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1 postcoitum, rising progressively to term (19 days postcoitum).
2 l the 6-somite-pair stage (approximately 8.5 days postcoitum).
3 late neural plate stage (approximately 7.75 days postcoitum).
4 es in amniotic fluid between E14.5 and E16.5 days postcoitum.
5 ly becomes critical in the chorion after 7.5 days postcoitum.
6 a window of sensitivity between 8.5 and 10.5 days postcoitum.
7 GCNF(lox/lox) mice die at 9.5-10.5 days postcoitum.
8 because of fetal death between 8.5 and 15.5 days postcoitum.
9 al delay and embryonic lethality at about 10 days postcoitum.
10 ays postcoitum followed by resorption by 9.5 days postcoitum.
11 bkap(-)(/)(-)) embryos surviving beyond 12.5 days postcoitum.
12 murine thymus was observed as early as 13.5 days postcoitum.
14 doglin mutation fail to progress beyond 10.5 days postcoitum and fail to form mature blood vessels in
15 y throughout the embryonic region at 5.5-6.5 days postcoitum and later in the node, midbrain, spinal
16 ortex of developing mouse fetuses (15.5-17.5 days postcoitum) and their nuclei were transferred into
17 had formed in the fetus (approximately 8.25 days postcoitum), and vascular continuity with the yolk
18 Ini1-null embryos die between 3.5 and 5.5 days postcoitum, and Ini1-null blastocysts fail to hatch
19 otes, placental defects are manifest at 10.5 days postcoitum as nearly complete loss of the labyrinth
21 development until midgestation (12.5 to 13.5 days postcoitum), at which time they undergo a dramatic
22 nt in heart tubes from Ncx1(-/-) mice at 9.5 days postcoitum but is activated in heart tubes from Ncx
23 Failure of cranial closure between 9 and 9.5 days postcoitum coincided with increased apoptosis in th
24 Examination of embryos between 8 and 10.5 days postcoitum confirmed that lethality was due to a fa
25 detected in transgenic mice as early as 13.5 days postcoitum, consistent with a defect of preplate de
26 embryos developed beyond 7.5 and up to 10.5 days postcoitum, demonstrating a requirement for SMAD2 i
28 lian inheritance was followed for up to 18.5 days postcoitum (dpc) and that approximately 90% of GYS1
29 e genomes of gonadal germ cells at 11.5-19.5 days postcoitum (dpc) are incompetent to support full-te
30 by in situ RNA hybridization as early as 10 days postcoitum (dpc) in developing gut, as early as 14.
31 first detected by immunofluorescence at 13.5 days postcoitum (dpc) in the mesenchyme surrounding the
32 the gonad begins to form shortly before 10.5 days postcoitum (dpc) on the ventromedial side of the me
35 heir development is retarded globally by 7.5 days postcoitum (dpc), and all the null embryos die befo
36 embryonic developmental process stops at 8.5 days postcoitum (dpc), and excessive cell death occurs a
37 tical in XX and XY genital ridges until 11.5 days postcoitum (dpc), by 12.5 dpc the XY gonad develops
39 GCNF(-/-) embryos cannot survive beyond 10.5 days postcoitum (dpc), probably due to cardiovascular fa
48 early to middle gestation (approximately 9.5 days postcoitum [dpc]) and exhibited a number of novel p
49 eadfold-stage allantoises (approximately 8.0 days postcoitum; dpc) were subdivided into three proximo
50 Smad1 mutant mice die at approximately 9.5 days postcoitum due to defects in allantois formation.
51 showing that inhibiting p38 activity in 5.5 days postcoitum embryo cultures leads to a switch from A
52 the electrical activation pattern of the 9.5-days postcoitum embryonic mouse heart and show that trea
53 lular bridges in the ovaries of 11.5 to 17.5 days postcoitum embryos; microtubules and organelles hav
55 lly with apparent developmental delay at 7.5 days postcoitum followed by resorption by 9.5 days postc
56 -/- embryos die in utero between 4.0 and 4.5 days postcoitum, following the depletion of their CAN fr
58 eries of high percentage null chimeras (8-10 days postcoitum) in which Gata4+/+ cells were restricted
59 injection of TM into a pregnant mouse at 8.5 days postcoitum leads to detectable recombination in the
60 is vital for sustaining pregnancy beyond 7.5 days postcoitum, likely by regulating the balance of coa
62 first detected in amniotic fluid (AF) at 17 days postcoitum, rising progressively to term (19 days p
63 tu hybridization on mouse inner ears (from 8 days postcoitum to postnatal day 5) to establish the exp
64 enesis detect Plac1 expression from 7.5 dpc (days postcoitum) to 14.5 dpc in ectoplacental cone, gian
65 orter expression is detected in 9.5 and 10.5 days postcoitum transgenic embryos in a manner consisten
66 matic cells of GCNF(lox/lox) embryos at 8.25 days postcoitum was not silenced as in the GCNF(+/+) emb
67 Forty-four percent of live embryos at 10.5 days postcoitum were morphologically normal when prematu
68 die after implantation at approximately 6.5 days postcoitum with a loss of epiblast cells, expansion
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