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1 nce accuracy was significantly reduced after Delta9-tetrahydrocannabinol.
4 f nabiximols (maximum daily dose, 86.4 mg of Delta9-tetrahydrocannabinol and 80 mg of cannabidiol) or
6 h as the nucleus accumbens after exposure to Delta9-tetrahydrocannabinol, but less is known about can
7 ociated with users' urinary 11-nor-9-carboxy-Delta9-tetrahydrocannabinol concentrations at study entr
8 odies with high affinity and specificity for delta9-tetrahydrocannabinol could be valuable immunophar
11 onic treatment of SIV-infected macaques with Delta9-tetrahydrocannabinol (Delta9-THC) increased survi
12 infected macaques, chronic administration of Delta9-tetrahydrocannabinol (Delta9-THC) inhibited viral
15 used to examine the effects of estrogen and delta9-tetrahydrocannabinol (delta9-THC) on learning and
16 Mice were treated with escalating doses of Delta9-tetrahydrocannabinol (Delta9-THC) or R+-[2,3-dihy
17 primary psychoactive ingredient in cannabis, Delta9-tetrahydrocannabinol (Delta9-THC), affects the br
18 e major psychoactive component of marijuana, Delta9-tetrahydrocannabinol (Delta9-THC), are presented.
21 The main active chemical in marijuana is delta9-tetrahydrocannabinol (delta9-THC); hence, monoclo
22 centres with oral cannabis extract (n=211), Delta9-tetrahydrocannabinol (Delta9-THC; n=206), or plac
25 principal bioactive component of marijuana, delta9-tetrahydrocannabinol, have been used for thousand
27 perimentally induced higher cannabinoid [(-)-Delta9-tetrahydrocannabinol] levels constrain preimplant
28 (CB) receptor agonism using R(+)-WIN 55,212, delta9-tetrahydrocannabinol, methanandamide and JWH-133
29 gn, investigating effects of the eCB agonist Delta9-tetrahydrocannabinol on WM function in 17 healthy
30 Both cannabinoid agonists WIN 55,212-2 and delta9-tetrahydrocannabinol shortened the modal response
34 ylamphetamine (MDEA), morphine, cocaine, and Delta9-tetrahydrocannabinol (THC) from a single blood sp
37 ating effects of the endocannabinoid agonist Delta9-tetrahydrocannabinol (THC) on executive function
38 examined the effects of repeated exposure to Delta9-tetrahydrocannabinol (THC) on performance of spat
39 ation of cannabinoid CB1 receptors (CB1R) by delta9-tetrahydrocannabinol (THC) produces a variety of
40 n of the psychoactive component of cannabis, delta9-tetrahydrocannabinol (THC), activated cerebellar
41 ), as well as exogenous cannabinoids such as Delta9-tetrahydrocannabinol (THC), are mediated by two s
42 ometry (UHPLC-MS/MS) for analysis of urinary Delta9-tetrahydrocannabinol (THC), cannabidiol and canna
43 after chronic exposure to vehicle solution, Delta9-tetrahydrocannabinol (THC), or the cannabinoid ag
48 of benzoylecgonine (BE) and 11-nor-9-carboxy-Delta9-tetrahydrocannabinol (THC-COOH) were shown to be
49 vation are mimicked by administration of (-)-Delta9-tetrahydrocannabinol (THC; the major psychoactive
50 binoid receptors also mediate the effects of Delta9-tetrahydrocannabinol, the primary psychoactive in
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