ddx19 to the cytoplasmic filaments of the NPC that is mediated by the
31 RanBP2/Nup358, the major component of the cytoplasmic filaments of the nuclear pore complex (NPC),
32 ther set of components appears to bridge the cytoplasmic filaments on the cytoplasmic side, and to be
34 a ribbon-like profile and consist of a major cytoplasmic filament protein subunit (CfpA, formerly cal
37 -deficient mutant of T. phagedenis possessed cytoplasmic filaments similar to those of the wild type,
38 ponema denticola, a ribbon-like structure of cytoplasmic filaments spans the cytoplasm at all stages
39 se proteins cause apoptosis by forming novel cytoplasmic filaments that recruit and activate pro-casp
40 Spirochaeta, and Leptonema contain multiple cytoplasmic filaments that run the length of the organis
42 leoplasmin-gold along the length of extended cytoplasmic filaments, whereas RanGDP caused accumulatio
43 ed in large irregular bodies associated with cytoplasmic filaments while at late stages, vRNA was dis
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