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1 sorders such as Parkinson's disease and DLB (dementia with Lewy bodies).
2 imer's disease, frontotemporal dementia, and dementia with Lewy bodies).
3 kinsonism, including Parkinson's disease and Dementia with Lewy body.
4 synucleinopathies Parkinson disease (PD) and dementia with Lewy bodies.
5 diseases, including Parkinson's disease and dementia with Lewy bodies.
6 connectivity was not found to be greater in dementia with Lewy bodies.
7 ent evidence links point mutants of betaS to dementia with Lewy bodies.
8 predict treatment response in patients with dementia with Lewy bodies.
9 s disease-related pathology in patients with dementia with Lewy bodies.
10 ms and visual-perceptual dysfunction seen in dementia with Lewy bodies.
11 ylcholinesterase inhibitors in patients with dementia with Lewy bodies.
12 minergic medication and not in patients with dementia with Lewy bodies.
13 ed in blocks from the striatum of cases with dementia with Lewy bodies.
14 ors are commonly used to treat patients with dementia with Lewy bodies.
15 the synucleinopathies Parkinson disease and dementia with Lewy bodies.
16 inson's disease, multiple system atrophy and dementia with Lewy bodies.
17 ing frontotemporal dementia and 14 as having dementia with Lewy bodies.
18 se familial forms of Parkinson's disease and dementia with Lewy bodies.
19 l characteristics of Parkinson's disease and dementia with Lewy bodies.
20 yloid plaques, in Alzheimer's disease and in dementia with Lewy Bodies.
21 opy data were consistent with recent data in dementia with Lewy bodies.
22 thological hallmark of Parkinson disease and dementia with Lewy bodies.
23 ion and loss in Parkinson's disease (PD) and dementia with Lewy bodies.
24 the structure and function of the synapse in dementia with Lewy bodies.
25 and imaging of nigrostriatal dysfunction in dementia with Lewy bodies.
26 al substrates of key psychiatric symptoms in dementia with Lewy bodies.
27 n dementing illnesses, Alzheimer disease and dementia with Lewy bodies.
28 s of psychiatric morbidity for patients with dementia with Lewy bodies.
29 e all significantly higher for patients with dementia with Lewy bodies.
30 l characteristics of Parkinson's disease and dementia with Lewy bodies.
31 ogy in human patients with autopsy-confirmed dementia with Lewy bodies.
32 ression of atrophy in patients with probable dementia with Lewy bodies.
33 disease at autopsy in patients with probable dementia with Lewy bodies.
34 ases, including Parkinson's disease (PD) and dementia with Lewy bodies.
35 decline over time in patients with probable dementia with Lewy bodies.
36 phosphorylated alpha-synuclein pathology in dementia with Lewy bodies.
37 pathies such as Parkinson's disease (PD) and dementia with Lewy bodies.
38 ve diseases, such as Parkinson's disease and dementia with Lewy bodies.
39 rkinson disease, multiple-system atrophy, or dementia with Lewy bodies.
40 lusions are found in Parkinson's disease and dementia with Lewy bodies.
42 cts aged 60 years and over: 15 subjects with dementia with Lewy bodies, 16 subjects with Alzheimer's
43 ensus Alzheimer's disease (17%), followed by dementia with Lewy bodies (29%) and were most common in
44 92 with Alzheimer's disease) and 80 (40 with dementia with Lewy bodies: 40 with Alzheimer's disease)
45 09 [67%] of Parkinson disease, 81 [17.6%] of dementia with Lewy bodies, 55 [11.9%] of Parkinson disea
46 s with Alzheimer's disease, 28 patients with dementia with Lewy bodies, 55 patients with frontotempor
47 94%), Parkinson's disease dementia (94%) and dementia with Lewy bodies (87%) to the other cluster.
48 ps--a clinical case register cohort (98 with dementia with Lewy bodies; 92 with Alzheimer's disease)
51 uropean ancestry who had been diagnosed with dementia with Lewy bodies according to established clini
52 dates the diagnosis of Parkinson disease and dementia with Lewy bodies after 4.67 +/- 2.61 years of f
54 as having Alzheimer's disease, 15 as having dementia with Lewy bodies and 13 as having frontotempora
57 sentation was a better discriminator between dementia with Lewy bodies and Alzheimer's disease than o
58 mentia with Lewy bodies, seven patients with dementia with Lewy bodies and Alzheimer's disease, 12 pa
60 ted which have helped to better characterize dementia with Lewy bodies and discriminate it from Alzhe
61 of PSEN1 mutations have been associated with dementia with Lewy bodies and familial Alzheimer's disea
62 ficantly increased in tissue from cases with dementia with Lewy bodies and familial Alzheimer's disea
64 fy the frequency of psychiatric morbidity in dementia with Lewy bodies and how it differs from probab
66 r monoamine transporter type 2 expression in dementia with Lewy bodies and in dementia with Lewy bodi
68 nucleinopathies such as Parkinson's disease, dementia with Lewy bodies and multiple system atrophy ar
70 al other neurodegenerative diseases, such as dementia with Lewy bodies and multiple system atrophy.
71 in subsets of alpha-synuclein inclusions in dementia with Lewy bodies and multiple system atrophy.
72 lie the pathogenesis of Parkinson's disease, dementia with Lewy bodies and multiple system atrophy.
74 ved to exert toxic effects at the synapse in dementia with Lewy bodies and other alpha-synucleinopath
75 d the high homogeneity between patients with dementia with Lewy bodies and Parkinson disease shows th
76 vascular dementia, frontotemporal dementia, dementia with Lewy bodies and Parkinson's disease dement
81 awing distinction among Alzheimer's disease, dementia with Lewy bodies and PD-related cognitive impai
82 visual hallucinations, but patients who had dementia with Lewy bodies and severe tangle pathology ha
83 ere made by using the consensus criteria for dementia with Lewy bodies and the Mirra et al. protocol
84 ng the McKeith et al. consensus criteria for dementia with Lewy bodies and the National Institute of
85 mprove differentiation between patients with dementia with Lewy bodies and those with Alzheimer's dis
86 icantly increased in brains from humans with Dementia with Lewy bodies and transgenic mice overexpres
87 ibed are often not observed, particularly in dementia with Lewy bodies and when synucleinopathy occur
88 connectivity with the posterior cingulate in dementia with Lewy bodies and with the hippocampus in Al
89 ognitive impairment (MCI), dementia (usually dementia with Lewy bodies), and Parkinson disease (PD).
90 inclusions are present in Parkinson disease, dementia with Lewy bodies, and 30% of Alzheimer disease
91 ssociation analyses in all participants with dementia with Lewy bodies, and also only in participants
93 arkinson's disease, atypical parkinsonism or dementia with Lewy bodies, and identifies patient groups
95 leinopathies, including Parkinson's disease, dementia with Lewy bodies, and multiple system atrophy,
96 ative disorders such as Parkinson's disease, dementia with Lewy bodies, and multiple system atrophy.
97 disease, Parkinson's disease with dementia, dementia with Lewy bodies, and multiple system atrophy;
98 rative diseases such as Parkinson's disease, dementia with Lewy bodies, and multiple systemic atrophy
99 ucleinopathies, such as Parkinson's disease, dementia with Lewy bodies, and multiple systems atrophy.
100 ristic of diseases like Parkinson's disease, dementia with Lewy bodies, and multiple-system atrophy.
101 med Lewy bodies-include Parkinson's disease, dementia with Lewy bodies, and multiple-system atrophy.
102 h multiple system atrophy with parkinsonism, dementia with Lewy bodies, and Parkinson disease dementi
103 ther disorders, including Alzheimer disease, dementia with Lewy bodies, and parkinsonian disorders.
104 diagnosis in over 25% (Alzheimer's disease, dementia with Lewy bodies, and progressive supranuclear
106 leinopathies, including Parkinson's disease, dementia with Lewy bodies, and some forms of Alzheimer's
107 f neuropsychiatric symptoms in patients with dementia with Lewy bodies, and the challenge of accurate
108 isease and dementia with Lewy bodies or pure dementia with Lewy bodies, and these alpha-syn aggregate
111 d Lewy neurites from Parkinson's disease and dementia with Lewy bodies are stained strongly by antibo
112 al diseases, including Parkinson disease and dementia with Lewy bodies, are characterized by the accu
113 tauopathies, and multiple system atrophy and dementia with Lewy bodies as alpha-synucleopathies are d
114 ne elevate the risk of Parkinson disease and dementia with Lewy bodies; both disorders are characteri
115 ysfunction occurs in Parkinson's disease and dementia with Lewy bodies but is not found in progressiv
116 cately related to synucleinopathies, such as dementia with Lewy bodies, but synuclein changes may not
117 ynaptic pathology in Parkinson's disease and dementia with Lewy bodies by disrupting synaptic vesicle
118 ein is found at the presynaptic terminals of dementia with Lewy bodies cases mainly in the form of sm
119 ed alpha-synuclein is present exclusively in dementia with Lewy bodies cases, where it can be identif
120 lo of Lewy bodies in Parkinson's disease and dementia with Lewy bodies, colocalizing with alpha-synuc
121 the known relative preservation of memory in dementia with Lewy bodies compared with Alzheimer's dise
122 ypothesized to be differentially affected in dementia with Lewy bodies compared with Alzheimer's dise
123 er, alternative underlying causes, including dementia with Lewy bodies, corticobasal degeneration, an
124 heimer's disease and the remaining patients, dementia with Lewy bodies, Creutzfeldt-Jakob disease, va
126 on in Parkinson's disease dementia (PDD) and dementia with Lewy bodies (DLB) affect cortical and subc
127 system atrophy-Parkinsonian type (MSA-P) and dementia with Lewy bodies (DLB) also were examined.
128 Seeking antemortem markers to distinguish Dementia with Lewy bodies (DLB) and Alzheimer's disease
129 athologies is found in most individuals with dementia with Lewy bodies (DLB) and in more than 50% of
130 tive diseases including Parkinson's disease, dementia with Lewy bodies (DLB) and multiple system atro
131 the development of Parkinson's disease (PD), dementia with Lewy bodies (DLB) and multiple system atro
132 esults in synucleinopathies that include PD, dementia with Lewy bodies (DLB) and multiple system atro
133 tance: The causes of cognitive impairment in dementia with Lewy bodies (DLB) and Parkinson disease (P
135 ated with the neurodegenerative processes in dementia with Lewy bodies (DLB) and Parkinson's disease
136 e such as familial Alzheimer's disease (AD), dementia with Lewy bodies (DLB) and Parkinson's disease.
137 al correlates of MTA in Alzheimer's disease, dementia with Lewy bodies (DLB) and vascular cognitive i
138 hallucinations experienced by subjects with Dementia with Lewy bodies (DLB) are associated with redu
143 transporter imaging was analyzed, including dementia with Lewy Bodies (DLB) as an additional control
144 sections from the brains of 18 patients with dementia with Lewy bodies (DLB) but lacking AD changes,
145 mes (PS), from essential tremor and probable dementia with Lewy bodies (DLB) from Alzheimer disease.
146 I]FP-CIT SPECT is helpful in differentiating dementia with Lewy bodies (DLB) from Alzheimer's disease
147 ase (AD), frontotemporal dementia (FTD), and dementia with Lewy bodies (DLB) from normal aging and fr
148 on disease (PD), PD with dementia (PDD), and dementia with Lewy bodies (DLB) has long been debated.
149 however the systemic inflammatory profile in dementia with Lewy bodies (DLB) has never before been in
153 differences between Alzheimer's disease and dementia with Lewy bodies (DLB) in resting occipital act
162 f LRRK2 in brain tissue from sporadic PD and dementia with Lewy bodies (DLB) subjects was also examin
163 ynaptic terminal density in 20 patients with dementia with Lewy bodies (DLB), 25 with Alzheimer's dis
165 without dementia (PDD and PD, respectively), dementia with Lewy bodies (DLB), and Alzheimer dementia
166 45) of patients with PD, in 73% (32/44) with dementia with Lewy bodies (DLB), and in only 7% (18/255)
167 of diseases, such as Parkinson disease (PD), dementia with Lewy bodies (DLB), and multiple system atr
168 rative diseases such as Parkinson's disease, dementia with Lewy bodies (DLB), and multiple systemic a
169 ological diagnoses into Alzheimer's disease, dementia with Lewy bodies (DLB), mixed Alzheimer's disea
170 arly stages of Alzheimer's disease (AD) from Dementia with Lewy Bodies (DLB), on peptide level the hi
171 l and neuropathological similarities between dementia with Lewy bodies (DLB), Parkinson's and Alzheim
184 r's disease patients (n = 28), patients with dementia with Lewy bodies (DLB, n = 17) and control subj
185 n = 239), Parkinson's disease (PD, n = 39), dementia with Lewy bodies (DLB, n = 58), other neurodege
186 ects with Alzheimer disease (AD; n = 38) and dementia with Lewy bodies (DLB; n = 30) and controls (n
188 human brain samples from five patients with dementia with Lewy bodies, five patients with Alzheimer'
189 ) and specificity (100%) for differentiating dementia with Lewy bodies from non-Lewy body dementia th
191 e who phenoconverted to Parkinson disease or dementia with Lewy bodies had decreased olfaction, a les
194 ional definitions of Alzheimer's disease and dementia with Lewy bodies have been developed, Parkinson
195 More than half of patients with probable dementia with Lewy bodies have high amyloid-beta deposit
196 eferent participants, followed by those with dementia with Lewy bodies (hazard ratio, 3.94; 95% CI, 2
197 In conclusion, in patients with probable dementia with Lewy bodies, higher amyloid-beta depositio
198 leinopathies, such as Parkinson's disease or dementia with Lewy bodies; Huntington's disease; and amy
199 synucleinopathy (Parkinson's disease in 11, dementia with Lewy bodies in 13, and multiple system atr
200 thors studied 112 autopsy-confirmed cases of dementia with Lewy bodies in patients who had had annual
202 stinguishing between Alzheimer's disease and dementia with Lewy bodies is a major diagnostic challeng
206 y formation, such as Parkinson's disease and dementia with Lewy bodies, is associated with alpha-synu
207 insonism or a clinical diagnosis of possible dementia with Lewy bodies; it is not helpful in differen
208 ative diseases, such as Parkinson's disease, dementia with Lewy body, Lewy body variant of Alzheimer'
211 ontotemporal dementias, Parkinson's disease, dementia with Lewy bodies, multiple system atrophy, as w
212 pranuclear palsy, corticobasal degeneration, dementia with Lewy bodies, multiple system atrophy, cere
213 follow-up, 25 of 74 subjects (34%) developed dementia with Lewy bodies (n = 13), Parkinson disease (n
214 cohort of clinically diagnosed patients with dementia with Lewy bodies (n = 20), who were consecutive
215 on consecutive treatment-naive patients with dementia with Lewy bodies (n = 54) from the Mayo Clinic
216 topsy-confirmed Alzheimer's disease (n = 4), dementia with Lewy bodies (n = 7) and Parkinson's diseas
218 heimer's disease, frontotemporal dementia or dementia with Lewy bodies on the basis of the combined 1
219 ificantly less connectivity in subjects with dementia with Lewy bodies or Alzheimer's disease compare
220 l clinical diagnosis of probable or possible dementia with Lewy bodies or non-Lewy body dementia, mea
221 ith a combination of Alzheimer's disease and dementia with Lewy bodies or pure dementia with Lewy bod
223 rontotemporal dementia, Alzheimer's disease, dementia with Lewy bodies or vascular dementia at presen
224 omic failure will develop Parkinson disease, dementia with Lewy bodies, or multiple system atrophy.
225 ipants with diagnoses of Lewy body dementia, dementia with Lewy bodies, or Parkinson's disease dement
226 ield, partly because of similarities between dementia with Lewy bodies, Parkinson's disease, and Alzh
227 he pathogenesis of synucleinopathy including dementia with Lewy bodies, Parkinson's disease, and mult
230 ue from five of each Parkinson's disease and dementia with Lewy bodies patients by performing biophys
231 a PET pattern suggestive of PD with dementia/dementia with Lewy bodies (PDD/DLB) exhibited a trend to
232 pression in dementia with Lewy bodies and in dementia with Lewy bodies plus Alzheimer's disease was r
234 ever, a mutation of beta-synuclein linked to dementia with Lewy bodies rendered the protein neurotoxi
235 eimer's disease, frontotemporal dementia and dementia with Lewy bodies, REST is lost from the nucleus
236 in postmortem striata from 19 patients with dementia with Lewy bodies, seven patients with dementia
237 ains from patients with Parkinson disease or dementia with Lewy bodies show aggregation of alpha-synu
239 he Parkinson disease-related synucleinopathy dementia with Lewy bodies showed less phosphorylation at
241 ease, Parkinson's disease with dementia, and dementia with Lewy bodies, suggesting common mechanisms
242 features were significantly more frequent in dementia with Lewy bodies than in Alzheimer's disease, a
243 er burden, are also reported to be higher in dementia with Lewy bodies than in Alzheimer's disease.
244 au but not SNCA in PSP, and abnormal SNCA in dementia with Lewy bodies that was quantitatively simila
245 signature inclusions of Parkinson's disease, dementia with Lewy bodies, the Lewy body variant of Alzh
246 s, Lewy neurites and neuraxonal spheroids in dementia with Lewy bodies, the Lewy body variant of Alzh
249 nosis) and 4454 controls (1216 patients with dementia with Lewy bodies vs 3791 controls in the discov
251 ms present in Lewy bodies from patients with dementia with Lewy bodies was carried out using two-dime
252 To better understand the genetic basis of dementia with Lewy bodies, we have done a genome-wide as
253 of progressive supranuclear palsy (PSP) and dementia with Lewy bodies; we observed increased abnorma
255 unctional change in primary visual cortex in dementia with Lewy bodies, which is hypothesized to acco
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