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1 ty of both Purkinje cells and neurons of the deep cerebellar nuclei.
2 g inhibition of cerebellar output neurons in deep cerebellar nuclei.
3 upts inhibition of the inferior olive by the deep cerebellar nuclei.
4 flexible selection of signals for output to deep cerebellar nuclei.
5 All injections labeled the deep cerebellar nuclei.
6 uced in Purkinje cells, but increased in the deep cerebellar nuclei.
7 otocols can drive synaptic plasticity in the deep cerebellar nuclei.
8 um without directly affecting neurons in the deep cerebellar nuclei.
9 stantia nigra pars reticulata, pallidum, and deep cerebellar nuclei.
10 enhanced hyperexcitability of neurons in the deep cerebellar nuclei.
11 sent in the olfactory bulb, red nucleus, and deep cerebellar nuclei.
12 es in the cerebellar cortex and in the lower deep cerebellar nuclei.
13 ulations vulnerable to weaver, including the deep cerebellar nuclei.
14 lum, it is expressed in Purkinje neurons and deep cerebellar nuclei.
15 ibular nuclei, inferior olivary complex, and deep cerebellar nuclei.
16 nsmission at synapses from Purkinje cells to deep cerebellar nuclei and at vestibular synapses in mic
17 onto discrete populations of neurons in the deep cerebellar nuclei and brainstem vestibular nuclei.
19 amatergic premotor projection neurons in the deep cerebellar nuclei and GABAergic neurons that feed b
21 ons from layers four and five of the cortex, deep cerebellar nuclei and other localized brain regions
22 clei, hypothalamus, midbrain, pons, medulla, deep cerebellar nuclei and spinal cord, with tau-immunor
25 components simulating cerebellar cortex and deep cerebellar nuclei, and it received input from a mid
26 trogliosis and vacuolation of neurons in the deep cerebellar nuclei, and the severe vacuolation of th
27 basal forebrain, the vestibular complex, the deep cerebellar nuclei, and the trapezoid body, a patter
29 or motor recovery, and lesions affecting the deep cerebellar nuclei are not fully compensated at any
30 s onto a Purkinje cell or onto a cell in the deep cerebellar nuclei become eligible for plasticity on
33 isinhibition of the cerebellar cortex on the deep cerebellar nuclei could treat oculopalatal tremor.
35 al lines of evidence have indicated that the deep cerebellar nuclei (DCN) are a site of memory storag
40 rat cerebellum, PNNs are found around large, deep cerebellar nuclei (DCN) neurons and Golgi neurons a
43 se, may be initiated by hyperexcitability of deep cerebellar nuclei (DCN) secondary to loss of inhibi
44 s within the mature fastigial pathway of the deep cerebellar nuclei (DCN), a region critical for bala
45 ween Purkinje neurons and the neurons of the deep cerebellar nuclei (DCN), a site that has been impli
46 reactive to several brain regions, including deep cerebellar nuclei (DCN), globus pallidus, and thala
47 requency bursting activity in neurons of the deep cerebellar nuclei (DCN), which comprise the bulk of
55 ibuted between the cerebellar cortex and the deep cerebellar nuclei; (ii) the cerebellar cortex plays
56 importance of the cerebellar cortex and the deep cerebellar nuclei in eyeblink conditioning is uncle
57 of the parabrachial, lateral lemniscal, and deep cerebellar nuclei, in addition to cerebellar granul
58 k comprising the inferior olive, vermis, and deep cerebellar nuclei including the dentate nucleus dur
59 ons but strikingly similar to neurons in the deep cerebellar nuclei, indicating a common role for int
60 making microelectrode penetrations into the deep cerebellar nuclei (mainly nucleus interpositus) of
61 f vestibular signals from the vestibular and deep cerebellar nuclei may be important components of fu
62 nput that can be achieved in this way in the deep cerebellar nuclei may be particularly important to
63 uit, with relative increases in perfusion in deep cerebellar nuclei (medial, interposed, lateral), th
66 ABAA receptor-mediated monosynaptic IPSPs in deep cerebellar nuclei neurons by stimulation of Purkinj
68 r Purkinje cells (which possess NR1 and 2D), deep cerebellar nuclei (NR1, 2A, 2B and 2D) and spinal c
69 ollowing bilateral lesions targeting lateral deep cerebellar nuclei, rats were subjected to a bridge
70 nular cell layer, and loss of neurons in the deep cerebellar nuclei; spheroids and loss of myelinated
72 inclusions at atypical sites (e.g. thalamus, deep cerebellar nuclei) that are not typical for Lewy bo
73 TXN1 messenger RNA levels were >/=30% in the deep cerebellar nuclei, the cerebellar cortex, inferior
74 -sensitive neurons in the most medial of the deep cerebellar nuclei, the rostral fastigial nucleus, w
76 indbrain, including, but not limited to, the deep cerebellar nuclei, the trapezoid body, the red nucl
77 coding of information also allows neurons of deep cerebellar nuclei to use a simple averaging mechani
78 glutamatergic neurons, namely neurons of the deep cerebellar nuclei, unipolar brush cells, and the la
79 luorescent protein (rAAV1.miS1eGFP) into the deep cerebellar nuclei using magnetic resonance imaging
80 iple brainstem motor nuclei, inferior olive, deep cerebellar nuclei, vestibular nuclear complex, nucl
81 cleus, oculomotor nucleus, substantia nigra, deep cerebellar nuclei, vestibular nucleus, and the thal
82 es prior to E12.5, with the exception of the deep cerebellar nuclei, we find that Math1 cells migrate
83 binding and AT2 receptor mRNA levels in the deep cerebellar nuclei were also not affected by 3-acety
84 etected in the putamen, globus pallidus, and deep cerebellar nuclei, where the most dense areas of 8B
85 ction of Purkinje-cell axon terminals in the deep cerebellar nuclei, whereas the dendritic trees grew
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