ライフサイエンスコーパス: delta power

1 lial protein, was increased in parallel with delta power.

2 TNFR KO mice showed higher baseline SWS delta power.

3 cortex changes electroencephalographic (EEG) delta power.

4 nd that those reductions reduce cortical EEG delta power.

5 leads showed increasing alpha (8-12 Hz) and delta power (0-4 Hz) and in the occipital leads delta po

6 an even greater postdeprivation reduction in delta power (60-75%) and a concomitant increase in wakef

7 oss bilaterally) caused a 60-70% decrease in delta power and a 50-60% decrease in nonrapid-eye-moveme

8 tween conditions, was predicted by increased delta power and decreased sigma power in RS compared wit

9 nd theta synchrony were reduced in patients; delta power and synchrony better distinguished between g

10 manual restraint can increase sleep and EEG delta power and that increases in sleep may persist acro

11 onist, reduces cortical electroencephalogram delta power and transiently inhibits spontaneous seizure

12 Lesions resulted in increases in slow-wave (delta) power and decreases in high-frequency (beta 2) po

13 ally significant improvements on deep sleep (delta power) and sleep consolidation at doses as low as

14 ortical ACh release, behavioral arousal, EEG delta power, and sleep.

15 ntensity [i.e., nonrapid eye movement (NREM) delta power] and increased rapid eye movement sleep time

16 of nNOS, they are unable to up-regulate NREM delta power appropriately.

17 The increase of NREMS delta power as a function of previous wake duration vari

18 at selective REM sleep deprivation increased delta power but decreased theta power during the residua

19 ly compensated for the SD-induced deficit in delta power, but the Per3(4/4) and wild-type mice did no

20 increased baseline NREM sleep by 4% and NREM delta power by 15%, and decreased REM sleep by 10%.

21 sitions between responsive states, while the delta power/connectivity changes were consistent with th

22 Delta power density (PD) was unchanged between age 9 and

23 Delta power during NREM sleep was increased in both grou

24 REM) sleep time, NREM bout duration, and EEG delta power during NREM sleep, an index of preexisting h

25 Mice receiving conditioning stimuli had more delta power during NREM sleep, whereas mice receiving fe

26 h voltage NREM sleep, sleep bout length, and delta power during NREM sleep.

27 M1, mNE and mFS significantly increased EEG delta power during NREM, but M2-3, NE and FS alone did n

28 e thus propose that slow waves, reflected in delta power during RS, act to restore brain function, th

29 phy (EEG) theta power during wakefulness and delta power during sleep, were greater in the Per3(5/5)

30 ollowed by increased sleep and increased EEG delta power during sleep.

31 s in increased electroencephalographic (EEG) delta power during subsequent non-rapid eye movement sle

32 frequency power in the electroencephalogram (delta power) during non-rapid eye movement sleep reflect

33 vely in TDW rather than all waking, predicts delta power dynamics both in Hcrt(ko/ko) and WT mouse ba

34 causes AS-like increases in neocortical EEG delta power, enhances seizure susceptibility, and leads

35 m (EEG) correlate of sleep pressure, non-REM delta power, failed to increase.

36 ta power (0-4 Hz) and in the occipital leads delta power greater than alpha power.

37 rapid eye movement sleep and a reduction of delta power in non-rapid eye movement sleep.

38 volunteers; (v) a decrease in the sleep EEG delta power in patients.

39 Effective forms of ECT resulted in increased delta power in prefrontal regions, and this change was a

40 , shortened sleep latency, and increased EEG delta power in rats.

41 TDW maintenance in baseline wake and blunted delta power in SWS, reproducing, respectively, narcoleps

42 ly 43% during the dark period, and increased delta power in the EEG during NREM sleep by approximatel

43 similar extent (>99%), and, as expected, the delta power increase during recovery sleep was quantitat

44 Power spectral analysis showed that delta power increased from responsiveness to unresponsiv

45 ep depth (lower nonrapid eye movement [NREM] delta power), increased NREM-to-REM transitions, hindere

46 peated sessions whereas the magnitude of EEG delta power may vary across sessions.

47 7; p < 0.001) and total electroencephalogram delta power (r = 0.79; p < 0.001) but not to rapid-eye-m

48 rmates, spontaneous waking fails to induce a delta power reflecting prior waking duration.

49 nimals failed to exhibit a compensatory NREM delta power response during the 4-h sleep opportunities

50 This divergent delta power response is consistent with the known cortic

51 heep did not show the increase in NREM sleep delta power seen in unaffected sheep.

52 mals showed a massive increase in NREM sleep Delta power, similarly to that occurring in natural torp

53 hM4Di receptors significantly increased EEG delta power spectrum and slightly decreased wakefulness.

54 ignificantly and lastingly decreased the EEG delta power spectrum, produced low-delta non-rapid eye m

55 nt as WT mice, with similar increases in EEG delta power, suggesting that their homeostatic control o

56 nt to SD was proportional to the increase in delta power that occurs in inbred strains: the strain th

57 Although both drugs affected delta power, these changes did not correlate with increa

58 tatic sleep drive into up-regulation of NREM delta power through an NO-dependent mechanism.

59 t paired mice exhibited significantly higher delta power throughout the dark period.

60 REM sleep frontal high delta power was a negative correlate of intelligence.

61 dose of mirtazapine (p = 0.42), but NREM EEG delta power was increased by more than 30% at all doses

62 le in modulating arousal states and NREM EEG delta power, which is widely recognized as a marker for