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1 ension and survival in PC-12 cells (up to 25 days in vitro).
2 neurones obtained from older cultures (14-27 days in vitro).
3 on was significantly suppressed for up to 21 days in vitro.
4  colonies generated new neurons as late as 7 days in vitro.
5 e and the experiments were carried out at 21 days in vitro.
6 d highly viable and could be cultured for 10 days in vitro.
7  patch-clamp recordings were made after 7-28 days in vitro.
8 thin minutes and is sustained for at least 3 days in vitro.
9 sed to 3.5 mM glutamate for 35 minutes at 21 days in vitro.
10 bled, CheA remains associated and active for days in vitro.
11 with 0-50 ng/mL rhVEGF(165) or rhFGF-2 for 7 days in vitro.
12 lls when mixed with irradiated T cells for 6 days in vitro.
13 eath between culture media over the first 14 days in vitro.
14 HUVECs) and enhancing tube stability up to 6 days in vitro.
15  undergo apoptosis beginning approximately 4 days in vitro.
16 180), 0.1 microg/ml to 10.0 microg/ml, for 8 days in vitro.
17 e, and these differences were enhanced by 10 days in vitro.
18 priate layer-specific projections during 5-7 days in vitro.
19  fibers had degenerated during the first few days in vitro.
20 oped layer-specific axonal arbors during 5-7 days in vitro.
21 robustly rhythmic and persisted for up to 32 days in vitro.
22  10-day-old rats and maintained for 10 to 12 days in vitro.
23 sed substantially when cells were cultured 3 days in vitro.
24  (0N3R), even at extended time points of 100 days in vitro.
25 ed strongly stained presynaptic boutons by 3 days in vitro.
26 en GH3 and GHRH-CL1 cells were treated for 7 days in vitro.
27 tch clamp recordings were obtained after 6-8 days in vitro.
28 urons were synaptically coupled after 3 or 4 days in vitro.
29 creased during the next few days to 39% at 4 days in vitro.
30 t begun also developed taste buds after 9-12 days in vitro.
31  the bladder and ileocecum and persisted for days in vitro.
32 nt failed to inhibit BrdU incorporation at 9 days in vitro.
33 ransmission in cultured hippocampal neurons (days-in-vitro 10-14) prepared from fetal rats.
34 l cultures containing both neurons and glia (days in vitro 13-15) were exposed to periods of oxygen-g
35 ) rat cortical neurons were cultured to DIV (days in vitro) 14.
36 ntly (-55.7+/-6.2%) in slices cultured for 2 days in vitro (2DIV) as compared to slices placed in cul
37 xpressed in axons and growth cones of young (days in vitro 3-6) hippocampal rat neurons.
38 ainst induced apoptosis and mature cells (5+ days in vitro) against glutamate toxicity, but its preci
39 tects immature cerebellar granule cells (1-3 days in vitro) against induced apoptosis and mature cell
40 tes a Th0 population that is evident after 3 days in vitro and becomes prevalent after successive enc
41  blood monocytes were differentiated for 1-7 days in vitro and examined with respect to 1) uptake of
42  cultures from brains of 2-day-old rats at 7 days in vitro and plating them (0 days post-shake, DPS).
43 ed marrow of the long bones were cultured 14 days in vitro and subsequent colonies were assessed by l
44 120 Cameroonian newborns were cultured for 7 days in vitro and supernatants were assessed by ELISA fo
45 ith either BMP4 or with various FGFs for two days in vitro and then grown under the kidney capsule of
46 tions increased markedly during the first 14 days in vitro and were inversely related to response lat
47 lation was constant at 64.2% for at least 30 days in vitro, and contractile activity was observed for
48 t after 100 days or more, stored again for 4 days in vitro, and retransplanted into 5 other diabetic
49 tnatal day 8 mice, cultured for 2-4 or 15-30 days in vitro, and studied by electron and fluorescence
50 vector and then exposed to 5-FC either for 4 days in vitro before transplantation or for 14 days imme
51 SBDPs was detected in cultures of at least 7 days in vitro but not in younger cultures.
52  in all cells taken from young cultures (4-7 days in vitro) but the latter was absent in 55% of the n
53                                      After 3 days in vitro, cellular aggregation suggesting VMH forma
54  lengths and fewer branches between 7 and 21 days in vitro compared with wild-type (WT) littermates.
55  medium from PS1 null neuronal cultures at 8 days in vitro contained higher levels of glutamate than
56  GAD67-labeled somata, whereas those grown 4 days in vitro contained primarily terminal-like varicosi
57                         Cells grown 11 to 15 days in vitro demonstrated GABA(A)R delta subunit expres
58                                     As of 18 days in vitro (DIV 18), ESNs were synaptically coupled,
59                                           At days in vitro (DIV) 7-8 PSD-95gfp-transfected cells had
60                  In most cells (96 %) at 6-7 days in vitro (DIV) and in a small proportion of cells (
61 th time in all ages during the initial three days in vitro (DIV) but the proportion of Schwann cells
62 d a lower potency for GABA between 11 and 12 days in vitro (DIV) resulting in a shift of the EC50 fro
63            Electrophysiological data over 95 days in vitro (DIV) showed an age-dependent increase of
64  100 microM glutamate/10 microM glycine at 5 days in vitro (DIV), but became vulnerable to the same s
65                                    After 3-4 days in vitro (DIV), cells that are plated into the soma
66                             In cells of > 11 days in vitro (DIV), exposure to 50 mM potassium or 100
67                  In young cultures after 1-7 days in vitro (DIV), GABA induced depolarizing membrane
68  mouse cortical neurons were cultured 8 or 9 days in vitro (DIV), loaded with the Ca(2+) indicator Fl
69                                      After 3 days in vitro (DIV), the average vesicle velocity was ab
70  GABAergic synapses appeared between 3 and 8 days in vitro (DIV), with GABAA receptor subunit cluster
71 re evaluated via immunocytochemistry over 21 days in vitro (DIV).
72 nerability to excitotoxicity with increasing days in vitro (DIV).
73 amic acid decarboxylase (GAD 65/67), over 17 days in vitro (DIV).
74 ultured cerebellar granule cells (CGCs) 6-14 days in vitro (DIV).
75 atory or inhibitory markers at 7, 14, and 21 days in vitro (DIV).
76 om the parent cell bodies, was observed at 2 days in vitro (DIV).
77 ls, surface galC expression was lost after 3 days in vitro (DIV).
78  from studies on immature cultures [< or = 6 days in vitro (DIV)] and cytoarchitectural analyses of m
79  rat hippocampal neurons increases over age [days in vitro (DIV)] in long-term primary cultures, appa
80          In cultured hippocampal neurons 7-8 days in vitro (DIV7-8), NMDAR stimulation leads to a con
81 d cultured hippocampal neurons with TTX at 7 days in vitro, during rapid synaptogenesis, and at 14 da
82                                  Over only 8 days in vitro, electrical field stimulation induced cell
83 reated chronically with 30 mM (140 mg%; 3-11 days in vitro) ethanol to study the actions of prolonged
84    Networks obtained from young cultures (14 days in vitro) exhibited a random topology, which evolve
85 tic neurons as well as mature neurons (15-82 days in vitro) for gene functional analysis.
86 te presynaptic release occurred after 3 or 4 days in vitro in high-density cultures but was absent in
87 died the molecular mechanisms activated at 2 days in vitro in these conditions.
88 n cortical neurons grown for 5, 8, 11, or 14 days in vitro indicate that sequences between -1253 and
89  24-h exposure to NMDA (100 mum) or K25 at 2 days in vitro induced long term survival of CGNs in K5 c
90 ltured rat cortical neurons from day 4 (four days in vitro or DIV 4) to day 20 (DIV 20), we observed
91 ted cultures compared to naive-cultures at 2 days in vitro (p<0.001).
92 -1 (KLS) and CD150CD48-KLS cells for up to 5 days in vitro prevented the culture-induced loss of Inte
93                                    After 1-7 days in vitro, punctate alpha(1C) staining occurred alon
94 nduced whole cell responses in mature (22-29 days in vitro) rat cortical neurons were potentiated nea
95                        Cultures maintained 7 days in vitro retained tight organization of neuronal la
96                                      After 5 days in vitro, the number of SMNs in treated cultures wa
97 hen isolated E14-E16 slices were grown for 4 days in vitro, the width of the GAD65-labeled ventral ma
98  loading, and sustained drug release over 15 days in vitro under sink conditions.
99 ium in organ culture is compromised after 14 days in vitro using an immersion system of tissue cultur
100 alysis revealed that the period from 6 to 12 days in vitro was the critical time for exposure to GDNF
101      Further, at extended time points of 365 days in vitro, we observe a switch in tau splicing to in
102  postnatal day 0 or 1 and examined after 3-4 days in vitro, we observed differences in the amplitude
103 specific antibodies (OS-2 and JH455) after 5 days in vitro, which corresponds to a time course simila

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