ライフサイエンスコーパス: default mode network

1 sk-negative" patterns of activity (e.g., the default mode network).

2 es, from the brain's salience network to the default mode network.

3 gnals in multiple expression datasets in the default mode network.

4 , and closely corresponded to regions of the default mode network.

5 beyond the temporal lobe, especially in the default mode network.

6 th greater activity in medial regions of the default mode network.

7 orrelated with activity in the task-negative default mode network.

8 in heteromodal hubs, and particularly in the default mode network.

9 whereas the dorsomedial module resembles the default mode network.

10 decreased functional connectivity within the default mode network.

11 left and right hippocampus with the anterior default mode network.

12 , parasympathetic regions predominate in the default mode network.

13 e network activation and deactivation of the default mode network.

14 on both the associated brain network and the default mode network.

15 revealed that these regions belonged to the default mode network.

16 e networks and inhibition of activity in the default mode network.

17 orting attention, cognitive control, and the default mode network.

18 ons of several neurotransmitters driving the default mode network.

19 ithin an intrinsic connectivity network, the default mode network.

20 y regions in the visual cortex and posterior default mode network.

21 ience network, dorsal attention network, and default mode network.

22 esonance imaging with a focus on the brain's default mode network.

23 enerally overlapped with the distribution of default mode network.

24 tentional capacity and tends to activate the default mode network.

25 aused by functional disconnection within the default mode network.

26 nternal organization, closely related to the default-mode network.

27 oscillatory power were seen in areas of the default-mode network.

28 l network and negative correlations with the default-mode network.

29 unctional connectivity mainly located in the default-mode network.

30 yzed resting-state data from the core of the default-mode network.

31 ogy, include the structural substrate of the default-mode network.

32 s that included dorsal/ventral attention and default mode networks.

33 right-biased ICs was connected to social or default mode networks.

34 mparisons of the salience, sensorimotor, and default mode networks.

35 networks including the dorsal attention and default mode networks.

36 uations were observed in fronto-parietal and default mode networks.

37 r insula modulation of central executive and default mode networks.

38 attention, central executive, salience, and default mode networks.

39 erior cingulate cortices, core nodes of the "default mode" network.

40 arietal, subcortical, cingulo-opercular, and default-mode networks.

41 in symptomatogenic areas (9/20; 45%) and the default mode network (13/20; 65%).

42 modulate long-range connectivity within the default mode network [15].

43 lacked the typical anticorrelation with the default mode network; 2) hypoconnectivity between left d

44 vity disequilibrium between the salience and default-mode networks, a finding of potential pathophysi

45 l beliefs produced increased activity in the default mode network-a set of interconnected structures

46 The posterior hippocampus and the associated default-mode network, across both resting-state connecti

47 role for the salience network in regulating default mode network activation.

48 tive control from difficulties shifting from default mode network activity to task-positive network a

49 nspection of the spatial maps related to the default mode network and a frontoparietal task control n

50 cortex and functional connectivities in the default mode network and central executive network in th

51 , we examined its influence on resting state default mode network and DN connectivity in healthy huma

52 ngs extend previous research implicating the default mode network and dopaminergic dysfunction in ADH

53 (ii) high connectivity between the posterior default mode network and hubs of high connectivity (many

54 tivity between the nucleus accumbens and the default mode network and increased connectivity between

55 s, and show that anticorrelation between the default mode network and parietal regions of the dorsal

56 It is a key node in the default mode network and shows increased activity when i

57 dren exhibited hyperconnectivity between the default mode network and subgenual anterior cingulate co

58 hronization) that affects key regions of the default mode network and temporal areas.

59 The anti-correlation strength between the default mode network and the central executive network w

60 vity and diminished connectivity between the default mode network and the cingulo-opercular network.

61 te aberrant involvement of the insula in the default mode network and the frontal frontoparietal task

62 structural connectivity between nodes of the default mode network and the frontal-thalamo-caudate reg

63 scussed in the context of MPH effects on the default mode network and the possible role of the defaul

64 divergent impact these disorders have on the default mode network and the salience network.

65 first primarily involves the regions of the default mode network and the second comprises the fronta

66 ork disruption, affecting key regions of the default mode network and the temporal cortex.

67 he precentral cortex, prefrontal cortex, and default mode network and these brain hyper-responses wer

68 Alterations in coupling of the salience and default mode networks and the inability to disengage fro

69 modes," including several that subdivide the default-mode network (and the regions anticorrelated wit

70 nal connectivity at rest (coupling with the "default mode network" and "frontoparietal control system

71 ch as Alzheimer's disease, which targets the default mode network, and behavioural variant frontotemp

72 e functional connectivities of the posterior default mode network, and positively correlated with fro

73 lts highlight the central role of the sgACC, default mode network, and salience network as predictors

74 d responsivity in the nucleus accumbens, the default mode network, and the cingulo-opercular network.

75 h functional connectivities in the posterior default mode network, and the frontal default mode netwo

76 posterior hippocampal connectivity with the default-mode network, and anterior hippocampal connectiv

77 R sample compared with the HC sample for the default-mode network, and increased covariance was obser

78 BOLD response in specific nodes of the default mode network (angular gyri, posterior cingulate,

79 s between SN, central executive network, and default mode network are a reproducible feature of child

80 network (SN), central executive network, and default mode network are dysregulated in children with A

81 sis and find that regions of interest within default mode network are encoding task-relevant informat

82 show a stronger deactivation of parts of the default mode network, as compared to more intelligent pe

83 These regions include core nodes of the default mode network, as well as multimodal association

84 ty between the precuneus and the rest of the default mode network at rest.

85 creased blood flow to the major nodes of the default mode network became more pronounced and widespre

86 Interactions within the default mode network can be assessed using resting state

87 reductions were especially pronounced in the default mode network, closely matching the previously de

88 ral dementia have reduced recruitment of the default mode network compared with healthy control subje

89 ut C9orf72 expansions exhibited increases in default mode network connectivity compared to controls a

90 ontrol subjects; chi(2) = 8.6, p = .003) and default mode network connectivity during a separate rest

91 reas the thalamus-medial visual and thalamus-default mode network connectivity emerged later, at 1 ye

92 ses revealed salience network disruption and default mode network connectivity enhancement in C9orf72

93 nses were found to have less contribution to default mode network connectivity in individuals with au

94 salience network connectivity and heightened default mode network connectivity.

95 ain network architecture in the salience and default-mode networks consistent with clinical manifesta

96 The default mode network consistently decreases its activity

97 The brain's default mode network consists of discrete, bilateral and

98 edicted change in memory, with no additional default mode network contributions.

99 ic connectivity imaging studies suggest that default mode network degradation in AD is accompanied by

100 Regions of the default-mode network demonstrate lower variability in pa

101 Several regions, mostly localized to the default mode network, demonstrated negative subsequent m

102 o either the dorsal attention network or the default mode network, depending on the attentional deman

103 ce the understanding of the PMC in posterior default mode network development.

104 ex and posterior cingulate cortex (i.e., the default mode network) disrupts ordering of utilities, pr

105 vestigate the functional organization of the Default Mode Network (DMN) - an important subnetwork wit

106 on network (FPCN/DAN) activity and decreased default mode network (DMN) activity during the CRT compa

107 graphy), we examined connectivity within the default mode network (DMN) and between the DMN and the c

108 rinsic functional connectivity in the dorsal default mode network (DMN) and executive control network

109 cific maturational lag in connections within default mode network (DMN) and in DMN interconnections w

110 The default mode network (DMN) and semantic network (SN) are

111 between large-scale networks, in particular, default mode network (DMN) and task-positive networks (T

112 bserved between the three groups between the default mode network (DMN) and the nodes of the task pos

113 were implanted with CPEs in two nodes of the default mode network (DMN) and two nodes in a lateral co

114 ns between the Salience Network (SN) and the Default Mode Network (DMN) are thought to be important f

115 r regions, and task-related deactivations in default mode network (DMN) areas.

116 h the IPS overlapped with regions within the default mode network (DMN) but the IPS also showed conne

117 e studied functional connectivity within the default mode network (DMN) by means of independent compo

118 GNIFICANCE STATEMENT Activation of the human default mode network (DMN) can be measured with fMRI whe

119 ive control task regions and deactivation of default mode network (DMN) components.

120 hildhood poverty was associated with reduced default mode network (DMN) connectivity.

121 resent study was to research the patterns of Default Mode Network (DMN) deactivation in Obsessive Com

122 We found that (i) pain-induced default mode network (DMN) deactivations were attenuated

123 ts is marked by aberrant connectivity of the default mode network (DMN) during resting state.

124 functional connectivity (FC) of the brain's default mode network (DMN) has been identified within th

125 The default mode network (DMN) has been shown to increase it

126 The default mode network (DMN) has been suggested to support

127 The default mode network (DMN) has been traditionally assume

128 Regions of the default mode network (DMN) have been frequently identifi

129 ies have emphasized the vulnerability of the default mode network (DMN) in Alzheimer's disease (AD),

130 terior cingulate cortex (PCC) regions of the default mode network (DMN) in dogs.

131 at had a spatial distribution similar to the default mode network (DMN) in humans, consistent with ea

132 ty indexed with goodness of fit (GOF) of the default mode network (DMN) in the drug group and decreas

133 umerous neuroimaging studies have implicated default mode network (DMN) involvement in both internall

134 The default mode network (DMN) is a collection of brain regi

135 The default mode network (DMN) is a commonly observed restin

136 The default mode network (DMN) is a complex dynamic network

137 The default mode network (DMN) is characterised by coherent

138 Reduced deactivation within the default mode network (DMN) is common in individuals with

139 The default mode network (DMN) is critical in this study, gi

140 The brain's default mode network (DMN) is highly active during wakef

141 The brain's default mode network (DMN) is highly heritable and is co

142 Functional connectivity in the default mode network (DMN) is known to be reduced in pat

143 Deactivation of the human brain's default mode network (DMN) is regarded as suppression of

144 ation and functional connectivity within the default mode network (DMN) of the brain while participan

145 resting relative to engaging in a task, the default mode network (DMN) pattern.

146 Previous studies have suggested that the default mode network (DMN) plays a central role in the p

147 Does the default mode network (DMN) reconfigure to encode informa

148 he view that the functional integrity of the default mode network (DMN) reflects "level of consciousn

149 We tested the accuracy of default mode network (DMN) resting state functional conn

150 ing that mindfulness meditation may increase default mode network (DMN) resting-state functional conn

151 Default mode network (DMN) to insula connectivity is ass

152 een the functional and structural changes of default mode network (DMN) underlying the cognitive impa

153 The default mode network (DMN), a brain system anchored in t

154 cingulate cortex (CGp) is a major hub of the default mode network (DMN), a set of cortical areas with

155 , including central executive network (CEN), default mode network (DMN), and salience network (SN).

156 referentially decreased in the thalamus, the Default Mode Network (DMN), and the bilateral Frontopari

157 retrial brain activity in key regions of the default mode network (DMN), but not the dorsal attention

158 The default mode network (DMN), comprising an anatomically d

159 The brain's default mode network (DMN), having a high rate of basal

160 connectivity between regions involved in the default mode network (DMN), implicated in divergent thin

161 One system, termed the default mode network (DMN), is thought to support intern

162 sses, most notably increased activity in the default mode network (DMN), suppressed activity within t

163 rontoparietal control network (FPCN) and the default mode network (DMN), two networks that do not str

164 everal resting-state networks, including the default mode network (DMN), which contains a set of cort

165 A unifying function associated with the default mode network (DMN), which is more active during

166 tional connectivity within the resting state default mode network (DMN), which may signal heightened

167 t, we examine the existence of the so-called default mode network (DMN)--a distributed functional bra

168 N) and the medial prefrontal-medial parietal default mode network (DMN)-are consistent findings in de

169 erior parietal lobe, a prominent node of the default mode network (DMN).

170 bolic RSN was topographically similar to the default mode network (DMN).

171 resting-state functional connectivity in the default mode network (DMN).

172 networks include regions that belong to the default mode network (DMN).

173 es, i.e., several of the core regions of the default mode network (DMN).

174 rest, including hyperconnectivity within the default mode network (DMN).

175 he typical balance of connections within the default mode network (DMN; prominent during introspectiv

176 ), and the medial prefrontal-medial parietal default mode networks (DMN).

177 Existing evidence suggests that the default-mode network (DMN) and fronto-pariatal network (

178 tive correlation between fluctuations in the default-mode network (DMN) and task-positive networks, w

179 The default-mode network (DMN) has become a well accepted co

180 erential operations performed by the brain's default-mode network (DMN) has prompted interest in exam

181 pth associated with the FNE was found in the default-mode network (DMN) involved with spontaneous int

182 The default-mode network (DMN) is known to be dysfunctional,

183 yses, increased RSFC was observed within the default-mode network (DMN) post-treatment.

184 Most studies have focused on the default-mode network (DMN), a primary locus of AD pathol

185 volving the so-called salience network (SN), default-mode network (DMN), and frontoparietal task cont

186 ss four major cortical association networks [default-mode network (DMN), salience network (SAL), dors

187 measured in dorsal attention network (DAN), default-mode network (DMN), salience network (SN), and e

188 ct and functionally antagonistic system--the default-mode network (DMN)--which typically deactivates

189 d connectivity between the precuneus and the default-mode network (DMN).

190 al regions that, in humans, are known as the default-mode network (DMN).

191 -task-positive) and decreased or "negative" [default-mode network (DMN)] fMRI responses during task p

192 CBF, and the correlation was stronger in the default-mode network (DMN; including medial frontal-pari

193 tive networks ["external-task positive" and "default-mode network" (DMN)] are responsive to increasin

194 ctivity of four known neural networks (i.e., default mode network, dorsal attention network, salience

195 reative-ideation regions associated with the default mode network (dorsomedial prefrontal cortex, mid

196 l influence from the salience network to the default mode network during moral reasoning.

197 odes of the salience network to nodes of the default mode network during moral reasoning.

198 However, integration of the default mode network emerged as a key feature differenti

199 al ganglia, D) meso/paralimbic, E) posterior default mode network, F) fronto-temporal/paralimbic and

200 findings of this paper are (i) the posterior default mode network fails before measurable amyloid pla

201 In the default mode network, fMRI amplitude was 0.57% (SD 0.26)

202 reduced ability to deactivate components of default mode networks, following the presentation of inf

203 cognitive control, and a segregation of the default mode network from task-related networks.

204 functional connectivity using seeds from the default mode network, frontoparietal network, and cingul

205 sk-based assessment of posterior hippocampal/default-mode network function, were used.

206 Our results of aberrant default mode network functional connectivity and distinc

207 The default mode network has distinct subsystems with unique

208 ired reward responsivity was associated with default mode network hyperconnectivity and diminished co

209 Finally, the default-mode network, identified in a separate resting-s

210 thin two major intrinsic brain networks: the default mode network, implicated in memory encoding, sto

211 problems with deactivation of a task-related default mode network in both disorders.

212 the functionally distinct inferior parietal default mode network in humans compared to monkeys resul

213 kin to that between the social brain and the default mode network in humans: this overlap specificall

214 and associated vasoconstriction, within the default mode network in hypoxia is supported by increase

215 he face patch resting state networks and the default mode network in monkeys show a pattern of overla

216 lt mode network and the possible role of the default mode network in MPH-mediated improvements in ina

217 nd between temporal pole and elements of the default mode network in NTSCUs.

218 e mechanism to explain the engagement of the default mode network in probability learning.

219 ted decreased functional connectivity in the default mode network in the cognitively normal older adu

220 dulate activity in the central executive and default mode networks in healthy individuals.

221 r insula modulation of central executive and default mode networks in patients with schizophrenia.

222 onnectivity in the executive control and the default mode networks in the bilingual, compared with th

223 ed anti-correlation with fronto-parietal and default mode networks in the right hemisphere; and (iii)

224 ter is linked to shifts into a task-inactive default-mode network in between task-relevant stimulus o

225 we show that functional connectivity of the default-mode network in children and adolescents is rela

226 and increased functional connectivity in the default-mode network in depression, no study has concurr

227 ated in autism, the salience network and the default mode network, in autistic subjects and age-, gen

228 tion in a set of brain regions linked to the default mode network, including posterior cingulate cort

229 ions belonged to two important networks: the default-mode network, including precuneus and angular gy

230 a network of areas reminiscent of the human "default-mode network," including posterior cingulate cor

231 Both Abeta and tau pathology affect default mode network integrity before clinical onset of

232 1 were independently associated with reduced default mode network integrity, with the most prominent

233 ivity magnetic resonance imaging measures of default mode network integrity.

234 ted with elevated amygdala-insula and insula-default-mode network interactions.

235 that smokers engage attentional networks and default mode networks involved in self-referential proce

236 The default mode network is affected in Alzheimer disease an

237 In light of recent findings that the default mode network is hypoactive in autism, our data r

238 in externally focused attention whereas the default mode network is involved in internally directed

239 Although the default mode network is recruited when healthy subjects

240 First, as previously reported, the default mode network is recruited when healthy subjects

241 al connectivity of the ventral attention and default mode networks is associated with behavioral inhi

242 r insula modulation of central executive and default mode networks is disrupted in schizophrenia and

243 duced connectivity between the sgACC and the default mode network, left dorsolateral prefrontal corte

244 usters of coactivated areas with an enlarged default mode network-like posterior region.

245 orks and the inability to disengage from the default mode network may be critical in cognitive/affect

246 gher cognitive functions such as the brain's default mode network, may be particularly vulnerable to

247 work, while moxibustion mainly regulated the default mode network of the brain.

248 htened internetwork connectivity between the default mode network, particularly the anterior cingulat

249 inal fasciculus and heritable aspects of the default mode network (phenotypic correlation, rhop = -0.

250 The brain's default mode network plays a central role in this work.

251 t of brain regions collectively known as the default mode network plays a crucial role in such "autop

252 the insula and constituents of the canonical default-mode network (posterior cingulate cortex, ventro

253 alamus, ventral striatum, insula) and in the default-mode network (precuneus, temporoparietal junctio

254 ollows: visual cortex, V3/V3A/V7; within the default mode network, precuneus, and inferior parietal l

255 tment connectivity between the sgACC and the default mode network predicted clinical improvement, as

256 igher FCD in visual and prefrontal cortices, default mode network regions and thalamus, while HD had

257 ere coupled with reduced integration of core default mode network regions in the ventromedial cortex

258 uations in brain activity within several key default mode network regions, as well as within the ante

259 characterized by significant deactivation in default mode network regions, suggesting suppression res

260 tive pattern of amyloid plaque deposition in default mode network regions.

261 ght insular subregions and central executive/default mode network regions.

262 ive that nAChR agonists suppress activity in default-mode network regions and enhance activity in exe

263 The discovery of the default mode network reignited a longstanding interest i

264 uding regions relevant to cognitive control, default mode network related self-referential thought, b

265 ductions in intranetwork connectivity of the default mode network relative to the HC group (p<0.05 co

266 to the dorsal-attention/fronto-parietal and default-mode networks, respectively.

267 well as decreases in BGluM in regions of the default mode network (retrosplenial cortex and cingulate

268 e-scale intrinsic connectivity networks (the default mode network, salience network and central execu

269 left lateral occipital cortex) included the default mode network seed.

270 s well as with parts of the self-referential default-mode network, seemed instrumental in establishin

271 regions, including hubs of the executive and default mode networks, showed a robust nonlinear age-by-

272 While applying learned rules, the default mode network shows both greater activity and con

273 es of the frontoparietal control network and default mode network strengthen their interaction with o

274 Initiative, we characterized the pattern of default mode network subsystem connectivity changes acro

275 w highly central cortical regions key to the default mode network such as the posterior and anterior

276 ected cortical regions that form most of the default mode network, such as the insula, cingulate cort

277 greater task-related deactivation within the default-mode network than comparison subjects.

278 h four canonical resting-state networks: the default mode network, the dorsal attention network, the

279 roparietal focus and a potential link to the default mode network, the other representing the sensori

280 resting-state networks (RSNs), including the default mode network, the somato-motor network, the visu

281 intrinsic brain networks in superaging: the default mode network, typically engaged during memory en

282 ate functional connectivity, focusing on the default mode network, ventral and dorsal salience networ

283 Conversely, integration of the default mode network was increased in the sibling group

284 nt VTA/midbrain connectivity strength to the default mode network was negatively correlated.

285 increased correlation of the insula with the default mode network was related to lower attentional ac

286 The default mode network was used as a control network.

287 prefrontal cortices, major components of the default-mode network, was reduced only in patients whose

288 human networks, such as the language and the default-mode networks, we detected topological correspon

289 hat the functional connectivities of (i) the default mode network were greater; (ii) the salience net

290 terior default mode network, and the frontal default mode network were observed.

291 ife memory test preferentially activated the default mode network, whereas hits in the picture memory

292 ease variants in the dorsal and left ventral default mode network, whereas significant differences we

293 cerebellar Crus I/II modulates the cerebral default mode network, whereas vermal lobule VII stimulat

294 within the visual, medial temporal lobe and default mode networks, whereas during task it was driven

295 f cortical areas which are components of the default-mode network, whereas regulation of feedback inh

296 y with the medial prefrontal-medial parietal default-mode network, whereas the anterior (rodent ventr

297 cate a memory-based "autopilot role" for the default mode network, which may have important implicati

298 The failure begins in the posterior default mode network, which then shifts processing burde

299 coupling between executive networks and the default mode network, which was associated with modafini

300 temporal lobe progressing along the cortical default mode network, with no or minimal involvement of