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1 rized by a fast (tau(1)) and a slow (tau(2)) decay time.
2 and prevents the shortening in NMDAR current decay time.
3 diction of brightness, stability, Phi(f), or decay time.
4 ctivity did not significantly alter the EPSC decay time.
5 cholinesterase inhibitor, prolonged the EPSC decay time.
6 cumulation but had no effect on the rise and decay time.
7 velength emission associated with the longer decay time.
8 using a long lifetime standard with a known decay time.
9 the deactivation kinetics by prolonging the decay time.
10 uCon A, resulting in increased intensity and decay time.
11 y, and the correlation between amplitude and decay time.
12 2+ transient amplitude and prolonged the 50% decay time.
13 (cis) isomer in DOPC (DPPC) presents a fast decay time.
14 smission to Bergmann glia and decreased EPSC decay time.
15 ise times, amplitudes, charge transfers, and decay times.
16 to CaN to maintain NMDAR currents with long decay times.
17 complexes led to variations in fluorescence decay times.
18 of the eIPSC could be distinguished by their decay times.
19 elations with relatively long characteristic decay times.
20 imilar to WT bumps, but with slightly slower decay times.
21 s of more than PhiPL = 90% at short emission decay times.
22 mations that fit the experimentally measured decay times.
23 current amplitudes with significantly faster decay times.
24 tes of LOV390 formation but exhibited adduct decay times 1 order of magnitude faster than wild type.
25 netically fast (rise time, 0.32 +/- 0.02 ms; decay time, 1.66 +/- 0.18 ms; mean +/- SD; n = 6 cells),
26 (31%), and prolongation of the Ca(2+) signal decay time (165%) than overexpression (2-fold) of wild t
27 imes greater), and decayed more slowly (half-decay time 189 ms for strontium and 32 ms for calcium).
29 tivity exhibit significantly longer AHP half-decay times (24.67 vs. 11.02 ms) and greater AHP amplitu
30 d trimer system, the fluorescence anisotropy decay time (35 fs) is found to be much shorter than that
32 pressure is typically much shorter than the decay time after cessation or decline in the volume of d
34 e different membrane phases via fluorescence decay time analysis, making this new probe versatile for
35 d in gas phase by measuring the fluorescence decay time and ion-neutral collision cross sections (CCS
36 Elevating quantal content lengthened EPSC decay time and prolonged both the fast (alpha7-nAChR-med
37 osolic Ca(2+) followed by an increase in the decay time and the spread of the spontaneous Ca(2+) spik
38 wo adjustable parameters instead of the many decay times and amplitudes required in standard analysis
41 ic modeling of temperature-dependent singlet decay times and quantum yields of fluorescence, isomeriz
43 peak parameters including shorter rise time, decay time, and half-width as compared to a bare carbon
45 peak amplitudes, prolonged current rise and decay times, and altered responses to benzodiazepine ago
48 (A) receptor, the peak amplitudes, 90-to-10% decay times, and total charge transfer of spontaneous mi
49 nged both GABA(A fast) and GABA(A slow) IPSC decay times approximately 2.5 fold, while having little
50 than 10-fold slower in nucleo-olivary cells (decay time, approximately 25 ms) than in large cells ( a
51 s (reductions of approximately 48% in 10-90% decay time, approximately 40% in tau, and approximately
53 ant glycine receptors, we show that response decay times are accelerated by addition of GABA, a weak
54 echnique in which fluorescence excited state decay times are measured as fluorescently labeled cells
56 We also rule out small increases in IPSC decay times (as caused by W170S and R414H) as a possible
57 es as photodetectors, for both intensity and decay-time based monitoring of the sensing element's PL.
58 ynapses with multiple release sites the EPSC decay time became faster when release probability was lo
63 2AP does not exhibit the long fluorescence decay time characteristic of the free nucleoside, sugges
66 The absorption spectrum and fluorescence decay time components of the complex at room temperature
69 postsynaptic currents (mIPSCs) (predominant decay time constant (tau(decay)), 1.0 ms) in addition to
70 inward current and prolonged the exponential decay time constant (tau) of Ca(2+)-activated Cl- 'tail'
71 ntly with distance from the soma whereas the decay time constant (taudecay) of Delta[Ca2+] decreases
73 +/- 0.59 to -4.15 +/- 0.73 nA with the fast decay time constant accelerating from 0.75 +/- 0.09 ms a
75 ial; (2) that the ratio between the synaptic decay time constant and the oscillation period be suffic
76 l (time to peak approximately 0.3-0.4 ms and decay time constant approximately 3-6 ms) served as the
79 tential was approximately 6 min, whereas the decay time constant for STP of the NMDA receptor-mediate
80 e recovery from desensitization is slow (the decay time constant is roughly 500 milliseconds), little
81 ronal firings are asynchronous, the synaptic decay time constant needs to be comparable to that of th
82 five cells, with a time to peak of 1.0 ms, a decay time constant of 2.3 ms, and a reversal potential
85 0 and 15 ms after an action potential, had a decay time constant of about 30 ms, and showed no accumu
86 Also, halothane considerably prolonged the decay time constant of evoked IPSCs in pyramidal cells a
87 howed that R(A) resulted in a slowing of the decay time constant of excitatory postsynaptic currents
88 he amplitude but increased its effect on the decay time constant of field EPSPs recorded under condit
89 at 38 h after ischemia; the rising slope and decay time constant of I(A) were accordingly increased a
90 gs progressively decreased the amplitude and decay time constant of miniature end-plate potential (ME
91 RZ (30 microM) increased the monoexponential decay time constant of miniature IPSCs (mIPSCs) in CA1 a
92 urthermore, zinc decreased the amplitude and decay time constant of mIPSCs from developing granule ce
94 opregnanolone caused an increase in the slow decay time constant of spontaneous GABA-mediated IPSCs i
97 enzodiazepine agonist zolpidem increased the decay time constant of the IPSCs of immature granule cel
98 ation had no effect on the peak amplitude or decay time constant of the NMDA component, or the I-V re
99 nnection, amplitude, latency, rise time, and decay time constant of the unitary EPSC were not differe
101 e induced absorption shows quadratic and the decay time constant shows linear dependence on the laser
102 nt changes in membrane conductance and had a decay time constant similar to the membrane time constan
104 ure/volume measurements (-dP/dtmin, pressure decay time constant tau-Glantz, and passive filling stif
105 a significantly higher frequency and faster decay time constant than those recorded from the medulla
106 9 +/- 0.0299 pA pF(-1) (n = 7 cells) and the decay time constant was tau = 790 +/- 76 ms (n = 5).
107 709 +/- 0.0299 pA pF-1 (n = 7 cells) and the decay time constant was tau = 790 +/- 76 ms (n = 5).
108 and the benzodiazepine potentiation of this decay time constant were both significantly increased in
109 ns (10-100 pA, 10 ms rise time constant, 5 s decay time constant) in the presence of various synaptic
110 cay time, weighted decay time constant, slow decay time constant, and, consequently, the total charge
112 GABAA synaptic maximal conductance, synaptic decay time constant, or the mean external excitatory dri
113 ak amplitude, 90-to-10% decay time, weighted decay time constant, slow decay time constant, and, cons
121 r is marked by a significant increase in the decay-time constant for evoked and spontaneous IPSCs and
122 e Pb2+ changed neither the amplitude nor the decay-time constant of the MPSCs, Pb2+-induced changes i
123 but not young, rats exhibit a twofold longer decay time-constant and temporally summate a train of st
124 erculoventral cells had significantly faster decay time constants (0.35-0.40 msec) than did those fro
125 bicuculline methiodide (BMI), and had longer decay time constants (4.5-6.0 ms) that were modulated by
127 ell recordings at 22 degrees C, the weighted decay time constants (tau(w)) of spontaneous IPSCs (sIPS
128 tants were increased by 195% and evoked IPSC decay time constants by 220% compared with age-matched c
133 wer IPSCs, with a 2.6-fold difference in the decay time constants of spontaneous IPSCs and a 5.3-fold
134 an inactivating potassium (IA) current with decay time constants of up to 225 ms, and small-amplitud
135 ated quantum states of multiple nuclei, have decay time constants that may exceed T1 by large factors
136 s methods for assessing ventricular pressure decay time constants to test whether sensitivity to slig
139 From P0 to P14, both the rise time and the decay time constants were significantly longer than in t
140 ication of L-glutamate to nucleated patches, decay time constants were similar at +/-60 mV in the pre
141 current amplitudes, altered desensitization decay time constants, and reduced GlyR clustering and sy
148 ak) of fAMPAsEPSCs was 1.5+/-1.05 ms and the decay time could be fitted with a single exponential wit
149 ch as high solubility and short fluorescence decay time, could be obtained from fluorophors composed
152 prolonged (63 +/- 14 %; mean +/- s.e.m.) the decay time course of miniature IPSCs (mIPSCs) without si
153 rong correlation between prolongation of the decay time course of sIPSCs and potentiation of single-c
154 e whether the magnitude of modulation of the decay time course of sIPSCs correlates with the extent o
159 The averaged macroscopic current exhibited a decay time course which was well described by a single e
162 receptor-mediated sEPSC with slower rise and decay time courses and larger peak amplitudes (sAMPAsEPS
164 bly phased bends, the relative birefringence decay times depend on the flexibility of each bend, not
166 Although pure GABAergic and glycinergic decay times did not differ depending on HM location, the
168 e temperature dependence of the luminescence decay time enables intrinsic temperature compensation of
169 ied out, as a function of expected diffusion decay time for a particular solute, and show that use of
170 seconds) was markedly greater than the half decay time for cytosolic Ca2+ sparks (31.2+/-0.56 ms) ob
171 thdrawal is due to a sixfold decrease in the decay time for GABA currents and consequent decreased in
177 g by electron transfer predict heterogeneous decay times from 50-500 ps that agree with our experimen
179 m2 mass transporting area), 90 s 10-90% rise/decay time glucose electrode, and an on-the-skin electro
182 er miniature inhibitory postsynaptic current decay time in null mice, with no change in miniature inh
183 ons displayed a larger amplitude and shorter decay time in spontaneously hypertensive rats (SHRs) tha
184 addition, the normal downregulation of NMDAR decay time in sSC neurons at P11 was absent after NMDA t
189 ontains at least seven transient states with decay times in the range from 10 micros to 200 ms, but t
191 Under control conditions, GABA(A slow) IPSC decay times increased linearly with membrane depolarizat
192 tic trauma (AT, loud sounds) slow AMPAR-EPSC decay times, increasing GluA1 and decreasing GluA4 mRNA.
193 es, such as (Pro-Pro-Gly)(10), show multiple decay times, indicating multiple scission locations and
198 he level of screening achieved at nanosecond decay times is shown to change with the coverage of elec
200 mplicated in developmental plasticity, shows decay time kinetics that shorten postnatally as NR2A sub
201 ncreased, while GABA-ACh pairing affects the decay time leading to elevated calcium levels during the
202 l populations, noise can render the measured decay times meaningless for small amplitude Ca2+ sparks.
203 lifetime of RuCon A allows phase-modulation decay time measurements using an amplitude-modulated blu
204 elationship between Ca2+ spark amplitude and decay time might be used to distinguish Ca2+ sparks from
205 curate diffusion constants for both species, decay times must be bounded by adequate minimum and maxi
207 ltrafast (tens of femtoseconds) hot electron decay times observed experimentally arise from electron-
208 the similarity of the emission spectrum and decay times observed for one-photon and two-photon excit
210 somer dissociates through thermolysis with a decay time of 14 min at 296 K to form the [6,6]-closed e
211 rong fluorescence at 1544 nm with a measured decay time of 3 ms and an estimated quantum efficiency o
213 ations of OHBI give an estimated first-order decay time of 476 fs for the S(1) state, which is larger
215 cated by increased rectification and reduced decay time of AMPAR-mediated excitatory postsynaptic cur
216 d triplet pair states, but the rapid singlet decay time of approximately 200 ps in solution-grown sin
219 a(2+) exchange activity (indexed by the half decay time of caffeine-elicited Ca(2+) transient) by 27%
221 ampal slices from CIE rats revealed that the decay time of GABAR-mediated miniature inhibitory postsy
224 ion as shown by increasing the frequency and decay time of mEPSCs, and simultaneously inhibiting GABA
229 ith these changes, the current amplitude and decay time of NMDARs in PFC was significantly reduced.
230 te the relatively slow transfer, the overall decay time of PSI-LHCI-LHCII remains fast enough to assu
234 stimulation or uncaging of IP3 increased the decay time of spontaneous Ca(2+) events without changing
237 cerebellar synapses selectively prolongs the decay time of synaptic currents, whereas a switch from G
239 simple strategy to control and modulate the decay time of the functionalized Yb(3+)-doped nanopartic
241 ns is however limited by the relatively fast decay time of the hyperpolarized spin state together wit
242 n of the gamma oscillation, in which the the decay time of the inhibitory cells is critical to the fr
245 hematite, this recombination exhibits a 50% decay time of ~6 ps, ~10(3) times faster than that of Ti
246 cay displays multiexponential character with decay times of 1.2 and 16 ps, and 0.6, 2.2, and 4.2 ns.
248 lipid hydrogen bonds are long lived, showing decay times of 50 ns, and forming strings of lipids, and
249 this issue, we have measured the rotational decay times of a 'gapped-duplex' DNA molecule possessing
250 is described by two kinetic components with decay times of approximately 20 and approximately 200 ns
251 tracellular space, is reflected by increased decay times of neuronal NR2A-mediated NMDA currents.
252 Increasing quantal content also prolonged decay times of pharmacologically isolated alpha7-nAChR-
253 The rise times, areas, half-widths, and decay times of sEPSCs and emEPSCs and interevent interva
254 lay very high luminescence quantum yields at decay times of several tens of mus even in solution unde
256 etectably, in that the amplitudes, areas and decay times of spontaneous miniature EJCs were unchanged
258 second carbamate is evident from bimodal T2 decay times of the approximately 163 ppm peak, indicatin
260 llows a bi-exponential time dependence, with decay times of the order of picoseconds, indicating that
261 ground state bacteriorhodopsin and the mean decay times of the photocycle M-state intermediates.
262 the similarities between the characteristic decay times of the time correlation function, as obtaine
265 d at P12-13; (3) the kinetics (rise time and decay time) of both mEPSCs and mIPSCs accelerated with a
266 t pure graphane has a very long nonradiative decay time, on the order of 100 ns, while epoxy- and hyd
269 s with multiple peaks and increased rise and decay times, reflecting "desynchronized" SV fusion.
270 ependent long-term depression decreased EPSC decay time, revealing a 'late' current that is present w
272 ectra (TRES) indicate that this fluorescence decay time should be ascribed to a highly quenched confo
274 EPSCs in low quantal-content conditions had decay times similar to the time course of receptor deact
275 c membrane input resistance or EPSC rise and decay time suggested that the effects of PPs on EPSCs we
276 large drop in NMDA receptor (NMDAR) current decay time synchronized across all neurons and occurring
277 with a 2-fold faster rise and 7-fold faster decay time (t1/2 of 40 ms) than GCaMP6f, indicating that
279 th GCaMP6fu displaying fluorescence rise and decay times (t1/2) of 1 and 3 ms (37 degrees C) in vitro
280 s between the current-time integrals or half-decay times (t1/2), regardless of whether or not neostig
281 ce (TEB) measurement in which the rotational decay times (taugap) of DNA molecules possessing central
285 2O8+x characterized by an excited population decay time that maps directly to a discrete component of
286 s that the mutant subunit increases synaptic decay times, thereby prolonging postsynaptic activity.
288 s accompanied by a nearly 2-fold increase in decay time, to values that are indistinguishable from th
289 anostructures exhibit extremely long carrier decay times up to 20 micros that are combined with high
291 with membrane depolarization, and this IPSC decay time voltage dependence was not significantly alte
293 The spike amplitude as well as rise and decay time were comparable with those measured by carbon
297 by the histograms of the rise times and half-decay times, which revealed modes at 38 and 65 ms, respe
299 e scaling of the characteristic fluorescence decay time with the vesicle diameter and the buildup of
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