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1 uscular junction blockers (sensitive only to decamethonium).
2 bisquaternary compounds such as BW284C51 and decamethonium.
3 haracterized with respect to edrophonium and decamethonium.
5 lycaconitine, d-tubocurarine, hexamethonium, decamethonium, and mecamylamine either failed to up-regu
6 ive antagonism by dihydro-beta-erythroidine, decamethonium, and methyllycaconitine; noncompetitive an
8 ke potent OCT1 blockers (1,10-diaminodecane, decamethonium, bistriethylaminodecane, and 1,10-bisquinu
9 resence or absence of the cationic additive, decamethonium bromide were ineffective for resolving IGF
10 inhibitor complexes (EDR/AChE, THA/AChE, and decamethonium (DCM)/AChE) provided information regarding
11 onstants increased 3.3-12.0-fold; 200 microM decamethonium produced a 1.6-3.0-fold enhancement of rea
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