ライフサイエンスコーパス: dbcAMP

1 dbcAMP also induced mechanosensitivity in the majority o

2 dbcAMP-induced sensitization was blocked by the PKA inhi

3 e 13-acetate (PMA) and dibutyryl cyclic AMP (dbcAMP) increased expression of the reporter in HEL cell

4 le cyclic AMP analogue dibutyryl cyclic AMP (dbcAMP) resulted in a strong upregulation of RhoE at 24

5 state acetate (PMA) or dibutyryl cyclic AMP (dbcAMP) to activate either protein kinase C (PKC) or pro

6 n and 143.4 microM for dibutyril cyclic AMP (dbcAMP).

7 ases, and by membrane-permeable cAMP analog (dbcAMP), exhibit a biphasic concentration dependency.

8 bation of mesangial cells with TNF-alpha and dbcAMP completely inhibited TNF-alpha-induced MCP-1 gene

9 cAMP-inducing agents, such as forskolin and dbcAMP.

10 -1 by activation of PKA but that neurons and dbcAMP regulate GLT-1 protein through convergent pathway

11 te cyclase, mimicked the effects of PGE2 and dbcAMP on TTX-R INa.

12 The effects of PMA and dbcAMP on promoter expression correlated with mRNA expre

13 serglycin mRNA and promoter constructs, but dbcAMP increased expression in HL-60 cells.

14 ocytes (CEO) maintained in meiotic arrest by dbcAMP or hypoxanthine, GVB was dose-dependently induced

15 ng of transient fusion pores was elevated by dbcAMP.

16 Finally, we found that RhoE upregulation by dbcAMP was significantly reduced under hypoxic condition

17 ication of the cAMP analogue dibutyryl cAMP (dbcAMP) at a concentration of 1 mM produced effects on b

18 vercome by administration of dibutyryl cAMP (dbcAMP) or a Rho kinase inhibitor in vitro and in vivo.

19 ained in meiotic arrest with dibutyryl cAMP (dbcAMP) were stimulated to undergo germinal vesicle brea

20 tor of adenylate cyclase and dibutyryl cAMP (dbcAMP), a membrane permeable analogue of cAMP suppresse

21 f the nonhydrolyzable analog dibutyryl cAMP (dbcAMP), can block the inhibitory effects of MAG and mye

22 differentiation, induced by dibutyryl cAMP (dbcAMP), on Ca2+ influx triggered by Ca2+ store depletio

23 se A, before the addition of dibutyryl cAMP (dbcAMP, 10(-3) M) for 30 minutes.

24 in medium supplemented with dibutyryl-cAMP (dbcAMP) caused a dramatic change in cell morphology, inc

25 Conversely, dbcAMP down-regulates OPG expression and up-regulates CS

26 in-dependent kinase 5 (Cdk5) is required for dbcAMP and putrescine to overcome MAG-mediated inhibitio

27 phorbol 12-myristate 13-acetate), forskolin, dbcAMP (N6, 2'-O-dibutyryladenosine 3':5'-cyclic monopho

28 Importantly, dbcAMP and putrescine increase expression of p35, the ne

29 minal vesicle (GV)-stage oocytes cultured in dbcAMP-containing medium plus AICAR possessed elevated l

30 erence resulted in a significant decrease in dbcAMP-induced fusion.

31 osine deaminase inhibitor, stimulated GVB in dbcAMP-arrested CEO, suggesting AMPK activation due to A

32 Meiotic induction by AICA riboside in dbcAMP-supplemented medium was initiated within 3 h in D

33 substances (pertussis toxin, isoproterenol, dbcAMP) did not induce mesangial cell MCP-1 mRNA transcr

34 oncentrations (50 mum forskolin and 10-15 mm dbcAMP).

35 centrations (2-10 mum forskolin and 2.5-5 mm dbcAMP) these agents stimulate prolactin release, an inh

36 utyryl adenosine 3',5'-cyclic monophosphate (dbcAMP, 100 microM), a stable membrane-permeant cAMP ana

37 The ability of dbcAMP and putrescine to overcome inhibition by MAG is a

38 inant negative Cdk5 abolishes the ability of dbcAMP or putrescine to enhance neurite outgrowth in the

39 tagonists, but unlike GLT-1, the addition of dbcAMP to mixed cultures of neurons and astrocytes cause

40 ession of GLT-1 protein, but the addition of dbcAMP to mixed cultures of neurons and astrocytes did n

41 -298, on MAPK activity after the addition of dbcAMP was also determined.

42 The effect of dbcAMP on EGF receptor activity and basal and stimulated

43 The addition of IBMX or dbcAMP to indomethacin-treated, UVB-exposed cells restor

44 nd 12-O-tetradecanoylphorbol-13-acetate plus dbcAMP) and were enriched for genes related to neuronal

45 Prior exposure of the neurones to dbcAMP occluded the effect of a subsequent treatment wit

46 Treatment of NSC-19 cells with dbcAMP and forskolin, thus increasing intracellular cAMP

47 Incubation with dbcAMP did not have any effect either on the EGF recepto

48 In transplanted rats infused with dbcAMP, approximately 80 ES cell-derived motor axons wer

49 n DO when meiotic arrest was maintained with dbcAMP or hypoxanthine.

50 served in transplanted rats not treated with dbcAMP.