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1 isceral WAT significantly correlate with the degree of obesity.
2 insulin resistance disproportionate for the degree of obesity.
3 NASH have severe Adipo-IR independent of the degree of obesity.
4 re self-selected and had restrictions on the degree of obesity.
5 rplasia that is out of proportion with their degree of obesity.
6 ht-SDS was significantly decreased given the degree of obesity.
7 adjustment for race or ethnic group and the degree of obesity.
8 nd beta-cell function, older age, and lesser degree of obesity.
9 ance, of similar ages, sex distribution, and degree of obesity.
10 Subjects were matched for age and similar degree of obesity.
11 One distortion is caused by the degree of obesity.
12 ion in obese mice increases with age and the degree of obesity.
13 ted with insulin resistance but not with the degree of obesity.
14 HD risk, and insulin resistance at any given degree of obesity accentuates the risk of CHD and type 2
21 e multivariate model, adjusting for age, the degree of obesity, and other important covariates, the r
23 mice fed a high-fat diet developed a similar degree of obesity as that of wild-type mice, but, simila
26 iving a transplant decreased with increasing degree of obesity, categorized by ranges of BMI (adjuste
27 w that glucose tolerance deteriorates as the degree of obesity increases due to worsening insulin res
28 ose uptake was independently associated with degree of obesity (inversely) and estimates of level of
32 ased ob mRNA expression and to determine the degree of obesity required to stimulate expression of ob
34 als of various ages, ethnic backgrounds, and degrees of obesity were analyzed electrophoretically and
37 be greater individual control over moderate degrees of obesity, whereas low HDL cholesterol may be l
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