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1 + signaling is correlated with the period of delayed neuronal death.
2 to increased postresuscitation mortality and delayed neuronal death.
3 olished the hyperactivity and the consequent delayed neuronal death.
4 potentially important in the pathogenesis of delayed neuronal death.
5 tivation ("excitotoxicity") induces acute or delayed neuronal death.
6 an excitotoxic component of ischemia-induced delayed neuronal death.
7 s been suggested to trigger ischemia-induced delayed neuronal death.
8 us glutamate are likely to contribute to the delayed neuronal death after global ischemia.
9 rotransmitter glutamate was a major cause of delayed neuronal death after head injury, and several ma
10                                              Delayed neuronal death after transient cerebral ischemia
11 ggest that caspase-3 activity contributes to delayed neuronal death after transient ischemia.
12 e a variety of traumatic responses including delayed neuronal death and later cognitive decline.
13 fording protection against the selective and delayed neuronal death associated with cardiac arrest or
14 triking implications for intervention in the delayed neuronal death associated with global ischemia.
15 he data implicate COX-2 in the mechanisms of delayed neuronal death at the infarct border and provide
16  after ischemia/reperfusion (I/R), extensive delayed neuronal death, DNA damage, and increases in rea
17  nanocrystal injection significantly reduced delayed neuronal death, DNA damage, as well as glial cel
18 i.e. when blood flow is restored, and causes delayed neuronal death (DND) in selective vulnerable reg
19 erlying cause for neuronal injury leading to delayed neuronal death (DND).
20 receptors are responsible for the secondary (delayed) neuronal death following neuronal injury, inclu
21                                        Since delayed neuronal death has the characteristics of apopto
22 ponse to endogenous glutamate; the resulting delayed neuronal death in CA1 exhibits many characterist
23 slices from hypoxia, and (b) reduce ischemic delayed neuronal death in hippocampal organotypic slice
24 family members participate in the process of delayed neuronal death in humans, we examined brain tiss
25 perimentally in animals causes selective and delayed neuronal death in pyramidal neurons of the hippo
26 -1790, on acute cortical neuronal injury and delayed neuronal death in the cerebellum after mild trau
27 s epilepticus and whether they contribute to delayed neuronal death in vivo.
28 ansient forebrain or global ischemia induces delayed neuronal death in vulnerable CA1 pyramidal cells
29 lastoma tumor suppressor protein) pathway in delayed neuronal death induced by ischemia.
30                              Hypoxia-induced delayed neuronal death is known to require de novo gene
31 l, our results indicate a role for Cdc25A in delayed neuronal death mediated by ischemia.SIGNIFICANCE
32 perimentally in animals can cause selective, delayed neuronal death of hippocampal CA1 pyramidal neur
33 Transient global ischemia induces selective, delayed neuronal death of pyramidal neurons in the hippo
34 atterns of IEGs to be associated with either delayed neuronal death, or cell survival, depending upon
35 ing hypoxia-dependent transcription, reduces delayed neuronal death that follows hypoxic stress.
36 schemia for even 8-minute duration can cause delayed neuronal death that is more severe in the caudop
37             However, no significant acute or delayed neuronal death was seen in the CN or white matte
38      To ask whether astrocytes contribute to delayed neuronal death, we first administered antagonist

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