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1 le of a cancer/testis antigen that is a also damage-associated molecular pattern.
2 s that senses PtdSer on apoptotic cells as a damage-associated molecular pattern.
3 ecule that promotes airway inflammation as a damage-associated molecular pattern.
4 flammation and is therefore referred to as a damage-associated molecular pattern.
5 acrophages stimulated with Wt or Cot/tpl2 KO damage-associated molecular patterns.
6 lammation and innate immunity, in particular damage-associated molecular patterns.
7 platforms that form upon sensing microbe- or damage-associated molecular patterns.
8 nction as sensors of endogenous or exogenous damage-associated molecular patterns.
9 f pathogen-associated molecular patterns and damage-associated molecular patterns.
10  pathogen-associated molecular patterns, and damage-associated molecular patterns.
11 rom dying cells may also engage with TLRs as damage-associated molecular patterns.
12 eath trigger inflammation through release of damage-associated molecular patterns.
13 les such as granular enzymes, cytokines, and damage-associated molecular patterns.
14 artner of distinct receptors of microbe- and damage-associated molecular patterns.
15 33 might function as an alarmin triggered by damage-associated molecular patterns.
16 ffectors, such as cytokines, chemokines, and damage-associated molecular patterns.
17 istic biomarkers of mitochondrial damage and damage-associated molecular patterns.
18 early inflammatory responses to pathogen and damage-associated molecular patterns.
19 kers as important signals of sepsis, whereas damage-associated molecular patterns, a class of inflamm
20                                 For example, damage-associated molecular patterns activate an inflamm
21 luding calgranulin A (S100a8), an endogenous damage-associated molecular pattern and TLR4 agonist.
22 avenger receptor of the Ig family that binds damage-associated molecular patterns and advanced glycat
23         The lipoproteins acquire features of damage-associated molecular patterns and trigger first a
24 (i.e., presence of circulating pathogen- and damage-associated molecular patterns), and their implica
25 isms by which different modes of cell death, damage-associated molecular patterns, and specific cell
26 ological signals induced by pathogen- and/or damage-associated molecular patterns are essential for a
27 unt for the chronic autoimmune response when damage-associated molecular patterns are released after
28                      Cytosolic pathogen- and damage-associated molecular patterns are sensed by patte
29               The data suggest a paradigm of damage-associated molecular pattern-based signaling wher
30 ed molecular pattern-based signaling whereby damage-associated molecular pattern binding integrins co
31 ognition of microbe, pathogen-associated and damage-associated molecular patterns by cytosolic sensor
32 e release of inflammatory mediators, such as damage-associated molecular patterns, by dying cells was
33 c anticancer treatment, tumor-derived DAMPs (damage-associated molecular patterns) can be sensed by i
34 ptors and, when challenged with pathogen- or damage-associated molecular patterns, can deliver cell-a
35 in-like lectin-G (Siglec-G) by noninfectious damage-associated molecular patterns controls innate imm
36 n pneumonia and increased levels of the mito-damage-associated molecular pattern (DAMP) cardiolipin c
37 vely during cell death; it is the prototypic damage-associated molecular pattern (DAMP) molecule and
38 ld-inducible RNA-binding protein (CIRP) is a damage-associated molecular pattern (DAMP) molecule whic
39 age through the recognition of extracellular damage-associated molecular pattern (DAMP) molecules.
40 ages stimulated by injury-induced release of damage-associated molecular pattern (DAMP) molecules.
41                                   Downstream damage-associated molecular pattern (DAMP) pathway activ
42                                Activation of damage-associated molecular pattern (DAMP) receptors and
43 trast-enhanced MRI, resulted in an immediate damage-associated molecular pattern (DAMP) response incl
44 d, Brennan et al report that a noninfectious damage-associated molecular pattern (DAMP), heparan sulf
45                               Uric acid is a damage-associated molecular pattern (DAMP), released fro
46 ile "danger signals" that are constituted by damage-associated molecular patterns (DAMP).
47 sociated immunostimulatory endogenous danger/damage associated molecular patterns (DAMPs) and a reduc
48 role of the inflammasome from one of sensing damage associated molecular patterns (DAMPs) to sensing
49 rly, cellular injury can release endogenous 'damage'-associated molecular patterns (DAMPs) that activ
50                            Mitochondrial DNA damage-associated molecular patterns (DAMPs) accumulate
51 n-associated molecular patterns (PAMPs), and damage-associated molecular patterns (DAMPs) activate fa
52 eds are carried out through the detection of damage-associated molecular patterns (DAMPs) and a respo
53  the cell content and subsequent exposure of damage-associated molecular patterns (DAMPs) and cytokin
54 ammasome activation in response to metabolic damage-associated molecular patterns (DAMPs) and discuss
55 ring infection, certain S100 proteins act as damage-associated molecular patterns (DAMPs) and interac
56                                              Damage-associated molecular patterns (DAMPs) are conside
57                                              Damage-associated molecular patterns (DAMPs) are critica
58                                              Damage-associated molecular patterns (DAMPs) are molecul
59                                              Damage-associated molecular patterns (DAMPs) are release
60           Although endogenous RNAs acting as damage-associated molecular patterns (DAMPs) for pattern
61 crotic cell death with subsequent release of damage-associated molecular patterns (DAMPs) is a charac
62 ng TLR4-dependent cytokine storm mediated by damage-associated molecular patterns (DAMPs) like HMGB1.
63 om dead and dying cells can be recognized as damage-associated molecular patterns (DAMPs) or pattern-
64 sociated molecular patterns (PAMPs/MAMPs) or damage-associated molecular patterns (DAMPs) recognized
65                               TLR binding to damage-associated molecular patterns (DAMPs) released by
66 LR4 (Ahmad et al., 2014) and ligands such as damage-associated molecular patterns (DAMPs) released fr
67              We propose that inflammatogenic damage-associated molecular patterns (DAMPs) released in
68 ctivated in response to structurally diverse damage-associated molecular patterns (DAMPs) such as ext
69                               The concept of damage-associated molecular patterns (DAMPs) was propose
70 cluding death of neural cells and release of damage-associated molecular patterns (DAMPs), and progre
71                Endogenous danger signals, or damage-associated molecular patterns (DAMPs), are genera
72                  Signals of tissue necrosis, damage-associated molecular patterns (DAMPs), cause infl
73 ry molecules released by dying cells, termed damage-associated molecular patterns (DAMPs), have been
74  two, which involves recognition of PAMPs or damage-associated molecular patterns (DAMPs), such as ur
75                            In SI, endogenous damage-associated molecular patterns (DAMPS), which are
76 endogenous signals referred to as danger- or damage-associated molecular patterns (DAMPs), which are
77 o-IL-1beta to a mature form is stimulated by damage-associated molecular patterns (DAMPs).
78 cognition of markers of tissue injury termed damage-associated molecular patterns (DAMPs).
79  alerted to cell death by molecules known as damage-associated molecular patterns (DAMPs).
80 , a CREB inducer and sensor for host-derived damage-associated molecular patterns (DAMPs).
81 th by necroptosis, accompanied by release of damage-associated molecular patterns (DAMPs).
82  activated by TLR ligands, IL4, TGFbeta, and damage-associated molecular patterns (DAMPS).
83 ndritic cells and macrophages in response to damage-associated molecular patterns (DAMPs).
84 vate plant innate immunity by functioning as damage-associated molecular patterns (DAMPs).
85  endogenous triggers of this process are the damage-associated molecular patterns (DAMPs).
86 gen-associated molecular patterns (PAMPs) or damage-associated molecular patterns (DAMPs).
87 e also been implicated in the recognition of damage-associated molecular patterns (DAMPs).
88 f immunostimulatory molecules referred to as damage-associated molecular patterns (DAMPs).
89 s can be considered to represent "danger (or damage)-associated molecular patterns" (DAMPs).
90 reas others bind endogenous plant compounds (damage-associated molecular patterns, DAMPs) such as pep
91 s emit immunostimulatory signals (so-called "damage-associated molecular patterns," DAMPs), as they d
92 ndogenous molecules released by dying cells (damage-associated molecular patterns; DAMPs) activate ce
93  similar signaling pathways are activated by damage-associated molecular patterns, epigenetic plastic
94 chondrial perturbations, which function as a damage-associated molecular pattern, exacerbate NLRP3 in
95                       CIH-induced release of damage-associated molecular patterns from injured CNS ce
96                 Intraperitoneal injection of damage-associated molecular patterns from necrotic hepat
97          Transcriptional hyper-activation of damage-associated molecular pattern genes occurs followi
98   Pathogen-associated molecular patterns and damage-associated molecular patterns have been found to
99 or blood transfusion releases the endogenous damage-associated molecular pattern, hemoglobin (Hb), in
100 ellular energy source, ATP can also act as a damage-associated molecular pattern in both animals and
101 onnections include responses to pathogen and damage-associated molecular patterns including alarmins
102 -associated molecular patterns as well as by damage-associated molecular patterns, including microRNA
103                    Both pathogen- and tissue damage-associated molecular patterns induce inflammation
104            This is associated with increased damage-associated molecular patterns, innate cytokine re
105 e airway microbiome, antimicrobial proteins, damage-associated molecular patterns, innate lymphoid ce
106 t enhancing mitochondrial resilience against damage-associated molecular patterns may play a pivotal
107 haracterize the pathophysiologic response to damage-associated molecular pattern mediated organ injur
108 n HMGB1 is released from cells and acts as a damage-associated molecular pattern molecule during many
109                                          The damage-associated molecular pattern molecule high-mobili
110                              Thus, CIRP is a damage-associated molecular pattern molecule that promot
111 lucidated the mechanism by which the nuclear-damage-associated molecular pattern molecule, high-mobil
112  aim was to investigate the role of IL-33, a damage-associated molecular pattern molecule, in adenovi
113 associated nuclear protein and extracellular damage-associated molecular pattern molecule, is a criti
114  EDA(+) isoform of fibronectin (EDA(+)Fn), a damage-associated molecular pattern molecule, which prom
115  show that reactive astrocytes can release a damage-associated molecular-pattern molecule called high
116                                 In contrast, damage-associated molecular pattern molecules (DAMPs) ar
117 mediates the cellular response to a range of damage-associated molecular pattern molecules (DAMPs) in
118 oids and a neutralizing antibody targeted at Damage-Associated Molecular Pattern molecules (DAMPs) su
119 e inflammatory response upon engagement with damage-associated molecular pattern molecules (DAMPs) su
120  extracellular compartment where they act as damage-associated molecular pattern molecules (or alarmi
121 ty group box-1 and S100A9, both of which are damage-associated molecular pattern molecules and are kn
122 y of RAGE is dictated by the accumulation of damage-associated molecular pattern molecules at sites o
123 either by bacterial lipopolysaccharide or by damage-associated molecular pattern molecules released f
124 ermore, it has been ascribed to the group of damage-associated molecular pattern molecules that promo
125 g, perturbations in calcium homeostasis, and damage-associated molecular pattern molecules, among oth
126 genous ligands from damaged cells, so-called damage-associated molecular pattern molecules, can activ
127 ) is a pattern recognition receptor for many damage-associated molecular pattern molecules.
128 he local inflammatory milieu by pathogen- or damage-associated molecular pattern, molecules, and acti
129 stem is primarily initiated by pathogen- and damage-associated molecular patterns on cellular surface
130 be triggered by endogenous mediators, called damage associated molecular patterns or alarmins.
131    Innate immune receptors for pathogen- and damage-associated molecular patterns (PAMPs and DAMPs) o
132                         Molecules containing damage-associated molecular patterns play an important r
133                                          The damage-associated molecular pattern protein HMGB1 has be
134 ng evidence that S100B acts as a cytokine or damage-associated molecular pattern protein not only in
135             Neuroinflammation is sterile, as damage-associated molecular patterns rather than microbi
136 to pathogen-associated molecular pattern and damage-associated molecular pattern recognition by the i
137 eceptors for sensing microbial molecules and damage-associated molecular patterns released from host
138 se microbe-associated molecular patterns and damage-associated molecular patterns represent a conserv
139 echanism of cross-talk between pathogen- and damage-associated molecular pattern-sensing pathways, wh
140             During infection, the release of damage-associated molecular patterns, so-called "alarmin
141 EI11 expression is controlled by PME-related damage-associated molecular patterns, such as oligogalac
142 triphosphate (ATP) is released and acts as a damage-associated molecular pattern that activates innat
143 ty group B1 (HMGB1) is a recently identified damage-associated molecular pattern that is released dur
144 nment generates versikine, a novel bioactive damage-associated molecular pattern that may facilitate
145 e is detected by TLR3 and that self-RNA is a damage-associated molecular pattern that serves as an en
146 dies reveal that melanocyte stress generates damage-associated molecular patterns that activate innat
147 terile inflammation), necrotic cells release damage-associated molecular patterns that bind to Toll-l
148 , necrotic, or apoptotic hepatocytes release damage-associated molecular patterns that initiate steri
149               However, upon stimulation with damage-associated molecular patterns, the activation of
150 ular triggers such as pathogen-associated or damage-associated molecular patterns, the NLRP1 inflamma
151 rs (TLRs), sense and respond to pathogen- or damage-associated molecular patterns, their communicatio
152 mune cells or necrotic tissues and acts as a damage-associated molecular pattern to activate Toll-lik
153 cellularly released, galectin-3 can act as a damage-associated molecular pattern to facilitate early
154 ve microbe-associated molecular patterns and damage-associated molecular patterns to activate innate
155 ciated molecular patterns and cell-intrinsic damage-associated molecular patterns to contextualize th
156 ansduction not only during MTI but also upon damage-associated molecular pattern-triggered immunity,
157 erapy, intestinal commensal bacteria and the damage-associated molecular pattern uric acid contribute
158                                 One of these damage-associated molecular patterns, uric acid, is incr
159           The alarmins S100A8 and S100A9 are damage-associated molecular patterns, which play a pivot
160 rtant oxidation-derived Th2 immunomodulatory damage-associated molecular pattern with potentially imp

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