ライフサイエンスコーパス: db-cAMP

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1 O: 90+/-15% versus 77+/-19% versus 24+/-12%; db-cAMP: 235%>172%>90%).

2 When a db-cAMP stimulus was applied to cells which had been pre

3 levels were not altered significantly after db-cAMP treatment.

4 Dibutyryl cyclic AMP (db-cAMP) (10(-3) M) was used to stimulate PKA and the ca

5 Both ISO and db-cAMP increased I(Ca,L) in NF- and LVAD- significantly

6 After ISO and db-cAMP, the I(Ca,L) activation was not significantly di

7 ich regulates intrinsic CBF, is inhibited by db-cAMP but not vice versa.

8 Addition of a forskolin-dibutyryl cAMP (db-cAMP) cocktail to the perfusate caused no significant

9 l neurons when perfused with dibutyryl cAMP (db-cAMP) or forskolin.

10 hours with the cAMP analogue dibutyryl cAMP (db-cAMP, 1 mmol/L).

11 evation of cAMP, either with dibutyryl-cAMP (db-cAMP) or by priming with a variety of neurotrophins,

12 ence of isoproterenol (ISO), dibutyryl-cAMP (db-cAMP), Bay K 8644 (BayK), Okadaic acid (OA, a phospha

13 ion of cAMP using forskolin, dibutyryl-cAMP (db-cAMP), BAY60-6583 or Cicaprost induced rapid cytoskel

14 timulated with protein kinase A or forskolin-db-cAMP using whole-cell patch clamping.

15 However, db-cAMP caused no change in the maximum rate of rise of

16 podia or lamellipodia were observed, even in db-cAMP; and 5) during active growth, axon tips containe

17 dunce neurons or wild-type neurons grown in db-cAMP) results in an increase in the frequency of chol

18 e in the number and size of gap junctions in db-cAMP-treated cells.

19 crease after 4 hours of exposure to 1 mmol/L db-cAMP; cycloheximide did not block this effect.

20 utyryl adenosine 3',5'-cyclic monophosphate (db-cAMP), carbachol, and the heat-stable toxin of Escher

21 utyryl adenosine-3',5'-cyclic monophosphate (db-cAMP, membrane-permeable form of cAMP), the bell-shap

22 In contrast to NL, db-cAMP failed to alter either HCO(3)(-) or Isc in CF ti

23 sal cAMP levels contribute to the effects of db-cAMP by pushing the combined levels of cAMP above a t

24 Alone, injection of db-cAMP into the DRG mimics completely a conditioning le

25 ore, combining rolipram with an injection of db-cAMP near the graft not only prevents the drop in cAM

26 Importantly, DRG injection of db-cAMP results in extensive regeneration of dorsal colu

27 ed when neurons were exposed to forskolin or db-cAMP, suggesting an involvement of a cAMP signaling p

28 monophosphate, N6,O2-dibutyryl, sodium salt (db-cAMP) resulted in reduction of INa to 62.8 +/- 5.5% w

29 EPSC frequency induced by repetitive, spaced db-cAMP exposure in wild-type neurons is absent in neuro

30 x45 protein synthesis in myocytes exposed to db-cAMP ( > 2-fold after a 4-hour exposure) but no chang

31 Furthermore, a brief exposure to db-cAMP induces two distinct changes in transmission at

32 ence of perfusions, cells pre-incubated with db-cAMP showed no further rise in response to stimulatio

33 rowth cones became active when perfused with db-cAMP.

34 Perfusion with db-cAMP (10(-3) M) alone showed an early rise in CBF (15

35 3) M), before the sequential perfusions with db-cAMP and 4-Br-A23187.

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