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1 thout the presence of any other cytotoxic or cytostatic agent.
2 II trials may not be appropriate for testing cytostatic agents.
3 dependent manner) may not be appropriate for cytostatic agents.
4 s for modifying trial designs to accommodate cytostatic agents.
5 onse to a broad range of cell cycle specific cytostatic agents.
6 s in the early development stage of targeted cytostatic agents.
7 ns for the early development of target-based cytostatic agents.
8  models for the treatment effect of targeted cytostatic agents.
9 esign to evaluate the activity of a putative cytostatic agent, acknowledging heterogeneity of tumor g
10 etagal-treated vessels (P<0.05), whereas the cytostatic agent Ad-p21 decreased lesion size by 58% (P<
11 ate that in SH-SY5Y cells, suramin acts as a cytostatic agent and can block IGF-II-dependent cell gro
12 ory nerve action potential amplitude for all cytostatic agents and a moderate reduction of nerve cond
13 al trials suggested that sorafenib acts as a cytostatic agent, as many patients experienced prolonged
14                 Although mTOR inhibitors are cytostatic agents, best used in combination therapy, we
15 e progression) to evaluate the efficacy of a cytostatic agent in a phase II trial is more relevant th
16 onged by subsequent treatment with the three cytostatic agents in all HCCs may be of clinical importa
17 is a growth suppressive program activated by cytostatic agents in some cancer cells.
18 intenance strategies with both cytotoxic and cytostatic agents in women who achieve a secondary respo
19  As known and described in detail, the three cytostatic agents inhibit different processes necessary
20 ongly suggest that the use of rapamycin as a cytostatic agent may be an efficient tool for the treatm
21                                         Such cytostatic agents may offer clinical benefits for patien
22 n colon carcinoma (HCC) cells, the effect of cytostatic agents reported to inhibit HCC growth [IFN-al
23                                     Over 300 cytostatic agents selected from the National Cancer Inst
24  regions of a tumor and release cytotoxic or cytostatic agents; several of these HAPs are currently i
25 erties have been reported previously for the cytostatic agents shown here to up-regulate beta-chemoki
26           These compounds are widely used as cytostatic agents, so this enzyme should be studied as a
27                                              Cytostatic agents targeted against angiogenesis and tumo
28                        Rapamycin is a potent cytostatic agent that arrests cells in the G1 phase of t
29     Exposure of motor neuron cultures to the cytostatic agent vincristine markedly decreased CAT leve
30 sintercalators (8, 9, 12, and 13) behaved as cytostatic agents, while the monosubstituted acridine an
31             These results suggest that novel cytostatic agents with efficacy against human prostate c

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