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1 time sleepiness) to 24 points (high level of daytime sleepiness).
2 nge from 0-24, with values <10 suggesting no daytime sleepiness).
3 ry disease (CAD), many of whom do not report daytime sleepiness.
4 and covariation of obesity with snoring and daytime sleepiness.
5 s and one important outcome, the severity of daytime sleepiness.
6 eep are a well recognized cause of excessive daytime sleepiness.
7 arding the relationship between mild OSA and daytime sleepiness.
8 quantity, poor sleep quality, and excessive daytime sleepiness.
9 lt in transient but substantial increases in daytime sleepiness.
10 nsomnia, delayed sleep habits, and excessive daytime sleepiness.
11 a sleep study for patients with unexplained daytime sleepiness.
12 ) display significant sleep disturbances and daytime sleepiness.
13 p apnea is a common disease, responsible for daytime sleepiness.
14 a nearly significant decrease in subjective daytime sleepiness.
15 ude hypothalamic injury, with inactivity and daytime sleepiness.
16 out (n=144) complaints of frequent excessive daytime sleepiness.
17 ict important consequences such as excessive daytime sleepiness.
18 ould prove useful in prediction of excessive daytime sleepiness.
19 may reduce sleep fragmentation and excessive daytime sleepiness.
20 idence interval): 2.97 (2.65-3.34)), regular daytime sleepiness (2.66 (2.34-3.01)), and regular insom
21 n 0.53 and 1.33; P < 0.001 for all domains), daytime sleepiness (-2.92; P < 0.001), mood state (-4.24
22 owed high risk for OSAS, 46.3% had excessive daytime sleepiness, 41.5% were positive for both the Bq
24 eads to annoyance, disturbs sleep and causes daytime sleepiness, affects patient outcomes and staff p
25 urologic disorder characterized by excessive daytime sleepiness and abnormal manifestations of REM sl
26 t (REM) sleep, is characterized by excessive daytime sleepiness and cataplexy, a loss of muscle tone
30 were significantly more likely to experience daytime sleepiness and dozing during daytime activities.
33 )) and between increased levels of excessive daytime sleepiness and increased measures for adiposity
36 th Sleepiness Scale (ESS) was used to assess daytime sleepiness and standardized questionnaires asses
37 dy aimed to explore the relationship between daytime sleepiness and the risk of ischemic stroke and v
39 ance syndrome (UARS) is defined by excessive daytime sleepiness and tiredness, and is associated with
40 he basis of characteristic history (snoring, daytime sleepiness) and physical examination (increased
41 variance in obesity, 40% of the variance in daytime sleepiness, and 23% of the variability in self-r
42 e evaluated sleep characteristics, excessive daytime sleepiness, and chronotype using the Pittsburgh
43 with self-reported parental sleep duration, daytime sleepiness, and dozing among employed adults.
44 leep restriction, irregular sleep schedules, daytime sleepiness, and elevated risk for sleep disturba
45 reasoning, as well as insomnia, depression, daytime sleepiness, and headaches), 2 ("confusion-ataxia
47 ale score, the visual analog scale score for daytime sleepiness, and sleep log-derived and actigraphy
48 ring sleep are not associated with excessive daytime sleepiness, and therefore appear unlikely to con
50 of the clinical features (i.e., significant daytime sleepiness, anxiety and depression symptoms, pot
51 and fatigue (aOR, 1.59; 95% CI, 1.16-2.19), daytime sleepiness (aOR, 1.81; 95% CI, 1.28-2.55), or in
52 r data show that: (1) poor sleep quality and daytime sleepiness are problems common to all types of I
54 ervous system disorders, including excessive daytime sleepiness, attention deficit hyperactivity diso
57 a sleep disorder characterized by excessive daytime sleepiness, cataplexy, and other pathological ma
59 ological disorder characterized by excessive daytime sleepiness, cataplexy, hypnagonic hallucinations
61 state, anxiety and depressive symptoms, and daytime sleepiness compared with conservative treatment.
64 ea/hypopnea syndrome can experience residual daytime sleepiness despite regular use of nasal continuo
66 polysomnography with complaints of excessive daytime sleepiness (EDS) and clinically suspected obstru
67 ted by exploring the links between excessive daytime sleepiness (EDS) and vulnerability to infectious
68 the treatment of disorders such as excessive daytime sleepiness (EDS) as well as other sleep or cogni
72 nd snoring and between obesity and excessive daytime sleepiness (EDS), although for the most part the
73 The percentage of patients with normalized daytime sleepiness (Epworth score < 10) was significantl
75 daytime sleepiness, patients with excessive daytime sleepiness (Epworth Sleepiness Scale score >/=10
77 d OSA (apnea-hypopnea index >/=15/h) without daytime sleepiness (Epworth Sleepiness Scale score <10)
78 quality (Pittsburgh Sleep Quality Index) and daytime sleepiness (Epworth Sleepiness Scale), and circa
79 ere habitual snorers, 18% reported excessive daytime sleepiness (ESS > or = 11), and 29% were obese (
80 der narcolepsy is characterized by excessive daytime sleepiness, fragmentation of nighttime sleep, an
85 a that self-reported symptoms of snoring and daytime sleepiness in older men have a genetic basis tha
86 ssessing multiple risk factors for excessive daytime sleepiness in older subjects (mean age, 78 years
87 ilation produces an improvement in excessive daytime sleepiness in patients with Cheyne-Stokes breath
88 nct treatment for the management of residual daytime sleepiness in patients with obstructive sleep ap
89 gmentation (SF) appear to underlie excessive daytime sleepiness in patients with sleep apnea (OSA).
93 ommon medical disorder that causes excessive daytime sleepiness, increasing the risk for drowsy drivi
94 ma associated with atopy, fatigue, excessive daytime sleepiness, insomnia, and only 0 to 3 nights of
95 re seen by neurologists, including excessive daytime sleepiness, insomnia, narcolepsy, rapid eye move
100 [SE] difference, -0.42 [0.09], P < .001) and daytime sleepiness (mean [SE] difference, -0.24 [0.09],
101 Secondary endpoints included changes in daytime sleepiness, mood state, anxiety, and depression.
102 ypertension, cardiovascular disease, stroke, daytime sleepiness, motor vehicle accidents, and diminis
103 BI in females and WDR27 in males), excessive daytime sleepiness (near AR-OPHN1) and a composite sleep
104 11-2.10) (P = .009, I2 = 74%), and excessive daytime sleepiness (OR, 2.27; 95% CI, 1.54-3.35) (P < .0
105 ted OR, 2.65; P=0.023), those with excessive daytime sleepiness (OR, 2.51; P=0.037), and those with >
106 14-1.88) (P = .003, I2 = 76%), and excessive daytime sleepiness (OR, 2.72; 95% CI, 1.32-5.61) (P = .0
107 es such that eczema associated with fatigue, daytime sleepiness, or insomnia was associated with even
108 ilability and fatigue, depression, excessive daytime sleepiness, or rapid eye movement sleep behaviou
111 igraphy measured duration and fragmentation, daytime sleepiness, overall quality, self-reported durat
112 Compared with PD patients without excessive daytime sleepiness, patients with excessive daytime slee
113 quantity, poor sleep quality, and excessive daytime sleepiness, studied according to an a priori pro
114 onclude that within a group of patients with daytime sleepiness, suspected OSA, and a normal RDI, the
118 s Scale (ESS) score, which ranges from 0 (no daytime sleepiness) to 24 points (high level of daytime
119 phalopathy might be unable, due to excessive daytime sleepiness, to accumulate the need/ability to pr
124 abnormal rapid eye movement (REM) sleep and daytime sleepiness, was examined using the canine model.
125 treatment) and OSA concomitant with habitual daytime sleepiness were estimated using repeated-measure
126 This condition causes recurrent insomnia and daytime sleepiness when the rhythms drift out of phase w
128 nea or >/= 2 hallmarks of OSA: loud snoring, daytime sleepiness, witnessed apnea, and hypertension.
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