ライフサイエンスコーパス: demineralization

1 promote a pathogenic environment, leading to demineralization.

2 ncy and that calcium intake may prevent bone demineralization.

3 s model in describing the kinetics of enamel demineralization.

4 enamel mineral may modify the rate of enamel demineralization.

5 ing the need for increased attention to bone demineralization.

6 of an acid-resistant layer was observed with demineralization.

7 dentin caries process at the early phases of demineralization.

8 le extant bone extractions, with and without demineralization.

9 rachitic changes and a five-point scale for demineralization.

10 lendronate, but the substance was removed by demineralization.

11 rtical and medullary tissues remaining after demineralization.

12 ity of adhesive displacement" for biological demineralization.

13 ibitors (PIs), has been associated with bone demineralization.

14 ones and its implementation can lead to bone demineralization.

15 dogenous lead exposure due to increased bone demineralization.

16 , the use of fluoride toothpaste, and enamel demineralization.

17 emia included marked overgrowth (5), diffuse demineralization (1), angular deformity (1) and length d

18 eptapeptide library increased the rate of Fe demineralization 3-fold (p<0.001), similarly to a mutati

19 opposite effect and decreased the rate of Fe demineralization 60% (p<0.001).

20 It would appear that over time, demineralization allows compensation of the acidosis.

21 and spectroscopic analyses indicated dentin demineralization and adhesive penetration throughout the

22 formation, favoring resorption, resulting in demineralization and leading to osteoporosis.

23 Increased dentin demineralization and loss of adhesive integrity were not

24 tment at their attachment site in which bone demineralization and matrix degradation occur.

25 ce between pathological factors that lead to demineralization and protective factors that lead to rem

26 The dynamic balance between demineralization and remineralization determines the end

27 ntegument that protects teeth against enamel demineralization, and abrasion.

28 o that present in the original bone prior to demineralization, and the re-calcified bone is palpably

29 White spot lesions (WSL) due to enamel demineralization are major complications for orthodontic

30 D deficiency, rachitic changes and definite demineralization are uncommon and fracture risk is low.

31 significantly contribute to the progress of demineralization around the margins, while fluoride rele

32 n solution for 192 h resulted in significant demineralization at noninfiltrated histologic points but

33 so common and is associated with more severe demineralization at the lumbar spine and hip.

34 ide protection of the underlying dentin from demineralization because of cracks and macroscopic voids

35 radiography showed typical enamel subsurface demineralization before cyclic demineralization/reminera

36 protein extraction method that does not use demineralization but instead uses a methodology from hyd

37 content of calcium (Ca) after 15 seconds of demineralization, but the Ca content increased after 180

38 nce of F-containing toothpaste in enamel re-/demineralization by varying the frequency of carbohydrat

39 eins from the extracellular matrix; however, demineralization can be a slow process that restricts su

40 Since demineralization can be quantified at early stages, befo

41 Previous studies have demonstrated that bone demineralization can improve consolidation in bone graft

42 ndicated a decrease in the driving force for demineralization compared with that seen with the contro

43 The other half received no demineralization (control group).

44 Bone demineralization deserves to be studied in periodontal a

45 Demineralization dissolved these tiles and revealed smal

46 ized gingival recessions, and 2) citric acid demineralization does not affect the clinical outcome of

47 There is no radiographic evidence of demineralization during the early phase of syndrome, as

48 Since demineralization exposes collagen to which fibronectin b

49 alidity of using scattering power to measure demineralization has been confirmed by a three-dimension

50 Root surface demineralization has been shown to promote the establish

51 er-modified surface may be more resistant to demineralization; hence, many investigators are proposin

52 stallized surface layer showed resistance to demineralization; however, the layer did not provide pro

53 osteoblast cells than for the other times of demineralization in all periods of cell culture (P < 0.0

54 ss of the laser-treated surface at resisting demineralization in an acid-gel solution.

55 d propionic acid solutions, whereas rates of demineralization in lactic acid were greater.

56 uate the effect, if any, of citric acid root demineralization in the outcome of subepithelial connect

57 es at both readings in two patients (5%) and demineralization in two patients (5%).

58 e concluded that copper might inhibit enamel demineralization in vitro.

59 ignificant differences in the rate of enamel demineralization in vivo.

60 th recent observations of dissolution (i.e., demineralization) in biological minerals.

61 The depth of demineralization induced by biological or chemical demin

62 ily to increased calcium excretion from bone demineralization induced by space flight.

63 Although enamel demineralization is important for our understanding of c

64 rachitic changes (kappa = 0.33) and 70% for demineralization (kappa = 0.37).

65 The new demineralization method minimized organic matter losses

66 g) recovery using existing techniques vs new demineralization method: 58% vs 78%; N(org) recovery: 60

67 ralization induced by biological or chemical demineralization models was measured using confocal lase

68 not uncommon in CKD and is linked with bone demineralization, muscle catabolism, and higher risks of

69 ergy dispersive spectrometry (SEM/EDS) after demineralization (n = 3).

70 e enamel group without composite had further demineralization of -26.1 +/- 16.2%.

71 ced osteoclast differentiation and function (demineralization of calcium surface).

72 c acid is efficacious and distinctive in the demineralization of dentinal root surfaces for periodont

73 s investigation was to compare the extent of demineralization of enamel slabs in situ, with a sugar-b

74 simultaneously for oil-water separation and demineralization of organic pollutants from the separate

75 We present a novel technique for demineralization of soil samples with HF and dilute HCl

76 unity of the oral cavity, protecting against demineralization of teeth (i.e. dental caries), a highly

77 levels, hyperplasia of the parathyroid, and demineralization of the bones.

78 Demineralization of the contacting surfaces between auto

79 These data indicate that demineralization of the dentin surface promotes prolifer

80 Tooth sensitivity and demineralization of the enamel are, however, common side

81 This study investigated the effect of acid demineralization of the graft-bed interface on graft con

82 l studies have demonstrated that citric acid demineralization of the root surface promotes tissue att

83 ates degradation of the interfacial bond and demineralization of the tooth by recruiting the pioneer

84 y and prevalent disease characterized by the demineralization of the tooth's enamel.

85 establishment of a cariogenic microflora and demineralization of the tooth.

86 C) technique to investigate the acid-induced demineralization of these tissues at a relative undersat

87 s in acidification of the plaque biofilm and demineralization of tooth enamel, marking the onset of d

88 For chemical demineralization, only "intensity of adhesive displaceme

89 about bone proteins that may be lost during demineralization or with the use of denaturing agents.

90 increased remineralization and inhibition of demineralization over a five-day period.

91 ficant in relation to all the other times of demineralization (P < 0.05).

92 he Ca content increased after 180 seconds of demineralization (P < 0.05).

93 eased significantly only after 30 seconds of demineralization (P < 0.5).

94 Following fixation and demineralization, part of the blocks were processed to o

95 ed-measures analysis of variance showed that demineralization period and adhesive type and their inte

96 The demineralization process effectively removed residual al

97 isphosphonate use and to examine whether the demineralization process removes alendronate from allogr

98 o decrease biofilm accumulation, inhibit the demineralization process, to be used for remineralizing

99 ne and analyzed to examine the effect of the demineralization process.

100 fluence of adhesives and marginal sealing on demineralization progress using optical coherence tomogr

101 al uses and may model regulators of ferritin demineralization rates in vivo or peptide regulators of

102 The model also accounts for reported demineralization rates of natural biogenic and synthetic

103 The demineralization rates showed significant differences, p

104 he ferritin protein nanocage to control iron demineralization rates.

105 tivity was localized; dramatic escalation of demineralization-remineralization dynamics is the likely

106 Many therapies directed at correcting demineralization-remineralization imbalance should, in p

107 se specimens were then treated with a cyclic demineralization/remineralization regimen for 30 days.

108 el subsurface demineralization before cyclic demineralization/remineralization treatment, and signifi

109 namel under the NACP nanocomposite after the demineralization/remineralization treatment.

110 For the purposes of creating a demineralization-resistant layer, threshold illuminance

111 However, extracts made after demineralization revealed that phosphatidylserine had be

112 It would appear that the bone demineralization secondary to increased osteoblastic and

113 In Crohn's disease, severe skeletal demineralization, secondary hyperparathyroidism, and mus

114 Incubation with demineralization solution for 192 h resulted in signific

115 ry of the SOM fraction solubilized in the HF demineralization solution via solid-phase extraction.

116 ad exposure during periods of increased bone demineralization, such as menopause.

117 the continuum from the first atomic level of demineralization, through the initial enamel or root les

118 Before demineralization, tiles of hydroxyapatite crystals were

119 on of bone proteins has relied on the use of demineralization to better retrieve proteins from the ex

120 or analysis, extracted both before and after demineralization to remove deposited mineral.

121 Chemicals used for the demineralization treatment did not affect delta(2)H(n) v

122 The degree of DFDBA demineralization varies between tissue banks and may aff

123 t when the subjects used a F toothpaste, net demineralization was evident only with the seven- and 10

124 othpaste was used, statistically significant demineralization was observed when the frequency exceede

125 Significantly less demineralization was observed with Cu2+ and fluoride in

126 When most raters determined that demineralization was present at both readings, serum 25-

127 Depth of demineralization was significantly affected by "adhesive

128 Surface demineralization was subsequently promoted by subjecting

129 the same DS(En) and acid activity, rates of demineralization were the same in the acetic and propion

130 gingival recession; 19 received citric acid demineralization, while 17 did not.

131 d in groups (n = 9) according to the time of demineralization with citric acid (50%, pH 1): 15, 30, 9

132 ese surface modification techniques involves demineralization with citric acid or treatment with tetr

133 oradiography (MRG) showed significantly less demineralization with the H(3)PO(4) cavity surface treat