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1 edominantly expressed in ciliated cells, and deltaF508 CFTR pathogenesis in native tissues, like hete
2 log enhanced the activities of wild-type and deltaF508-CFTR channels both in excised membrane patches
3 describe potent activators of wild-type and deltaF508-CFTR channels that are structurally related to
4 ietary compound recently reported to augment deltaF508-CFTR function in mice by an unknown mechanism.
6 ggest that cell-type specific differences in deltaF508 CFTR processing are likely to complicate effor
10 the differences in rescue and activation of deltaF508 CFTR in the two cell lines suggest that cell-t
11 normal airways and 2) the metabolic fate of deltaF508 CFTR and associated ERM proteins in the cystic
12 functional assays confirmed the presence of deltaF508 CFTR at the cell surface in both cell lines af
13 compound increased the open probabilities of deltaF508-CFTR channels in excised membrane patches by 1
16 essing and function of wild-type and rescued deltaF508 CFTR at the cell surface under non-polarized a
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