ライフサイエンスコーパス: dark period

1 recorded for 20 h (8-hour light and 12-hour dark period).

2 ed with a 16-hour light period and an 8-hour dark period.

3 nd the peak appeared at the beginning of the dark period.

4 d but only in the supine position during the dark period.

5 ite light (W) night break given 8 h into the dark period.

6 Sleep was encouraged in the dark period.

7 the light-wake period and supine during the dark period.

8 adults, with the peaks occurring in the late dark period.

9 ight-induced processes separated by a slower dark period.

10 re they were all but undetectable during the dark period.

11 nes were negatively affected after the first dark period.

12 and had low values (165-574 ppm) during the dark period.

13 nificantly higher delta power throughout the dark period.

14 ng the SD procedure was decreased during the dark period.

15 rom late afternoon into the first 2 h of the dark period.

16 uring both wakefulness and NREM sleep in the dark period.

17 period but more time in SWS and REMS in the dark period.

18 f leaves to export sugars during a prolonged dark period.

19 rease of active wake was observed during the dark period.

20 sured delta18O discrimination throughout the dark period.

21 midday and the low point midway through the dark period.

22 except for an increase in the middle of the dark period.

23 day, and modest decrease at the start of the dark period.

24 -type plants, especially at the start of the dark period.

25 nd of the light period and a peak in the mid-dark period.

26 ificantly inhibited food intake in the early dark period.

27 ce also had less non-REMS (NREMS) during the dark period.

28 nsufficient carbohydrate reserves during the dark period.

29 in the kinetics of response during the 5-day dark period.

30 malto-oligosaccharides generated during the dark period.

31 ) mRNA, and oscillations continued in a 48 h dark period.

32 fferent phase and remained low during a 48 h dark period.

33 Sleep was encouraged in the dark period.

34 nd the peak appeared at the beginning of the dark period.

35 be thoroughly degraded toward the end of the dark periods.

36 the early light period (2-6 h) and the late dark period (4-6 h).

37 ant loss of photosynthetic efficiency during dark periods, a greater level of oxidative stress, and r

38 d PsaB was maximal in the dark or subjective-dark periods, a period during which PSI was primarily in

39 exhibited reductions in REM during the 12-h dark period after contextual fear, whereas mice receivin

40 PIF1 reaccumulates in the subsequent dark period after light-induced degradation, signifying

41 upted baseline and during 8-h light and 12-h dark periods after three sessions of 5-min manual restra

42 o basal forebrain during the normally active dark period also increases ATP.

43 nviable in cycling conditions when light and dark periods alternate.

44 operiod, reaches a peak in the middle of the dark period and correlates with kinase activity.

45 during wakefulness in both the light and the dark period and during both wakefulness and NREM sleep i

46 ff followed by recording throughout the 12-h dark period and following 12-h light period.

47 in the marmosets: IOP was higher during the dark period and lower during the light period (mean chan

48 n concentrations were the highest during the dark period and lowest in the light period.

49 1 levels then gradually increase through the dark period and remain high following movement of Aaop1

50 ep-to-wake transitions are affected by light/dark period and sleep pressure.

51 creased total sleep, NREM and REM during the dark period and total sleep and NREM during light period

52 e approximately 1.6 times more active during dark periods and approximately 4 times more active durin

53 was dependent on both the ratio of light to dark periods and JH III.

54 d total sleep and NREM during both light and dark periods and significantly increased dark period REM

55 d, as demonstrated by intermittent light and dark periods and thus allowing access to spatiotemporal

56 level at 4 to 10 h in the dark or subjective-dark periods and were shown by Western blotting and elec

57 of the human light-dark cycle, but the mouse dark period) and the rest phase (the human dark period,

58 (NREM) sleep by approximately 43% during the dark period, and increased delta power in the EEG during

59 arch is degraded progressively during a 12-h dark period, and then accumulates during the following 1

60 h glucans and lipids was observed during the dark period, and transcription profile data indicated th

61 At the end of this dark period, animals were exposed to constant light of 2

62 Transitions between light and dark periods appear to be major environmental events tha

63 and increased NREM and REM sleep during the dark period, as previously reported, and unexpectedly de

64 d to determine if light intensity (including dark periods) at time of harvest impacts concentrations

65 ference is projected to be higher during the dark period because of the change in IOP.

66 awakening event throughout the entire light/dark period but that this effect was diminished with sle

67 e dark period) and the rest phase (the human dark period, but the mouse light period), but also synch

68 em promotes wakefulness throughout the light/dark period by activating multiple downstream targets, w

69 PPC displayed up to a 66% reduction in total dark period CO2 fixation.

70 hen a chilling stress was applied during the dark period, concomitant with an increase in ABA levels.

71 sands of genes at the end of the reoccurring dark periods (dawn), including those involved in photosy

72 ght stimulation of both genotypes during the dark period did not change the Avp expression in the SCN

73 r SCN samples collected during the light and dark periods did show differences in expression and as s

74 e is readily lost during protracted (1-10 s) dark periods during photoactivation of Synechocystis cel

75 duced removal of Pfr at the beginning of the dark period (End-of-Day-FR (EOD-FR) treatment) results i

76 -)mfERG stimulates with flashes separated by dark periods, facilitating interpretation of late first-

77 The dark period had a differential effect on (13)C incorpora

78 imals had increased food intake in light and dark periods, higher weight gain per day, and more body

79 copic lights at the beginning and end of the dark period; (iii) wearing either +6 D lenses, -6 D lens

80 However, when the dark period in a normal diurnal cycle was cut short arti

81 d increase in ambulatory activity during the dark period in comparison to the light period and a 'W-s

82 prolongs the activity of PPC throughout the dark period in K. fedtschenkoi, optimizing CAM-associate

83 The changes in fru-2,6-P2 at the start of dark period in leaves and in the cell experiments genera

84 r) neurons during the light period and early dark period in photostimulated vs. control animals.

85 with increased oxidative stress late in the dark period in the mutant.

86 Prolonged light treatment followed by a dark period induced stress and cell death marker genes w

87 ormone release, especially at the end of the dark period, maintains high temperature.

88 was projected to be 5.26 mm Hg higher in the dark period (mean, 17.10 mm Hg) than in the light period

89 tle light exposure before entering the night/dark period, MeSA and its metabolizing enzymes were esse

90 and PEPC carboxylations alone, such that the dark period mesophyll conductance, g(i), was 0.044 mol m

91 sponse by 26% only in wakefulness during the dark period of the diurnal cycle to a level observed dur

92 noparticles, the distributions of bright and dark periods ('on' and 'off' times) follow Levy statisti

93 activity was significantly higher during the dark period (P < 0.01).

94 hotoperiod (phase IV of CAM), throughout the dark period (phase I), and into the light (phase II).

95 PPCK phosphorylates PPC during the dark period, reducing its sensitivity to feedback inhibi

96 NPY, injected at the start of the dark period, reliably increased 2 h food intake.

97 and dark periods and significantly increased dark period REM.

98 55 (36.9%) of the 149 neurones tested in the dark period responded to optic nerve stimulation while o

99 ulation of this truncated GluTR is higher in dark periods, resulting in increased protochlorophyllide

100 ements of CO2 response curves throughout the dark period revealed changing phosphoenolpyruvate carbox

101 e flash experiments, with a 10 s intervening dark period, reveals a faster, 15 ms phase that is accen

102 avenous infusion of ghrelin during the early dark period stimulates food intake in freely feeding rat

103 Average IOP was significantly higher in the dark period than in the light-wake period in both groups

104 y, while the abundance of HMG1 mRNA during a dark period that induced photoperiodically controlled fl

105 MSR activity occurred at the end of the 16-h dark period that was absent in pmsr2-1 plants.

106 display high levels of basal activity in the dark period (the rodent's awake/active time) that are at

107 During the dark period, the mean spontaneous firing rate (5.00 +/-

108 13C signal; all declined at the start of the dark period, then increased to a maximum 2 h before dawn

109 nes exhausted their carbohydrates during the dark period to a greater extent than the wild type and a

110 respiration in the early dark or subjective-dark periods to permit nitrogenase activity.

111 Both NE and mNE increased dark period total sleep, NREM and REM; however, mNE also

112 he nifHDK operon during the first 4 h of the dark period under light-dark conditions or during the fi

113 increases linearly with the duration of the dark period up to the longest period we could examine (1

114 as high after a 14-h night, but low when the dark period was 12 h or less.

115 Mean IOP in the dark period was significantly higher than mean IOP in th

116 ning of the light period or beginning of the dark period, we sought to determine whether the muscarin

117 for the 12-hour light period and the 12-hour dark period were compared.

118 wake period and the supine IOP data from the dark period were considered, elevation and reduction of

119 ings from the 10-h light period and the 12-h dark period were examined separately.

120 ns or during the first 6 h of the subjective-dark period when grown in continuous light.

121 tion of CNA on sleep and activity during the dark period when rats show higher arousal and less sleep

122 During the dark period, when these animals are normally active, 6 h

123 e-movement sleep (REMS) increased during the dark period, whereas during the light both NREMS and REM

124 -h intervals early in the dark or subjective-dark period, whereas photosynthesis was approximately 12

125 were injected systemically during the early dark period with melatonin (0.6 mg) or 2% ethanol vehicl

126 creases in slow-wave sleep (SWS) only in the dark period with no changes in rapid-eye-movement sleep

127 nd day periods and low movement rates during dark periods with highest nighttime rates at 10-<50% lun