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1 at the junction of the epididymis and ductus deferens.
2 rve terminals in the guinea-pig isolated vas deferens.
3 tate, coagulating gland, epididymis, and vas deferens.
4 bule and collected by cannulation of the vas deferens.
5 e lungs, pancreas, liver, intestine, and vas deferens.
6 in precapillary arterioles in ureter and vas deferens.
7 inje cell bodies and cells lining the ductus deferens.
8 icle, bulbourethral gland, and caudal ductus deferens.
9 greater in femoral artery as compared to vas deferens.
10 lular CPI-17 concentration of the phasic vas deferens.
11 smission has a physiological role in the was deferens.
12 rescence in the tunica muscularis of the vas deferens.
13 e to congenital bilateral absence of the vas deferens.
14 edominantly to the cell periphery in the vas deferens.
16 rtility due to congenital absence of the vas deferens, 9 patients with nonclassic CF, and 27 unaffect
19 oth muscle preparation of the guinea-pig vas deferens and from isolated bovine adrenal chromaffin cel
20 ompounds described in the isolated mouse vas deferens and guinea pig ileum bioassays revealed that al
21 Evaluation of the ligands in the mouse vas deferens and guinea pig ileum preparations revealed that
23 ermined from functional assays in rabbit vas deferens and inhibition constant (Ki) of 0.02 nM measure
25 ormal P2X1 receptor functionality in the vas deferens and that its absence leads to impaired peristal
26 Fluorogold into the junction between the vas deferens and the cauda labeled a population of neurons i
29 mal escape reflex, mispositioning of the vas deferens and uterus, and mitotic chromosome loss and mul
30 ity toward delta-opioid receptors (mouse vas deferens) and produced analgesia in mice in a naloxone r
31 ype, congenital bilateral absence of the vas deferens, and determined whether mutant CFTR could regul
32 hidism and abnormal tortuosity of the ductus deferens, and female transheterozygotes exhibited abnorm
33 cific structures such as the epididymis, vas deferens, and seminal vesicle from a straight Wolffian d
35 at similar cells are also present in the vas deferens, and that a bafilomycin-sensitive proton flux c
36 rincipal cells of the epididymis and the vas deferens, and that both NHERF1 and CFTR co-immunoprecipi
38 ive alpha1A-ARs from rat tail artery and vas deferens are also desensitized by OXY, but not by NE or
41 e3 was required for bioactivity in mouse vas deferens but not for interaction with delta opioid recep
42 ffian duct and formed the epididymis and vas deferens, but failed to elaborate the efferent ductules,
43 in single cells from the Syrian hamster vas deferens cell line DDT1MF-2 were investigated using the
44 detected in epithelia of the epididymis, vas deferens, coagulating gland, preputial gland and salivar
48 or and sperm counts, but abnormal distal vas deferens convolution resulting in complete and incomplet
50 with electrical field stimulation (EFS; vas deferens), dimethylphenylpiperazinium (chromaffin cells)
51 onium ileus (MI); pancreatic, liver, and vas deferens disease; and a predisposition to lung infection
55 structing cysts of the seminal vesicles, vas deferens, ejaculatory ducts, or prostate in 26 (9.4%) pa
57 le response to electrical stimulation in vas deferens from alpha2B-AR knockout, alpha2C-AR knockout,
58 ) in smooth muscle of the mouse isolated vas deferens has been used to detect the packeted release of
59 lionic sympathetic axon bundles in mouse vas deferens have been characterized using confocal microsco
60 .6%) patients; unilateral absence of the vas deferens in 31 (11.2%) patients; obstructing cysts of th
61 uded congenital bilateral absence of the vas deferens in 94 (34.1%) patients; bilateral occlusion of
65 e CB1 and CB2 receptors and in the mouse vas deferens in vitro assay and the mouse tetrad in vivo ass
67 o 80% of the net proton secretion in the vas deferens is inhibited by bafilomycin, consistent with a
69 devoid of agonist activity in the mouse vas deferens (MVD) and guinea pig ileum (GPI) preparations b
70 unctional activity in the mouse isolated vas deferens (MVD) and guinea pig isolated ileum (GPI) assay
71 in the guinea pig ileum (GPI) and mouse vas deferens (MVD) functional bioassays were determined for
75 the guinea pig ileum (GPI) and the mouse vas deferens (MVD) with EC50 values of 1.82 +/- 0.16 and 2.9
77 h muscle was investigated in bladder and vas deferens of mice carrying a targeted mutation in both al
78 ostganglionic sympathetic neurons in the vas deferens of mice injected with IgG from LEMS patients or
81 n-selective cation channels from the rat vas deferens (P2X1 receptors) were stably expressed in HEK 2
82 d; of these, one was cloned from the rat vas deferens (P2X1) and another from pheochromocytoma (PC12)
83 purines and noradrenaline (NA) from the vas deferens preparation differed from the pattern of overfl
84 f NA and the purines from the guniea-pig vas deferens preparation was examined after treatment with t
87 nduced gene (FR-1) (80% identity), mouse vas deferens protein (MVDP) (76%), and human aldose reductas
88 evident within sympathetic fibers of the vas deferens, reflecting a high degree of spatial organizati
89 thetic nerves innervating the guinea-pig vas deferens releases not only neuronal ATP, but also solubl
91 1%) patients; bilateral occlusion of the vas deferens, seminal vesicles, and ejaculatory ducts by cal
97 e ryanodine receptor (RyR) in aortic and vas deferens smooth muscle was localized using immunofluores
99 versus 33.2% +/- 10.3% (P < 0.0001) for vas deferens sperm and 40.1% +/- 9.6% versus 33.2% +/- 7.5%
100 of the testis and associated structures (vas deferens, testicular vessels) and subsequent treatment o
101 itization than those found in the native vas deferens tissue, in agreement with previous reports.
102 ly reduced contraction of the guinea pig vas deferens to electrical field stimulation (EFS) and norep
103 eptor-deficient mice, contraction of the vas deferens to sympathetic nerve stimulation is reduced by
104 onent of the contractile response of the vas deferens to sympathetic nerve stimulation, which propels
111 rminals and smooth muscle cells in mouse vas deferens were loaded with the Ca2+ indicator Oregon Gree
112 voked contractions of the mouse isolated vas deferens when administered at submicromolar concentratio
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