ライフサイエンスコーパス: daughter

1 tivation of distinct fate programmes in each daughter.

2 between the father, who is a smoker, and his daughter.

3 air comprising an older mother and a younger daughter.

4 roportion to the cytoplasm inherited by each daughter.

5 microtubule structure does not template the daughter.

6 s that divide to produce small, mononucleate daughters.

7 priate terminal differentiation of posterior daughters.

8 y increased drug efflux activity relative to daughters.

9 ifferentiation of the prefollicle cell (pFC) daughters.

10 number allowing production of uniform-sized daughters.

11 e electrical conductance of such parents and daughters.

12 t fertility of resulting daughters and grand-daughters.

13 es supernumerous mitotic divisions and small daughters.

14 FGF8 from the other parent to both affected daughters.

15 d between NBs and their ganglion mother cell daughters.

16 h unique properties, distinct from displaced daughters.

17 levels compared to the younger ones and the daughters.

18 tching causes mother cells to shun their own daughters.

19 n (aged 64 years), and the recently affected daughter (aged 61 years) recounted non-contributory medi

20 at a faster exponential rate than the large daughter, an observation that potentially challenges pre

21 highest DNA preservation demonstrate mother-daughter and grandmother-grandson relationships, evidenc

22 oteins are distributed unequally between the daughter and mother cells.

23 eductions in hematocrit and flow rate in the daughter and mother vessels.

24 ntification of cell division events, mother, daughter and neighbouring cells, and computing statistic

25 ld potentially affect fertility of resulting daughters and grand-daughters.

26 ers, whereas there was no difference between daughters and mothers.

27 te the gradual aging of the mother cell, all daughters are born rejuvenated and enjoy a full replicat

28 les: mothers are physically removed, whereas daughters are eliminated in the cytoplasm, preparing the

29 itioned into the daughter cells, and how the daughters are positioned within the tissue.

30 l cellular materials are partitioned between daughters at cell division, but by various mechanisms an

31 d childhood growth factors with adult BMI in daughters at midlife using quantile, linear, and logisti

32 change in early childhood growth with BMI in daughters at midlife.

33 Cytokinesis cleaves a cell into two daughters at the end of mitosis, and must be spatially c

34 Displaced WNT(lo) suprabasal daughters become SCs that respond to paracrine SHH and s

35 icrotubules of T. gondii, established during daughter biogenesis and regulated by protein expression

36 rogram father's germ-line and modulate their daughters' birth weight and likelihood of developing bre

37 gregate size was marginally increased in the daughter branches for a range of flow rates, mainly due

38 re studied for a range of flow splits in the daughter branches of the bifurcation.

39 ype oligomer (m-OPE) is modified to yield a "daughter" by inserting one nitrogen atom into the m-OPE

40 g of damage by a mother bacterium to its two daughters can evolve through genetic assimilation.

41 The daughter CD8(+) T cells with disparate LFA-1 expression

42 cause BASL polarity is only exhibited by one daughter cell after an asymmetric cell division, we stud

43 elf-renew, clonally yielding a TCF1-silenced daughter cell as well as a sibling cell maintaining TCF1

44 Paired daughter cell assays demonstrate that Asxl2 loss enhance

45 ation being synchronous and coordinated with daughter cell assembly(2,3).

46 n the differences in c-Myc expression in one daughter cell compared to the other.

47 flows from the ring and enriches the nascent daughter cell cortices.

48 and divide in order to be inherited by each daughter cell during mitosis.

49 ar diversity and defects can lead to altered daughter cell fates and numbers.

50 tric cell division to ensure the fidelity of daughter cell formation.

51 nvironment contains sufficient resources for daughter cell generation.

52 at facilitate polarized vesicle delivery and daughter cell growth.

53 DNA asymmetrically, preferentially into the daughter cell harboring the young centrosome.

54 assembly checkpoint (SAC) ensures that each daughter cell inherits an identical set of chromosomes.

55 sma membrane in the furrow, and separate the daughter cell membranes.

56 Here, we show that most growth of a new daughter cell occurs in mitosis.

57 ndergo asymmetric cell division, wherein the daughter cell proximal to the APC is more likely to diff

58 dicated that flagella-based forces initiated daughter cell separation and provided a source for membr

59 sembly of a contractile ring, which promotes daughter cell splitting.

60 boost of egl-1 that occurs specifically in a daughter cell that is programmed to die.

61 re required for faithful transmission to the daughter cell to accurately maintain cell identity durin

62 All daughter cell types of engineered NSCs (neurons, astrocy

63 ell size, yielding a smaller differentiating daughter cell.

64 and, upon partitioning, reassembles in each daughter cell; however, it is not clear whether the disa

65 Our data reinforce the importance of daughter-cell-associated factors and centrosome-based re

66 tioning of the replicated chromosomes to the daughter cells (M phase) during eukaryotic cell division

67 replicated chromosomes are partitioned into daughter cells and can serve as a platform for the re-es

68 accurate partition of the cytoplasm between daughter cells and the correct localization of the daugh

69 Importantly, NPC-derived daughter cells appeared to be latently infected with HIV

70 Therefore, two daughter cells are differentiated by BASL polarity-media

71 In particular, budding yeast daughter cells are more vulnerable to stresses than the

72 e final stage of cell division where the two daughter cells are separated, is mediated by the endosom

73 red in partitioning of molecules between two daughter cells are significant.

74 n, culminating in the physical separation of daughter cells at the end of mitosis.

75 other cell produces only a limited number of daughter cells before it slows division and dies.

76 tric and asymmetric cell divisions producing daughter cells capable of self-renewal.

77 Thus, daughter cells control the proliferation-quiescence deci

78 Daughter cells derived from PGCCs showed attenuated capa

79 l indeed biases stochastic fate decisions of daughter cells despite mitotic rounding.

80 to gene expression that can be passed on to daughter cells during cell division, whereas RNAi does n

81 MiQ aggregate deposit was not transferred to daughter cells during cell division.

82 ferentiation and the relative positioning of daughter cells during development.

83 ursors and prevents them from spreading into daughter cells during division by subjecting them to the

84 or disassembly, and their partitioning among daughter cells during division.

85 roper segregation of the genetic material to daughter cells during mitosis and meiosis.

86 faithful distribution of chromosomes between daughter cells during mitosis as well as for other cellu

87 To segregate the episomes into daughter cells during mitosis, they are tethered to cell

88 equal segregation of duplicated DNA into two daughter cells during mitosis.

89 stack is duplicated and partitioned into two daughter cells during the cell cycle of the protozoan pa

90 stomatal lineage but is inherited equally by daughter cells following an asymmetric cell division.

91 gets mRNAs acquired in the nucleus either to daughter cells for translation or to stress granules (SG

92 ing cell division is critical for preventing daughter cells from inheriting an abnormal number of chr

93 active transcriptional states from mother to daughter cells has the potential to foster precision in

94 terphase expression pattern is transduced to daughter cells have been unclear.

95 s complete mitosis, a fraction of newly born daughter cells immediately enter the next cell cycle, wh

96 on of [PSI(+) ] propagons between mother and daughter cells in a sub-population of cells during cell

97 -lived growth differences between mother and daughter cells in the presence of subinhibitory drug con

98 f cell division that defines the position of daughter cells in the tissue.

99 number plasmids, diffusion ensures that both daughter cells inherit plasmids after cell division.

100 ance proliferation with the incorporation of daughter cells into organ primordia.

101 ial amino acids to generate large numbers of daughter cells necessary for effective immunity to patho

102 erminants, ensuring asymmetric division into daughter cells of different fates.

103 of adult cells and demonstrate that the two daughter cells of many early embryonic cell-doubling eve

104 tudies indicate that replicative lifespan in daughter cells of Sacchraromyces cerevisiae depends on t

105 ne, NR4A3 that was downregulated in MKN28 GC daughter cells overexpressing a constitutively activated

106 selectively required for the maintenance of daughter cells produced by castration-resistant Nkx3.1-e

107 How do daughter cells re-establish the original transcription p

108 ions retract their basal processes, and both daughter cells regrow a new process following cytokinesi

109 regation of protein aggregates by mother and daughter cells remains controversial, in part because of

110 threshold necessary to trigger death only in daughter cells that are programmed to die.

111 ate within a parasitophorous vacuole to form daughter cells that eventually exit (egress) by sequenti

112 undergo asymmetric division, giving rise to daughter cells that exhibit distinct tendencies to adopt

113 After stem cell division, daughter cells that exit the stem cell domain acquire tr

114 nequally distributing factors to the nascent daughter cells that influence their eventual fate toward

115 tric divisions, increasing the proportion of daughter cells that inherit high amounts of effector fat

116 This additionally allows the daughter cells to be born damage free.

117 lly duplicated and accurately transmitted to daughter cells to preserve cell identity during the cell

118 alignment of the mitotic spindle allows the daughter cells to stay within the epithelium.

119 Feedback signals from daughter cells to stem cells are well studied and known

120 ytoplasmic components determines the fate of daughter cells upon asymmetric division.

121 d to ensure equal chromosome distribution in daughter cells upon mitosis.

122 h activation of senescence, while budding of daughter cells was associated with senescence escape.

123 d frequency at which circles redistribute to daughter cells was not due to changes of anaphase durati

124 on generates cellular diversity by producing daughter cells with different fates.

125 iability each time a cell divides, producing daughter cells with different sizes and growth rates.

126 e replicated copies of their genome to their daughter cells with extremely high fidelity.

127 iminate transmission of mRNAs from mother to daughter cells, and favors the response capacity of the

128 of the mother cell are partitioned into the daughter cells, and how the daughters are positioned wit

129 ve to errors in partitioning of volume among daughter cells, and not surprisingly, this process is we

130 faithful distribution of chromosomes between daughter cells, and spindle orientation is a major deter

131 pigenetic information is transmitted only to daughter cells, but evidence is emerging, in both verteb

132 ng the cleavage plane are pulled between the daughter cells, making a new interface between neighbors

133 nes involved in the growth and rebuilding of daughter cells, rather than cell type-specific functions

134 basement membrane is lost in differentiating daughter cells, where YAP and TAZ become mostly cytoplas

135 , repair them, or avoid their propagation to daughter cells, which would be particularly detrimental

136 a stable cytoplasmic bridge between the two daughter cells, Z2 and Z3 Depletion of several regulator

137 ired for their proper segregation to the two daughter cells.

138 l repartition of chromosomes between the two daughter cells.

139 e products of which proceeded to form viable daughter cells.

140 DSB at ori5, preventing mtDNA segregation to daughter cells.

141 consequently, abnormal bud-site selection in daughter cells.

142 arate replicated chromosomes accurately into daughter cells.

143 for asymmetric division and rejuvenation of daughter cells.

144 ng increasing mRNA synthesis rate in smaller daughter cells.

145 induced cyclin D1 (CCND1) mRNA to newly born daughter cells.

146 n asymmetric cell division yielding distinct daughter cells.

147 a severe decline of replicative life span of daughter cells.

148 ures accurate segregation of chromatids into daughter cells.

149 cate sufficient raw material to generate two daughter cells.

150 hromosome dimers during their segregation to daughter cells.

151 e equal division of genetic material between daughter cells.

152 of events by which a cell gives rise to two daughter cells.

153 and lowered SPCH abundance in one of the two daughter cells.

154 slation, and facilitate the transport to the daughter cells.

155 o physically cleave the mother cell into two daughter cells.

156 r the final abscission and separation of the daughter cells.

157 sentangling and delivery of DNA molecules to daughter cells.

158 dramatic shape change as it divides into two daughter cells.

159 ned to mitosis, it resulted in aneuploidy in daughter cells.

160 ides the chromosomes between the two nascent daughter cells.

161 catalyzes membrane cleavage to create equal daughter cells.

162 ucleus formation and increased aneuploidy in daughter cells.

163 how BASL differentially functions in the two daughter cells.

164 per separation of the cell contents into two daughter cells.

165 dividing cell wall to separate newly formed daughter cells.

166 r the generation of two viable and identical daughter cells.

167 es of gametes, zygotes, and apical and basal daughter cells.

168 y regulated partitioning of chromosomes into daughter cells.

169 eposition of a new cell wall between the two daughter cells.

170 lusively determines cell-cycle commitment in daughter cells.

171 nd equally segregate its genome into two new daughter cells.

172 cular vascular plexus, and then divided into daughter cells.

173 of cytoplasmic connections between dividing daughter cells.

174 aused premature formation of deposits in the daughter cells.

175 nd results in the physical separation of two daughter cells.

176 on have significant effects on the resulting daughter cells.

177 eostasis, and chromosome distribution within daughter cells.

178 ntrols septal PG synthesis and separation of daughter cells.

179 ayer of separating membranes between the two daughter cells.

180 triggers petite formation preferentially in daughter cells.

181 division and appropriate differentiation of daughter cells.

182 o create the new cell poles of the separated daughter cells.

183 an internal cue for establishing PCP in new daughter cells.

184 oviding a simple way to ensure equally sized daughter cells.

185 their chromosomes and partition them to two daughter cells.

186 ith symmetrical distribution of viral DNA to daughter cells.IMPORTANCE A mechanistic understanding of

187 Cep192 is the first recruited to the site of daughter centriole formation and regulates the centriola

188 We found that manipulations that prevent daughter centriole formation or induce its separation fr

189 A recent study has implicated the daughter centriole in centriole amplification in multici

190 Our data support a model in which the daughter centriole promotes ciliogenesis through Neurl-4

191 centriole in a process that required mother-daughter centriole proximity.

192 In contrast, the single daughter centriole remaining in the egg is eliminated be

193 ation may be caused by effects on Neurl-4, a daughter centriole-associated ubiquitin ligase cofactor,

194 Centriole numbers are tightly regulated, and daughter centrioles (which assemble in S phase) cannot t

195 Polo-docking site that helps recruit Polo to daughter centrioles and is required for the subsequent r

196 e acquisition of distal appendage markers on daughter centrioles and the loss of procentriolar marker

197 The formation of new daughter centrioles is guided by a pre-existing, mother

198 Paradoxically, mother and daughter centrioles overcome this distance in early mito

199 etic data suggest that SAS-7 is required for daughter centrioles to become competent for duplication,

200 A protein called SAS-7 is required for daughter centrioles to become mothers in C. elegans.

201 The proximity between mother and daughter centrioles was proposed to restrict new centrio

202 store ciliogenesis in cells with manipulated daughter centrioles.

203 Nin localizes asymmetrically to the younger (daughter) centrosome, yet it is not required for the asy

204 e and the relative positioning of mother and daughter centrosomes.

205 stereotypical inheritance of the mother and daughter centrosomes.

206 ur data demonstrate that the roles of ESP in daughter chromatid separation and cell expansion are con

207 Incorporating each set of daughter chromosomes into a single nucleus at the end of

208 s organized within the nucleoid, and how two daughter chromosomes segregate has yet to emerge.

209 killer whales and show that when mothers and daughters co-breed, the mortality hazard of calves from

210 ant breeders a heritable mechanism for their daughters' competitive edge.

211 other, destructive QI is alleviated and the daughter conductance is high.

212 tion relative to both acetylene linkers, the daughter conductance remains as low as the parent.

213 samples produced (234)U and both mother and daughter could be identified unequivocally using HR-ICP-

214 crypt fission, in which 2 crypts fuse into 1 daughter crypt.

215 t, crypts multiply via fission, generating 2 daughter crypts from 1 parental crypt.

216 ion, the division of a single crypt into two daughter crypts.

217 g-term outcomes of cancer-bereaved sons' and daughters' distrust in the care that was provided to a d

218 In order to produce rejuvenated daughters, dividing budding yeast cells confine aging fa

219 '-exonuclease activity on the lagging strand daughter DNA, but its DNA binding activity mediated load

220 Using this platform, the volume of one daughter droplet could be reduced up to 84% compared to

221 plet volume), and droplet splitting (1:1-1:5 daughter droplet ratio).

222 using a Y-shaped junction, resulting in two daughter droplets, one of which containing all or most o

223 n mother-daughter pairs, social positions of daughters during the disrupted period were predicted by

224 s and tended to be slightly higher in mother-daughter dyads than in mother-son dyads.

225 sed capacity for terminal differentiation of daughter enteroblasts (EBs).

226 Both daughters exhibited worse vision than their mother, desp

227 hat perturbed polarized divisions and skewed daughter fates.

228 s lie on the plane of the substrate, whereas daughter filaments have random deviations out of this pl

229 e-focus progenitor is locally excursive, the daughter foci undergo directed segregation.

230 nt accumulation of p21 during mother G2- and daughter G1-phases.

231 o acids are available for multiple rounds of daughter generation.

232 quent amino acid substitutions in duplicated daughter genes selectively restricted protein conformati

233 ollowing an asymmetrical division, the small daughter grew at a faster exponential rate than the larg

234 Both sons and daughters had a significantly lower percentage of energy

235 l grandmother smoking in pregnancy and grand daughters having adverse scores in Social Communication

236 spatial organization of the actin mother and daughter (i.e., branch) filaments within this network.

237 Distinct daughter identities can result from the physical polarit

238 al (peripheral to central) axis, leaving one daughter in the peripheral RSC niche and the other more

239 ale leopards also cared for sons longer than daughters, in line with the sex-allocation hypothesis.

240 Resident grandmothers also decreased their daughters' inter-birth intervals by one year.

241 into an effector-like T cell and the distal daughter is more likely to differentiate into a memory-l

242 elements involved, either as "parent" or as "daughter" isotopes, in six radiogenic isotope systems us

243 When the plasmid replicates, the daughters largely display motilities similar to that of

244 of 2.97-5.79 Ma and 2.40-5.39 Ma in the two daughter lineages.

245 believed to result in divisions in which one daughter loses contact with the basal lamina.

246 nal peri-conceptional body weight may affect daughters' mammary development and breast cancer risk an

247 es, in which they grow and divide to release daughter merozoites, which in turn invade new erythrocyt

248 to distribute newly replicated nucleoids to daughter mitochondria.

249 cle spawns a slightly tilted, consequential 'daughter' nanoparticle, which by amplification over vari

250 NSCs overexpressing BDNF generated increased daughter neuronal cell numbers post-differentiation, wit

251 tive mother cells and the behaviors of their daughter nuclei after mitosis.

252 s ensure accurate mitotic NPC segregation to daughter nuclei by linking mitotic DNA and NPC segregati

253 (225)Ac, assuming instant decay of unstable daughter nuclides.

254 In some studies, the daughters of BPA-exposed dams have shorter AGD than cont

255 upregulates PI3K/Tor signaling in the early daughters of CySCs.

256 Daughters of overweight fathers had higher rates of carc

257 ously, with cells of one phenotype producing daughters of the opposite within four cell doublings.

258 data from a longitudinal study of 421 mother-daughter pairs enrolled in an integrated health services

259 For analyses using known mother-daughter pairs, social positions of daughters during the

260 um falciparum occurs via schizogony, wherein daughter parasites are formed by a specialized cytokines

261 develop normally, the structural scaffold of daughter parasites, the inner membrane complex (IMC), fa

262 dependently related to a higher adult BMI in daughters, particularly for the highest 90th quantile of

263 The ingrowth of the (230)Th daughter product in the material was followed by measuri

264 ollowed by measuring the accumulated (230)Th daughter product relative to its parent (234)U nuclide u

265 Consideration of reactions of (*)ClO and its daughter products (e.g., ClO2(-)), not included in previ

266 h production and simultaneous degradation of daughter products, especially benzene.

267 d the identification of corresponding stable daughter products, which are likely to differ significan

268 itosis, spindle alignment, and the choice of daughter progenitors to differentiate or remain stem-lik

269 chloramine scavenging by the (*)Cl and (*)OH daughter radicals.

270 ived progenitors, generating differentiating daughters rather than SCs.

271 By contrast, basal daughters remain WNT(hi).

272 trosome-dependent processes, the role of the daughter remains poorly understood.

273 As such, daughters replicated the social network roles of their m

274 henotypes as observed in an XY father and XY daughter, respectively.

275 igated the impact of paternal body weight on daughters' risk of this disease.

276 /height (m)(2)) >/=30) was associated with a daughter's earlier transition to breast and pubic hair s

277 d their inclusive fitness by enhancing their daughter's reproductive rate and success irrespective of

278 obesity and maternal GDM with timing of the daughter's transition to pubertal maturation stage 2 or

279 PLC resolution of lithiated TAGs followed by daughter scan MS/MS of positive ions revealed several in

280 Accumulation of Cdc5 at the daughter SPB in late anaphase is controlled by Bfa1.

281 depleted cells also fail to elongate nascent daughter strand DNA following UV irradiation and show re

282 ible with MMR and protects the discontinuous daughter strand from unnecessary degradation by MMR mach

283 ssive dilution through DNA synthesis without daughter strand methylation and active enzymatic process

284 ng CpG methylation signatures from parent to daughter strands, producing heritable methylation patter

285 uration of Okazaki fragments into continuous daughter strands.

286 associated with significantly shorter AGD in daughters, suggesting that BPA may alter the hormonal en

287 The other daughter T cell has relatively low mTORC1 activity and i

288 This results in the generation of two daughter T cells, one of which shows increased mTORC1 ac

289 condary parietal cells then predominantly to daughter tapetal cells.

290 pH-sensitive triplex DNA bonds enable parent-daughter templating, while in the second species, triple

291 daughter that differentiates and a posterior daughter that continues to divide.

292 symmetric divisions give rise to an anterior daughter that differentiates and a posterior daughter th

293 and deterministically more damage to the old daughter, the one receiving the mother's old pole.

294 em cells and frequently-dividing short-lived daughter transit cells.

295 e in different ways to produce heterogeneous daughter types at the right time and in proper numbers t

296 After birth, daughters underwent exams that included two measures of

297 r between sisters, those between mothers and daughters were unrelated.

298 1 leads to fewer mitotic divisions and large daughters, while mis-expression of CDKG1 causes supernum

299 velopment, cell division often generates two daughters with different developmental fates.

300 er cells and the correct localization of the daughters within growing tissue.