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1 re-forming subunit of the rapidly activating delayed rectifier potassium channel.
2 e (HERG) that encodes the rapidly activating delayed rectifier potassium channel.
3 ith KVLQT1 to form the slow component of the delayed rectifier potassium channel.
4 side chains which influence binding to brain delayed rectifier potassium channels.
6 rteriosus involves the coordinated action of delayed rectifier potassium channels and voltage-gated c
7 currents (primarily through rapid and slowed delayed rectifier potassium channels) and that block of
10 d neurotransmitter/neuromodulator receptors, delayed-rectifier potassium channels, calcium-dependent
11 a-go-go-related gene (HERG), which encodes a delayed-rectifier potassium channel, cause the disease i
12 al features of inactivation of the rat Kv2.1 delayed rectifier potassium channel, expressed in Xenopu
14 d gene (hERG) encodes the rapidly activating delayed rectifier potassium channel (I(Kr)) which plays
15 re-forming subunit of the rapidly activating delayed rectifier potassium channel (IKr), which is impo
19 tions in the gene encoding the voltage-gated delayed rectifier potassium channel Kv1.1 as underlying
23 achea correae, inhibits Kv1.3, a Shaker-type delayed rectifier potassium channel present in human T l
25 assium 2.1 (Kv2.1) channels are the dominant delayed rectifier potassium channels responsible for act
26 The Kv2.1 gene encodes a highly conserved delayed rectifier potassium channel that is widely expre
28 ongation of the action potential by block of delayed rectifier potassium channels would be expected t
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