ライフサイエンスコーパス: damage to

1 f phonological impairments in the absence of damage to 15% (15/100).

2 to the client proteins preventing oxidative damage to [4Fe-4S] clusters.

3 phonological impairments in the presence of damage to 85% (46/54) and also reduced the incidence of

4 CTA expression deficits were associated with damage to a critical zone that included the posterior ha

5 ord being thrown so as to inflict worthwhile damage to a medium-sized animal over distances up to 25

6 Oomycete pathogens cause serious damage to a wide spectrum of plants.

7 ow that (i) three common macroalgae are more damaging to a common coral when they compete under CO2 c

8 is increasingly bound to PARP1, causing DNA damage to accumulate, a process rapidly reversed by rest

9 ible relevance of the different types of DNA damage to AD-in particular, those caused by HSV1-and mec

10 This research shows UV-induced damage to adenoviral proteins across the germicidal UV s

11 al economies but can also cause considerable damage to agriculture, particularly damage to lucrative

12 , and sufficient stability against radiation damage to allow for multiple images to be obtained with

13 cells offers a promising strategy to reduce damage to an allograft by the recipient's immune system.

14 allow a more detailed analysis of structural damage to assess disease progression in clinical routine

15 : Whereas excessive oxidant challenge causes damage to biomolecules, maintenance of a physiological l

16 Symptoms of DENV infection involve damage to blood vessels and, in rare cases, hemorrhage a

17 Er,Cr:YSGG laser, despite producing thermal damage to bone tissue, is comparable to that with conven

18 Overall, genetic damage to both TGEF domains altered TRIO catalytic activ

19 ons has the potential to result in permanent damage to brain function.

20 omes from double dissociation data, in which damage to brain structure A causes deficits in Task 1 bu

21 , and that these lesions are likely to cause damage to brain structure and function that extends beyo

22 aused by increased precipitation followed by damages to buildings associated with near-surface permaf

23 ny defensive mechanisms that limit or repair damage to cell components.

24 solve a long-standing mystery regarding how damage to cell membranes occurs during ferroptosis, an i

25 ell) and scalable microsampling with minimal damage to cells in the 8-cell embryo enabled duplicate a

26 Oxidative stress is damaging to cells and contributes to aging and neurodege

27 nit volume of tissue traversed and thus more damaging to cells relative to low-LET radiation such as

28 ctive oxygen species (ROS) induces oxidative damage to cellular constituents, ultimately leading to i

29 the HBoV1 NS1 protein does not cause obvious damage to cellular DNA or arrest of the cell cycle.

30 activity is prevented from doing collateral damage to cellular proteins are not well understood.

31 Evidence of alcohol-induced damage to cholangiocytes, but not ongoing alcohol abuse,

32 show that live-cell DNA-binding dyes induce damage to chromatin within seconds, and demonstrate a di

33 necessitates long residence times and causes damage to clusters during release.

34 d production of cytokines to prevent further damage to CNS tissue.

35 the virus and did not induce any measurable damage to co-incubated antibodies.

36 nd balance are most commonly associated with damage to cochlear and vestibular hair cells or neurons.

37 mation, to detect molecular level subfailure damage to collagen in mechanically stretched rat tail te

38 many citrus varieties that causes extensive damage to commercial crops worldwide.

39 bstacles, postponing repair of the offending damage to complete the cell cycle and maintain cell surv

40 GNE-7915, either prevented or restored mtDNA damage to control levels.

41 We show that anger is damaging to cooperation as it leads to retaliation and a

42 ether, these activities represent widespread damage to coral ecosystems through physical burial as we

43 ved warming was unlikely to cause widespread damage to coral reefs in the region, and the coral reef

44 essable animals without detectable radiation damage to critical organs, including bone marrow and kid

45 Damage to dentine leads to release of fossilized factors

46 that remove biofilm without causing physical damage to disks.

47 In this model, initial damage to DNA (genomic, telomeric, mitochondrial and epi

48 Importantly, copper-induced damage to DNA and lipids was significantly greater under

49 Damage to DNA from the metabolites of drugs and pollutan

50 Ionizing radiation produces clustered damage to DNA which is difficult to repair and thus more

51 s of RT on cancer cells arise primarily from damage to DNA.

52 damage to the inferior frontal gyrus, or via damage to dorsal lateral prefrontal cortex regions, resu

53 d by inhibition persist unchanged even after damage to downstream motor nerves.

54 adiation therapy and chemotherapy induce DNA damage to drive cells into apoptosis or senescence as ou

55 Damage to elastin in the extracellular matrix results in

56 rgical language assessment can help minimize damage to eloquent cortex during resective epilepsy surg

57 n, induced DNA fragmentation, and attenuated damage to endothelial cells independently of the express

58 US, aHUS, and secondary HUS are simultaneous damage to endothelial cells, intravascular hemolysis, an

59 Selective damage to endothelium midway between FAs and primary art

60 igh doses of ionizing radiation induce acute damage to epithelial cells of the gastrointestinal (GI)

61 ory distress syndrome, thus suggesting lower damage to epithelial cells.

62 (RFC) acts as the initial sensor of telomere damage to establish predominance of DNA polymerase delta

63 ian brain is a complex system, implying that damage to even a single functional area could have wides

64 Damage to excitatory synapses in the hippocampus occurs

65 s from post-mortem DNA fragmentation and DNA damage to experimental sequencing errors, and reproduces

66 has been suggested as a mechanism of tissue damage to explain optic radiation abnormalities seen in

67 cal lesions and functional markers of kidney damage to facilitate a potential noninvasive detection f

68 ohol, we provide data on alcohol dependence, damage to families, and the documented increase in alcoh

69 might be directly impacted by frost through damage to flower buds and reproductive structures.

70 n the HRGS system in promoting the oxidative damage to food proteins.

71 mplicated in the oxidative and glycosylative damage to food proteins.

72 For example, the amount of damage to forebrain neurotransmitter and neuromodulator

73 nificant, cannot substantially alleviate the damages to forest ecosystems.

74 fungus-associated extracellular DNA and less damage to fungal filaments, suggesting that neutrophil e

75 Introducing the term 'clustered damage to G-quadruplexes' we report here on the structur

76 ing productivity and moderating heat-induced damage to grain quality in the coming decades, without s

77 lls or parasites) without causing collateral damage to healthy or to host cells is complicated by the

78 is necessary to prevent immune cell-mediated damage to healthy tissues.

79 Damage to hematopoietic stem cells (HSCs) in the bone ma

80 Previous studies have linked cell damage to high energy dissipation rates (EDR) and have p

81 ategories of stress on the beetles: physical damage to hindwings, predation, desiccation, and cold sh

82 E2 treatment also prevented BCCAO-induced damage to hippocampal myelin sheaths and oligodendrocyte

83 king floods in September 2013 caused massive damage to homes and infrastructure across the Colorado F

84 hibition of PDGFRB reduces Mucorales-induced damage to host cells.

85 as a commensal while minimizing the risk of damage to host tissue.

86 de secreted toxins responsible for extensive damage to host tissues and evasion of the immune system

87 vertases and, consequently, avoiding serious damage to host tissues.

88 symbiotic microbes mediate pathogen-induced damage to hosts.

89 urce footprints are highly representative of damage to human health and biodiversity.

90 t operations and impacts result in monetized damages to human health and the environment.

91 ng plant and animal fluids cause devastating damage to humans, livestock, and agriculture worldwide,

92 ed that CTA-impaired rats had more extensive damage to IC2 than did unimpaired rats.

93 pulses, a technique allowing for very local damage to individual filaments.

94 ts, the bone-forming cells, greatly reducing damage to infected cells.

95 g of host-derived DNA limits immune-mediated damage to infected tissues.

96 sed in previous studies of lightning-induced damage to infrastructure materials to determine the effe

97 tive estimated expenses from climate-related damage to infrastructure without adaptation measures (he

98 l loss was a secondary effect of excitotoxic damage to innervating neurons, I exposed neurog1a morpha

99 n equipment malfunction or may cause serious damage to instruments due to strong electromagnetic fiel

100 e excision repair protein OGG1 for oxidative damage to interact with the damage-induced base 8-hydrox

101 etogenic stimuli that cause oxidative stress damage to islets.

102 ion, DNA plays an important role because any damage to its molecular structure can affect the whole c

103 t (NFL) concentration is a general marker of damage to large-caliber myelinated axons.

104 Ozone (O3 ) damage to leaves can reduce plant photosynthesis, which

105 itical determinant of the spread of embolism damage to leaves during drought.

106 Damage to left inferior frontal cortex has been associat

107 decreases will not avert serious stress and damage to life on Earth, and additional steps are needed

108 study species to avoid hydraulic failure and damage to living cells.

109 iderable damage to agriculture, particularly damage to lucrative crops.

110 e evidence that this may be a consequence of damage to lysosomal membranes by reactive oxygen species

111 h levels of these species in the cell causes damage to macromolecules including several proteins and

112 se through direct and irreversible oxidative damage to macromolecules, as well as disruption of redox

113 Colletotrichum graminicola may cause severe damage to maize, affecting normal development of the pla

114 ablation secondary to heat-sink effects and damage to major blood vessels; however, needle tract see

115 ani is a fungal pathogen causing substantial damage to many of the worlds' largest food crops includi

116 spill accidents has brought about tremendous damages to marine ecological environment.

117 mage had impaired credit assignment, whereas damage to medial orbitofrontal cortex meant that patient

118 ynamic phosphorylation of DNA-PKcs after DNA damage to mediate NHEJ.

119 Microscale damages to membranes used in large-scale filtration proc

120 coming a humanitarian migrant is potentially damaging to mental health.

121 There are many more causes of non-enzymatic damage to metabolites than reactive oxygen species and f

122 om large amounts of cumulative, irreversible damage to microstructure during cyclic deformation, lead

123 Oxidative damage to mitochondria (MT) is a major mechanism for agi

124 Oxidative damage to mitochondrial DNA (mtDNA) in the retinal pigme

125 Ageing is associated with an accumulation of damage to mitochondrial DNA and proteins.

126 In particular, damage to mitochondrial proteins in skeletal muscle, whi

127 function, but the contribution of oxidative damage to morbidity is still debated.

128 n 15 fatalities, more than 260 injuries, and damage to more than 150 buildings.

129 pression of polyglutamine-expanded AR causes damage to motor neurons and skeletal muscle cells.

130 complex immune response resulting in chronic damage to multiple tissues.

131 h surface runoff, groundwater discharge, and damage to municipal water infrastructure.

132 y of targeted killing and prevent collateral damage to neighboring healthy cells.

133 For example, damage to neuromodulatory neurotransmitter systems is co

134 eptors, is a mechanism that causes secondary damage to neurons.

135 as it substantially minimises the radiation damage to non-tumour cells of healthy tissues.

136 s numerous, but their implementation costly, damaging to non-target species, and ineffective at low p

137 high-LET radiation induces greater long-term damage to normal lung tissue than the relative equivalen

138 experience radiation-induced toxicity due to damage to normal tissue in the irradiation field.

139 Severe late damage to normal tissue is a major limitation of cancer

140 ieving an optimal therapeutic index to avoid damage to normal tissue is extremely important.

141 re often needed during RT, leading to severe damages to normal tissues adjacent to tumors.

142 clude that S. aureus uses CodY to limit host damage to only the most severe starvation conditions, pr

143 o maintain tissue homeostasis despite severe damage to organelles.

144 has superior penetration and causes far less damage to organic molecules than UV.

145 of lost neurons may be insufficient because damage to other neurons and non-neuronal cells is common

146 ulation in healthy participants, we show how damage to our transcranial magnetic stimulation-guided r

147 ral neuropathy (CIPN) arises from collateral damage to peripheral afferent sensory neurons by antican

148 se activity could be activated following DNA damage to phosphorylate specific DNA repair proteins and

149 This is crucial to prevent photo-oxidative damage to photosystem II (PSII) and is controlled by the

150 Drought can cause major damage to plant communities, but species damage threshol

151 GluRs induces hair-cell death independent of damage to postsynaptic terminals.

152 ulnerability of breast CSCs to mitochondrial damage to potently and selectively kill breast CSCs.

153 ility of using AE immediately after cochlear damage to prevent or alleviate the emergence of central

154 for using sound therapy soon after cochlear damage to prevent the development of central processing

155 ystem that protects the nucleus and DNA from damage to promote cell survival during confined migratio

156 Tornadoes cause loss of life and damage to property each year in the United States and ar

157 and form EETs at sites of airway epithelial damage to protect the host from infections in patients w

158 challenge increased lung levels of oxidative damage to proteins (3-nitrotyrosine), lipids (8-isoprost

159 Such oxidative damage to proteins may lead to the formation of insolubl

160 Chemical damage to proteins of the crystallin family, accumulated

161 re reactive oxygen species, thereby limiting damage to proteins.

162 tal effects on cellular functions by causing damages to proteins, lipids and nucleic acids.

163 large-scale screening trial to evaluate ADB damage to provenances of F. excelsior sourced from throu

164 vent and to mitigate the direct and indirect damages to public health and resources.

165 onic pillararenes caused detectable membrane damage to red blood cells or toxicity to human cells in

166 ate cardiomyocyte (CM) division after tissue damage to regenerate lost heart muscle.

167 mportant for action and emotion recognition, damage to regions in this network should lead to deficit

168 ghboring residue (Tyr393) in response to DNA damage to regulate p53-dependent apoptosis.

169 Damage to renal tubular epithelial cells by genetic, env

170 uka did not increase the risk of planthopper damage to rice fields.

171 raction methods that do not cause detectable damage to root cells or soil-inhabiting microbes, thus p

172 ectrostatic repulsion and abolishes membrane damage to S. oneidensis.

173 ans about dangerous solar bursts and prevent damage to satellites and power stations from space weath

174 All are characterised by damage to several seemingly disparate cellular processes

175 es' are sometimes claimed to be the cause of damage to ships at sea and to offshore structures.

176 SEM reveals persistent damage to some surviving outer hair cell stereocilia.

177 Photoactivation causes oxidative damage to specific histidine residues in the key protein

178 s deficits in Task 1 but not Task 2, whereas damage to structure B produces the reverse pattern of ef

179 oligos generated during repair of UV-induced damage to study that process at both mechanistic and sys

180 Thus, a key question is whether damage to such a substrate is the basis for the perceptu

181 processes usually occur without significant damage to surrounding host tissues, dysregulated/excessi

182 n 2007, coincident with the first reports of damage to TC1507 corn.

183 A1 pulley release and detect any collateral damage to the A2 pulley, interdigital nerves, or underly

184 hat a reduced cardiac autonomic function and damage to the adrenergic receptors may contribute to the

185 ase, and how it might be targeted to prevent damage to the aging choriocapillaris in AMD.

186 ted, and the consequences of immune-mediated damage to the allograft.

187 terventions cause morphologic and functional damage to the artery in situ.

188 Causing collateral damage to the B-cell genome during CSR and SHM, AID indu

189 In addition, as damage to the blood-brain barrier is one of the major co

190 al in lethally irradiated mice by mitigating damage to the BM vascular microenvironment.

191 the polymerization stress and the potential damage to the bonded interface have dominated research i

192 n the form of cavitation bubble collapse, on damage to the brain's perineuronal nets (PNNs).

193 rome (MetS) has been associated with chronic damage to the cardiovascular system.

194 UV-A exposed PG caused oxidative damage to the cell and significantly higher damage to th

195 damage to the cell and significantly higher damage to the cell membrane than GA + UV-A treatment, ex

196 Damage to the cerebral tissue structural connectivity as

197 Severity of acute ischemic damage to the choroidal, outer retinal and optic nerve h

198 Damage to the CNS can cause a differential spatio-tempor

199 into the CNTS by capillary force without any damage to the CNTS structure.

200 g time, freeze-thaw cycling caused much more damage to the coal strength.

201 ed by decreased corneal sensation because of damage to the corneal sensory fibers.

202 e acuity and the potential for life-changing damage to the coronary arteries that distinguish KD from

203 cies provides insights into the mechanism of damage to the D1 and D2 proteins under light stress.

204 when abnormal experience could cause lasting damage to the developing brain.

205 the developing human fetus and can result in damage to the developing central nervous system.

206 MENT Zika virus (ZIKV) can cause substantial damage to the developing human brain.

207 s include loss of neuronal progenitor cells, damage to the developing vascular system of the brain, a

208 or photo-reconfiguration, while avoiding any damage to the DNA structures.

209 We found that damage to the dorsolateral prefrontal cortex (dlPFC) led

210 n in BM B cell lymphopoiesis associated with damage to the endosteal BM niche.

211 but also leads to the lowest total N loss or damage to the environment.

212 esistance exercise-induced, Ca(2+)-dependent damage to the fibre by the reduction of [Ca(2+)]cyto to

213 (PPP), resulting in a reduction in oxidative damage to the flight muscles.

214 ger without the potential risk of structural damage to the framework.

215 st organic solvent free, and causes far less damage to the fruit.

216 Damage to the gastrointestinal tract is a major cause of

217 We propose a model for NSAID-induced damage to the gastrointestinal tract that includes these

218 Oxidative damage to the genome can yield the base 8-oxo-7,8-dihydr

219 These contribute to structural damage to the GI tract and systemic translocation of GI

220 duces heparanase expression in the podocyte, damage to the glycocalyx, proteinuria, and renal failure

221 obility due to increased doping arising from damage to the GNR edges.

222 associated with dose-dependent, irreversible damage to the heart.

223 ic interventions to minimize further genetic damage to the hepatocyte population should be reconsider

224 its and disability as a result of structural damage to the hippocampal memory system.

225 This deficit pattern suggests differential damage to the hippocampus and basal ganglia (specificall

226 adequate immunotherapy to prevent persistent damage to the hippocampus.

227 immune response, thereby limiting collateral damage to the host and the risk for sepsis.

228 microbial protection with minimal collateral damage to the host.

229 t excessive inflammation to limit collateral damage to the host.

230 or T cell expansion and limiting nonspecific damage to the host.

231 the transmitted virus determines the initial damage to the immune system, setting the pace for later

232 lly repaired, and 2 incidents of inadvertent damage to the implant during surgery (P3 and P4).

233 ion recognition can be seen by either direct damage to the inferior frontal gyrus, or via damage to d

234 We observed that damage to the inferior temporal gyrus, to the fusiform g

235 Prevailing theories predict that damage to the input of the hierarchy will detrimentally

236 e that oral acrolein exposure in mice caused damage to the intestinal epithelial barrier, resulting i

237 Damage to the lateral hypothalamus (LH) causes profound

238 details of energy required to cause visible damage to the lung parenchyma are lacking.

239 itiating immunity without causing collateral damage to the lungs because of an exaggerated inflammato

240 In two experiments, patients with damage to the medial temporal lobe (MTL) and healthy con

241 a(2+) levels ([Ca(2+)]cyto), which can cause damage to the muscle.

242 ibition is due to FICZ/UVA-induced oxidation damage to the NER proteome and DNA excision repair is im

243 duced skin cancer by promoting photochemical damage to the NER proteome and thereby preventing the re

244 ght serve as objective markers of structural damage to the nervous system.

245 vered a cytoskeletal structure that prevents damage to the nucleus during migration in confined micro

246 on asymmetrically and deterministically more damage to the old daughter, the one receiving the mother

247 , after optic neuritis, there is progressive damage to the optic radiations, greater in patients with

248 bottom-contact configuration to minimize the damage to the organic semiconductors, for the first time

249 ensory neuron-macrophage communication after damage to the peripheral nerve.

250 are well known, the contribution of hypoxic damage to the peripheral nervous system (PNS) has not be

251 In nephrotic syndrome, damage to the podocytes of the kidney produces severe hy

252 etween the rate of regularization errors and damage to the posterior half of the left middle temporal

253 to identify the cause of initial excitotoxic damage to the postsynaptic neuron.

254 Unilateral damage to the primary visual cortex (V1) leads to clinic

255 ent ROS release from the active site, lessen damage to the protein, and produce higher total turnover

256 e evidence that transradial procedures cause damage to the RA, and discuss the impact this could have

257 ere observed in 74% (40/54) of patients with damage to the regions and 21% (21/100) of patients spari

258 corneal transplantation revealed unexpected damage to the retina and optic nerve in chemically burne

259 s and Relevance: Zika virus can cause severe damage to the retina, including the internal and externa

260 ring cross-section of biomaterials and X-ray damage to the sample.

261 photo-chemical effects such as light-induced damage to the sample.

262 feedback positioning control without risking damage to the SECM probe, we implement cyclic voltammetr

263 y responses and increased risk of alloimmune damage to the second allograft.

264 imers (CPD) in the skin, which further cause damage to the skin cells.

265 disease causes immune-mediated inflammatory damage to the small intestine.

266 stagmus, a highly symptomatic consequence of damage to the substrates of oculomotor control, often is

267 n intact biofilms and measured the impact of damage to the surrounding EPS-matrix.

268 reat microvessels without causing collateral damage to the surrounding tissue.

269 patients tested, only the four patients with damage to the temporoparietal junction showed impaired s

270 letely protects mice against ZIKV-associated damage to the testes and sperm and prevents viral persis

271 eron knockout mice to ZIKV results in severe damage to the testes, epididymis and sperm.

272 trochemical properties that induce extensive damage to the tissue and significantly degrade the long-

273 Damage to the urinary barrier initiates bladder pain and

274 Damage to the ventromedial frontal lobe also impaired le

275 n antiviral effect on RSV by inducing direct damage to the viral envelope, disrupting viral particles

276 This brief review examines damage to the visual system in both humans and animal mo

277 in a tubewell was positively associated with damage to the well pad for DTWs, the amount of human loa

278 Toxic leukoencephalopathy results from damage to the white matter caused by various toxins.

279 Radiation damage to the whole lung is a serious risk in nuclear ac

280 of wild-type (WT) mice; however, such severe damages to the GI significantly decreased in fat-1 trans

281 s on the oil metabolisms to avoide oxidative damages to the imbibed seeds, and the seed shell played

282 A polyphagous insect, it is particularly damaging to the billion dollar cole crop industry.

283 ictyota) cervicornis were significantly more damaging to the coral Acropora intermedia growing in the

284 und that sugar-fed moths had lower oxidative damage to their flight muscle membranes than unfed moths

285 Some people who are blind due to damage to their primary visual cortex, V1, can discrimin

286 ing abilities between those with and without damage to these 'transcranial magnetic stimulation-guide

287 of learned vocal behaviors because bilateral damage to this area renders humans unable to speak but h

288 ial prefrontal cortex (vmPFC) in humans, and damage to this region significantly increases the freque

289 mely sensitive to therapeutic radiation, and damage to this tissue results in various oral complicati

290 iation induces massive cell death and severe damage to tissues containing actively proliferating cell

291 Damage to tonometer prisms can be caused by sodium hypoc

292 w tool to inflict localized and irreversible damage to tumor tissue by hyperthermia, without harming

293 iderable losses of, and perhaps irreversible damages to, unique coral reef ecosystems.

294 Oxidative stress is capable of causing damage to various cellular constituents, including DNA.

295 o that may be the underlying cause of severe damage to ventral root axons resulting in a Wallerian-li

296 eatest loss of protein signature, indicating damage to viral proteins, occurred below 240 nm.

297 d efficacy has been attributed to UV-induced damage to viral proteins.

298 entin and does not cause irreversible pulpal damage to vital teeth when the air pressure employed is

299 During assembly of PSII, oxidative damage to vulnerable assembly intermediate complexes mus

300 Damages to water filtration membranes during installatio