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1 lating host immunity through mechanisms that dampen inflammation.
2 33R up-regulation on CD8 effector T cells to dampen inflammation.
3 by killing B lymphocytes as a rapid means to dampen inflammation.
4 ytosis) prevents post-apoptotic necrosis and dampens inflammation.
5 ed by a lower intestinal parasite burden and dampened inflammation.
6 iption factor (TF) activating TF 3 (ATF3) in dampening inflammation.
7 rolonged survival of Trex1(-/-) mice without dampening inflammation.
8 nsatory T(reg) activation by TLR ligation to dampen inflammation and autoimmune diseases.
9                     Hence, therapeutics that dampen inflammation and enhance resolution are of consid
10 ed down-regulation of JNK activity serves to dampen inflammation and tissue injury.
11  harboring a microbiota, neutralizing IL-17A dampened inflammation and restored lung function.
12 H2 adaptive immune responses associated with dampened inflammation and tissue repair are elicited in
13 is an enzyme that generates adenosine, which dampens inflammation and improves vascular barrier funct
14 ovel strategy to rebalance T-cell responses, dampen inflammation, and ultimately improve outcomes for
15 tochondria, thus counteracting degeneration, dampening inflammation, and preventing unwarranted cell
16 re small lipid mediators that act locally to dampen inflammation, but they have not been linked direc
17 the phosphatidylserine recognition receptors dampen inflammation by inducing the production of anti-i
18 aps by reducing cell attachment/invasion and dampening inflammation by maintaining/inducing IL-10 pro
19 leukin 13 (IL-13)-dependent regulatory loop, dampens inflammation by promoting the egress of inflamma
20 inical potential of sialidase inhibition for dampening inflammation caused by infection.
21 with alpha1-antitrypsin (AAT), an agent that dampens inflammation, does not directly inhibit T cell a
22  a non-T-cell regulatory pathway involved in dampening inflammation during repeated exposure to A. fu
23 tion of matriptase from LEKTI-deficient mice dampened inflammation, eliminated aberrant protease acti
24 ts of type I IFNs may therefore be useful in dampening inflammation in lung diseases characterized by
25  targets for therapeutic strategies aimed at dampening inflammation in pathologies such as chronic ob
26                               The ability to dampen inflammation is imperative for prevention of chro
27 Anti-inflammatory cytokines had some role in dampening inflammation, since interleukin-10 (IL-10)-def
28                                   Genes that dampen inflammation (Socs3, Il10, Crem, Stat3, Thbd, Thb
29                                              Dampening inflammation through signaling via the adenosi
30 fy additional Klebsiella factors required to dampen inflammation, we standardized and applied a high-

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