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1 rized by a fast (tau(1)) and a slow (tau(2)) decay time.
2 and prevents the shortening in NMDAR current decay time.
3 diction of brightness, stability, Phi(f), or decay time.
4 ctivity did not significantly alter the EPSC decay time.
5 cholinesterase inhibitor, prolonged the EPSC decay time.
6 cumulation but had no effect on the rise and decay time.
7 velength emission associated with the longer decay time.
8  using a long lifetime standard with a known decay time.
9  the deactivation kinetics by prolonging the decay time.
10 uCon A, resulting in increased intensity and decay time.
11 y, and the correlation between amplitude and decay time.
12 2+ transient amplitude and prolonged the 50% decay time.
13  (cis) isomer in DOPC (DPPC) presents a fast decay time.
14 smission to Bergmann glia and decreased EPSC decay time.
15 ise times, amplitudes, charge transfers, and decay times.
16  to CaN to maintain NMDAR currents with long decay times.
17  complexes led to variations in fluorescence decay times.
18 of the eIPSC could be distinguished by their decay times.
19 elations with relatively long characteristic decay times.
20 imilar to WT bumps, but with slightly slower decay times.
21 s of more than PhiPL = 90% at short emission decay times.
22 mations that fit the experimentally measured decay times.
23 current amplitudes with significantly faster decay times.
24 tes of LOV390 formation but exhibited adduct decay times 1 order of magnitude faster than wild type.
25 netically fast (rise time, 0.32 +/- 0.02 ms; decay time, 1.66 +/- 0.18 ms; mean +/- SD; n = 6 cells),
26 (31%), and prolongation of the Ca(2+) signal decay time (165%) than overexpression (2-fold) of wild t
27 imes greater), and decayed more slowly (half-decay time 189 ms for strontium and 32 ms for calcium).
28 entedly fast conductance cycle (current half-decay time 2-4 ms depending on voltage).
29 tivity exhibit significantly longer AHP half-decay times (24.67 vs. 11.02 ms) and greater AHP amplitu
30 d trimer system, the fluorescence anisotropy decay time (35 fs) is found to be much shorter than that
31 nses were slow (rise time, 1.28 +/- 0.35 ms; decay time, 6.71 +/- 1.46 ms; n = 8 cells).
32  pressure is typically much shorter than the decay time after cessation or decline in the volume of d
33 ecays at intermediate pH values and that the decay time amplitudes are greatly dependent on pH.
34 e different membrane phases via fluorescence decay time analysis, making this new probe versatile for
35 d in gas phase by measuring the fluorescence decay time and ion-neutral collision cross sections (CCS
36    Elevating quantal content lengthened EPSC decay time and prolonged both the fast (alpha7-nAChR-med
37 osolic Ca(2+) followed by an increase in the decay time and the spread of the spontaneous Ca(2+) spik
38 wo adjustable parameters instead of the many decay times and amplitudes required in standard analysis
39  that elicited responses show layer-specific decay times and frequency-dependent facilitation.
40 rents in mutant neurons have slower rise and decay times and lower NMDAR/AMPAR current ratios.
41 ic modeling of temperature-dependent singlet decay times and quantum yields of fluorescence, isomeriz
42 cal role in recovering accurate and reliable decay times and resulting diffusion constants.
43 peak parameters including shorter rise time, decay time, and half-width as compared to a bare carbon
44 2+) signaling, the specific effects on peak, decay time, and/or frequency were different.
45  peak amplitudes, prolonged current rise and decay times, and altered responses to benzodiazepine ago
46 that display long emission wavelengths, long decay times, and high quantum yields.
47 etected and analysed for amplitude, rise and decay times, and latency.
48 (A) receptor, the peak amplitudes, 90-to-10% decay times, and total charge transfer of spontaneous mi
49 nged both GABA(A fast) and GABA(A slow) IPSC decay times approximately 2.5 fold, while having little
50 than 10-fold slower in nucleo-olivary cells (decay time, approximately 25 ms) than in large cells ( a
51 s (reductions of approximately 48% in 10-90% decay time, approximately 40% in tau, and approximately
52 eld and comparable reduction of luminescence decay time are observed.
53 ant glycine receptors, we show that response decay times are accelerated by addition of GABA, a weak
54 echnique in which fluorescence excited state decay times are measured as fluorescently labeled cells
55                                          The decay times are shorter for the purines than for the pyr
56     We also rule out small increases in IPSC decay times (as caused by W170S and R414H) as a possible
57 es as photodetectors, for both intensity and decay-time based monitoring of the sensing element's PL.
58 ynapses with multiple release sites the EPSC decay time became faster when release probability was lo
59 rylated by postnatal day 11 as NMDAR current decay times become faster.
60 t on the observation wavelength, with longer decay times being observed at longer wavelengths.
61                The temperature dependence of decay times can be attributed to changes in the viscosit
62 ample, we show that very short synthesis and decay times can perturb the wild-type pattern.
63   2AP does not exhibit the long fluorescence decay time characteristic of the free nucleoside, sugges
64 taining proteins are well fit using only two decay times compared to three required for Trp.
65 uum has a rise time of less than 80 ns and a decay time component of approximately 300 micros.
66     The absorption spectrum and fluorescence decay time components of the complex at room temperature
67 ection into cells had fast rates of rise and decay (time constant, 5-20 ms).
68                                   The second decay time constant (tau 2) was significantly longer in
69  postsynaptic currents (mIPSCs) (predominant decay time constant (tau(decay)), 1.0 ms) in addition to
70 inward current and prolonged the exponential decay time constant (tau) of Ca(2+)-activated Cl- 'tail'
71 ntly with distance from the soma whereas the decay time constant (taudecay) of Delta[Ca2+] decreases
72  an amplitude of 68 +/- 6 pA, and a weighted decay time constant (tauw) of 15.8 +/- 2.9 ms.
73  +/- 0.59 to -4.15 +/- 0.73 nA with the fast decay time constant accelerating from 0.75 +/- 0.09 ms a
74                  However, the miniature IPSC decay time constant and the benzodiazepine potentiation
75 ial; (2) that the ratio between the synaptic decay time constant and the oscillation period be suffic
76 l (time to peak approximately 0.3-0.4 ms and decay time constant approximately 3-6 ms) served as the
77                        In contrast, the IPSC decay time constant depended only on the postsynaptic cl
78                                 However, the decay time constant for STP of the AMPA receptor-mediate
79 tential was approximately 6 min, whereas the decay time constant for STP of the NMDA receptor-mediate
80 e recovery from desensitization is slow (the decay time constant is roughly 500 milliseconds), little
81 ronal firings are asynchronous, the synaptic decay time constant needs to be comparable to that of th
82 five cells, with a time to peak of 1.0 ms, a decay time constant of 2.3 ms, and a reversal potential
83 al of -83 mV, a time to peak of 2.6 ms and a decay time constant of 6.5 ms.
84 r the catalytic effect of temperature on the decay time constant of a synaptic current.
85 0 and 15 ms after an action potential, had a decay time constant of about 30 ms, and showed no accumu
86   Also, halothane considerably prolonged the decay time constant of evoked IPSCs in pyramidal cells a
87 howed that R(A) resulted in a slowing of the decay time constant of excitatory postsynaptic currents
88 he amplitude but increased its effect on the decay time constant of field EPSPs recorded under condit
89 at 38 h after ischemia; the rising slope and decay time constant of I(A) were accordingly increased a
90 gs progressively decreased the amplitude and decay time constant of miniature end-plate potential (ME
91 RZ (30 microM) increased the monoexponential decay time constant of miniature IPSCs (mIPSCs) in CA1 a
92 urthermore, zinc decreased the amplitude and decay time constant of mIPSCs from developing granule ce
93 37), but did not alter the amplitude and the decay time constant of mIPSCs.
94 opregnanolone caused an increase in the slow decay time constant of spontaneous GABA-mediated IPSCs i
95 on progressively decreases the amplitude and decay time constant of spontaneous mEPSPs.
96                                  The average decay time constant of the AR for orientation-tuned cell
97 enzodiazepine agonist zolpidem increased the decay time constant of the IPSCs of immature granule cel
98 ation had no effect on the peak amplitude or decay time constant of the NMDA component, or the I-V re
99 nnection, amplitude, latency, rise time, and decay time constant of the unitary EPSC were not differe
100 ncy, dose-dependent hyperpolarization with a decay time constant on the order of a few seconds.
101 e induced absorption shows quadratic and the decay time constant shows linear dependence on the laser
102 nt changes in membrane conductance and had a decay time constant similar to the membrane time constan
103                                Deactivation (decay time constant tau = 0.6 ms at 24 degrees C) was su
104 ure/volume measurements (-dP/dtmin, pressure decay time constant tau-Glantz, and passive filling stif
105  a significantly higher frequency and faster decay time constant than those recorded from the medulla
106 9 +/- 0.0299 pA pF(-1) (n = 7 cells) and the decay time constant was tau = 790 +/- 76 ms (n = 5).
107 709 +/- 0.0299 pA pF-1 (n = 7 cells) and the decay time constant was tau = 790 +/- 76 ms (n = 5).
108  and the benzodiazepine potentiation of this decay time constant were both significantly increased in
109 ns (10-100 pA, 10 ms rise time constant, 5 s decay time constant) in the presence of various synaptic
110 cay time, weighted decay time constant, slow decay time constant, and, consequently, the total charge
111                             In addition, the decay time constant, but not the amplitude of the mEPSCs
112 GABAA synaptic maximal conductance, synaptic decay time constant, or the mean external excitatory dri
113 ak amplitude, 90-to-10% decay time, weighted decay time constant, slow decay time constant, and, cons
114 f speckle patterns to obtain the exponential decay time constant, tau.
115 i, resulting primarily from a slowing of the decay time constant.
116 12), without affecting the amplitude and the decay time constant.
117 PSCs) without changing the amplitude and the decay time constant.
118 e difference was accounted for by the second decay time constant.
119 affecting their mean amplitude, rise time or decay time constant.
120 of the unitary IPSC: latency, rise time, and decay time constant.
121 r is marked by a significant increase in the decay-time constant for evoked and spontaneous IPSCs and
122 e Pb2+ changed neither the amplitude nor the decay-time constant of the MPSCs, Pb2+-induced changes i
123 but not young, rats exhibit a twofold longer decay time-constant and temporally summate a train of st
124 erculoventral cells had significantly faster decay time constants (0.35-0.40 msec) than did those fro
125 bicuculline methiodide (BMI), and had longer decay time constants (4.5-6.0 ms) that were modulated by
126 , and zolpidem-sensitive mIPSCs had weighted decay time constants (tau(w)) of 4-6 ms.
127 ell recordings at 22 degrees C, the weighted decay time constants (tau(w)) of spontaneous IPSCs (sIPS
128 tants were increased by 195% and evoked IPSC decay time constants by 220% compared with age-matched c
129                                          The decay time constants change e-fold per 22.1 mV above the
130 e in Ng+/+ compared with Ng-/- mice, but the decay time constants did not differ.
131                                              Decay time constants of cytosolic Ca2+ transients from c
132                                              Decay time constants of EPSCs increased (or decreased) i
133 wer IPSCs, with a 2.6-fold difference in the decay time constants of spontaneous IPSCs and a 5.3-fold
134  an inactivating potassium (IA) current with decay time constants of up to 225 ms, and small-amplitud
135 ated quantum states of multiple nuclei, have decay time constants that may exceed T1 by large factors
136 s methods for assessing ventricular pressure decay time constants to test whether sensitivity to slig
137                             Spontaneous IPSC decay time constants were increased by 195% and evoked I
138                                 EPC and MEPC decay time constants were prolonged because of a slowed
139   From P0 to P14, both the rise time and the decay time constants were significantly longer than in t
140 ication of L-glutamate to nucleated patches, decay time constants were similar at +/-60 mV in the pre
141  current amplitudes, altered desensitization decay time constants, and reduced GlyR clustering and sy
142 ated EPSCs showed an unusually wide range of decay time constants, from <5 to >500 ms.
143 (mIPSCs) without affecting the rise time and decay time constants.
144  ACh concentrations and the synaptic current decay time constants.
145  GABA(A) receptors, isoflurane prolonged the decay-time constants of both eIPSCs and mIPCSs.
146 EPSCs accelerate over development to achieve decay time-constants of 2.5 ms.
147 tal maturation accelerate to sub-millisecond decay time-constants.
148 ak) of fAMPAsEPSCs was 1.5+/-1.05 ms and the decay time could be fitted with a single exponential wit
149 ch as high solubility and short fluorescence decay time, could be obtained from fluorophors composed
150                                          The decay time course of excitatory postsynaptic currents ge
151                       Here, we show that the decay time course of GABAergic inhibitory synaptic curre
152 prolonged (63 +/- 14 %; mean +/- s.e.m.) the decay time course of miniature IPSCs (mIPSCs) without si
153 rong correlation between prolongation of the decay time course of sIPSCs and potentiation of single-c
154 e whether the magnitude of modulation of the decay time course of sIPSCs correlates with the extent o
155 MPARs in stellate cells but did not slow the decay time course of synaptic currents.
156                                          The decay time course of the patch currents in response to b
157                                 The rise and decay time course of the sAMPAsEPSCs and NMDAsEPSCs were
158                             Furthermore, the decay time course ofEPSCs mediated by GluR2-containing r
159 The averaged macroscopic current exhibited a decay time course which was well described by a single e
160 increasing its width and by slowing down its decay time course.
161 rents (EACs) with no effect on the NMDAR EAC decay time course.
162 receptor-mediated sEPSC with slower rise and decay time courses and larger peak amplitudes (sAMPAsEPS
163                       In parallel, NMDA EPSC decay times decreased over a similar developmental time
164 bly phased bends, the relative birefringence decay times depend on the flexibility of each bend, not
165 ay that is generally stepwise linear and the decay time depends strongly on [erg].
166      Although pure GABAergic and glycinergic decay times did not differ depending on HM location, the
167 ive decay paths reduces the overall emission decay time distinctly.
168 e temperature dependence of the luminescence decay time enables intrinsic temperature compensation of
169 ied out, as a function of expected diffusion decay time for a particular solute, and show that use of
170  seconds) was markedly greater than the half decay time for cytosolic Ca2+ sparks (31.2+/-0.56 ms) ob
171 thdrawal is due to a sixfold decrease in the decay time for GABA currents and consequent decreased in
172                                  The average decay time for PSI-LHCI-LHCII is approximately 65 ps upo
173                        The measured rise and decay time for the 1(st) order is 7.62 ms and 6.75 ms, a
174 he formation time for both peaks matches the decay time for the delocalized LMCT excited state.
175 that the decay is exponential with different decay times for other, simpler dipeptides.
176        A significant correlation of rise and decay times for the overall population of unitary IPSCs
177 g by electron transfer predict heterogeneous decay times from 50-500 ps that agree with our experimen
178 ed consistently over a wide range of tension decay times from a few seconds to over 100 s.
179 m2 mass transporting area), 90 s 10-90% rise/decay time glucose electrode, and an on-the-skin electro
180           Long "in-bag" (129)Xe polarization decay times have been measured (T1 approximately 38 min
181 w the carrier-recombination-limited hologram decay time in a photorefractive crystal.
182 er miniature inhibitory postsynaptic current decay time in null mice, with no change in miniature inh
183 ons displayed a larger amplitude and shorter decay time in spontaneously hypertensive rats (SHRs) tha
184 addition, the normal downregulation of NMDAR decay time in sSC neurons at P11 was absent after NMDA t
185 er Ames medium, we also observed >30% longer decay time in the PCP2-null rod bipolar cells.
186 e early and rapid reduction of NMDAR current decay time in visual neurons.
187              However, the invariance of *Trp decay times in ferric and ferrous Mbs raises the questio
188  release, as measured by EPSP amplitudes and decay times in postsynaptic neurons.
189 ontains at least seven transient states with decay times in the range from 10 micros to 200 ms, but t
190                              We find that PL decay times increase by a factor of 2 on addition of CsC
191  Under control conditions, GABA(A slow) IPSC decay times increased linearly with membrane depolarizat
192 tic trauma (AT, loud sounds) slow AMPAR-EPSC decay times, increasing GluA1 and decreasing GluA4 mRNA.
193 es, such as (Pro-Pro-Gly)(10), show multiple decay times, indicating multiple scission locations and
194 light response directly, and accelerates the decay time indirectly via the inhibitory network.
195                   Thus, the overall emission decay time is distinctly reduced.
196        When bound to a protein or lipid, the decay time is near 3 microseconds and the quantum yield
197                                The spin echo decay time is ~40 mus, both with one and four echo pulse
198 he level of screening achieved at nanosecond decay times is shown to change with the coverage of elec
199  then becomes saturated, whereas the average decay time keeps increasing linearly.
200 mplicated in developmental plasticity, shows decay time kinetics that shorten postnatally as NR2A sub
201 ncreased, while GABA-ACh pairing affects the decay time leading to elevated calcium levels during the
202 l populations, noise can render the measured decay times meaningless for small amplitude Ca2+ sparks.
203  lifetime of RuCon A allows phase-modulation decay time measurements using an amplitude-modulated blu
204 elationship between Ca2+ spark amplitude and decay time might be used to distinguish Ca2+ sparks from
205 curate diffusion constants for both species, decay times must be bounded by adequate minimum and maxi
206 cay schemes and the dramatic drop in channel decay time observed at erg mol % = 50.
207 ltrafast (tens of femtoseconds) hot electron decay times observed experimentally arise from electron-
208  the similarity of the emission spectrum and decay times observed for one-photon and two-photon excit
209 the same order as the S(1)-S(0) nonradiative decay time obtained previously for the (6,4) CNT.
210 somer dissociates through thermolysis with a decay time of 14 min at 296 K to form the [6,6]-closed e
211 rong fluorescence at 1544 nm with a measured decay time of 3 ms and an estimated quantum efficiency o
212 tion time of 1.68 +/- 0.05 ps and an initial decay time of 31.7 ps.
213 ations of OHBI give an estimated first-order decay time of 476 fs for the S(1) state, which is larger
214  assay for glucose based on the luminescence decay time of a long lifetime metal-ligand complex.
215 cated by increased rectification and reduced decay time of AMPAR-mediated excitatory postsynaptic cur
216 d triplet pair states, but the rapid singlet decay time of approximately 200 ps in solution-grown sin
217 mately 20-500 fF (mean = 123 fF) with 10-90% decay time of approximately 30-500 ms.
218 , because of the low proton density and fast decay time of bone tissue.
219 a(2+) exchange activity (indexed by the half decay time of caffeine-elicited Ca(2+) transient) by 27%
220                         The effect upon EPSC decay time of changing quantal content was 5-10 times mo
221 ampal slices from CIE rats revealed that the decay time of GABAR-mediated miniature inhibitory postsy
222 educed light spread in the detector and fast decay time of GSO.
223  cells in adult female rats and prolongs the decay time of IPSCs in these cells.
224 ion as shown by increasing the frequency and decay time of mEPSCs, and simultaneously inhibiting GABA
225                                Amplitude and decay time of mEPSCs, decay time of mIPSCs, and spine si
226          Amplitude and decay time of mEPSCs, decay time of mIPSCs, and spine size were unaltered.
227                             In addition, the decay time of NMDA currents was decreased in BDNF(Met/Me
228  a rapid, activity-induced shortening in the decay time of NMDA receptor (NMDAR) currents.
229 ith these changes, the current amplitude and decay time of NMDARs in PFC was significantly reduced.
230 te the relatively slow transfer, the overall decay time of PSI-LHCI-LHCII remains fast enough to assu
231 o MIMG resulted in a decreased intensity and decay time of RuCon A.
232          At the same time, the rise time and decay time of sEPSCs significantly decreased, suggesting
233 without affecting mean current and inhibited decay time of sIPSCs.
234 stimulation or uncaging of IP3 increased the decay time of spontaneous Ca(2+) events without changing
235                The amplitude, rise time, and decay time of SSCs were not affected, indicating a presy
236                      The emission maxima and decay time of such tandem luminophores can be readily ad
237 cerebellar synapses selectively prolongs the decay time of synaptic currents, whereas a switch from G
238                              We show how the decay time of the El Nino anomaly in this data set can b
239  simple strategy to control and modulate the decay time of the functionalized Yb(3+)-doped nanopartic
240 ion, both protein kinase A and C shorten the decay time of the glycine current.
241 ns is however limited by the relatively fast decay time of the hyperpolarized spin state together wit
242 n of the gamma oscillation, in which the the decay time of the inhibitory cells is critical to the fr
243                                          The decay time of the oscillations is 14.5 +/- 2.7 minutes f
244 delay) is crucial because tdelay defines the decay time of the reactive intermediate.
245  hematite, this recombination exhibits a 50% decay time of ~6 ps, ~10(3) times faster than that of Ti
246 cay displays multiexponential character with decay times of 1.2 and 16 ps, and 0.6, 2.2, and 4.2 ns.
247 able properties overall, which included fast decay times of 2 ns.
248 lipid hydrogen bonds are long lived, showing decay times of 50 ns, and forming strings of lipids, and
249  this issue, we have measured the rotational decay times of a 'gapped-duplex' DNA molecule possessing
250  is described by two kinetic components with decay times of approximately 20 and approximately 200 ns
251 tracellular space, is reflected by increased decay times of neuronal NR2A-mediated NMDA currents.
252    Increasing quantal content also prolonged decay times of pharmacologically isolated alpha7-nAChR-
253      The rise times, areas, half-widths, and decay times of sEPSCs and emEPSCs and interevent interva
254 lay very high luminescence quantum yields at decay times of several tens of mus even in solution unde
255  the voltage-time integrals, and between the decay times of spontaneous and evoked signals.
256 etectably, in that the amplitudes, areas and decay times of spontaneous miniature EJCs were unchanged
257 size, without affecting the average rise and decay times of synaptic currents.
258  second carbamate is evident from bimodal T2 decay times of the approximately 163 ppm peak, indicatin
259                           The characteristic decay times of the autocorrelation functions report the
260 llows a bi-exponential time dependence, with decay times of the order of picoseconds, indicating that
261  ground state bacteriorhodopsin and the mean decay times of the photocycle M-state intermediates.
262  the similarities between the characteristic decay times of the time correlation function, as obtaine
263          This is reflected in short emission decay times of the triplet states of only 34 mus (1) and
264                                The rise- and decay-times of local mIPSCs were also independent of the
265 d at P12-13; (3) the kinetics (rise time and decay time) of both mEPSCs and mIPSCs accelerated with a
266 t pure graphane has a very long nonradiative decay time, on the order of 100 ns, while epoxy- and hyd
267        Nonlinear fitting of the fluorescence decay times provides activation parameters for singlet d
268                 Two months after AT the EPSC decay times recovered to control values.
269 s with multiple peaks and increased rise and decay times, reflecting "desynchronized" SV fusion.
270 ependent long-term depression decreased EPSC decay time, revealing a 'late' current that is present w
271 e of the fluctuations controlled by the NMDA decay time scale.
272 ectra (TRES) indicate that this fluorescence decay time should be ascribed to a highly quenched confo
273 ed patches to synaptically released GABA had decay times similar to brief pulse responses.
274  EPSCs in low quantal-content conditions had decay times similar to the time course of receptor deact
275 c membrane input resistance or EPSC rise and decay time suggested that the effects of PPs on EPSCs we
276  large drop in NMDA receptor (NMDAR) current decay time synchronized across all neurons and occurring
277  with a 2-fold faster rise and 7-fold faster decay time (t1/2 of 40 ms) than GCaMP6f, indicating that
278 tor (IP3R), significantly prolonged the half-decay time (t1/2) of IP3-induced Ca2+ transients.
279 th GCaMP6fu displaying fluorescence rise and decay times (t1/2) of 1 and 3 ms (37 degrees C) in vitro
280 s between the current-time integrals or half-decay times (t1/2), regardless of whether or not neostig
281 ce (TEB) measurement in which the rotational decay times (taugap) of DNA molecules possessing central
282       The multiquantal events exhibit faster decay time than the GluR4 receptor desensitization time
283             Pure GABAergic events had slower decay times than glycinergic events.
284       These events had slower rise times and decay times than sparks and were more heterogeneous.
285 2O8+x characterized by an excited population decay time that maps directly to a discrete component of
286 s that the mutant subunit increases synaptic decay times, thereby prolonging postsynaptic activity.
287                                       Median decay time to half-life ranged from 13.8 to 15.1 months.
288 s accompanied by a nearly 2-fold increase in decay time, to values that are indistinguishable from th
289 anostructures exhibit extremely long carrier decay times up to 20 micros that are combined with high
290                                      The two decay times vary only mildly with the water concentratio
291  with membrane depolarization, and this IPSC decay time voltage dependence was not significantly alte
292               IPSC peak amplitude, 90-to-10% decay time, weighted decay time constant, slow decay tim
293      The spike amplitude as well as rise and decay time were comparable with those measured by carbon
294 pproximately 400 msec, whereas rise time and decay time were not altered significantly.
295                   However, faster rising and decay times were obtained for the bright to dark than th
296        In contrast, GlyR IPSC and NMDAR-EPSC decay times were unchanged.
297 by the histograms of the rise times and half-decay times, which revealed modes at 38 and 65 ms, respe
298 opic transmission to glia and decreased EPSC decay time with closely similar time courses.
299 e scaling of the characteristic fluorescence decay time with the vesicle diameter and the buildup of
300 on, significantly increases glycinergic IPSC decay times without causing motor side effects.

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